Documente Academic
Documente Profesional
Documente Cultură
Diagnosis
Endodonticperiodontal lesions present challenges to the clinician as far as diagnosis, prognosis and treatment.
The dental pulp and periodontal tissues are closely related. three main avenues for exchange of infectious elements between the two compartments are created by :
Non-physiologic Pathways
Vertucci 2005
De Deus 1975
Trauma
Iatrogenic
Developmental
Iatrogenic
Trauma
- 2- 4- 3-
Trauma
- 5Non-infective
- 6-
- 1- 2- 5Trauma
Developmental malformations
- 3- 4-
Coronal leakage
Perforations
Non-infective
- 6-
Resorptions
infective
PIRR
EIRR
PGG -1Provide a nidus for accumulation of bacterial biofilm and an avenue for the progression of periodontal disease that may also affect the pulp.
Trauma -2
1. loss of tooth structure and loss of fracture resistance after overzealous root canal preparation and subsequent restorative procedures leaving thin dentin walls.
2. Notches, ledges, and cracks induced by root canal preparation, root canal filling procedures, and seating of threaded pins and posts.
3. teeth serving as terminal abutments in cantilever bridges
Molars and premolars appear more often affected than incisors and canines
Clinical signs and symptoms associated with vertical root fractures vary hugely
pronounced pain and abscess formation or tenderness on mastication with mild dull pain and discomfort
Radiographic signs:
- Widening of the PDL - Lateral radiolucency along one or both of the lateral root surfaces
Often the diagnosis of a vertical root fracture has to be confirmed by surgical exposure
Treatment:
- There are reports of successful management of fractured teeth by re-attaching the fragments after extraction followed by re-implantation. - Fractured teeth are normally candidates for extraction.
Leakage -3Root canals may become recontaminated by microorganisms due to delay in placement of a coronal restoration and fracture of the coronal restoration and/or the tooth
Poor RCT -4Poor endodontic treatment allows canal re-infection and treatment failure. Endodontic failures can be treated either by orthograde or retrograde retreatment techniques.
Perforations
*size *location *time of diagnosis and treatment *degree of periodontal damage *sealing ability and biocompatibility of the repair material
MTA, Super EBA, Cavit , IRM, glass ionomer cements, composites, and amalgam
is a reparative process that occurs in response to minor trauma to the normal functioning teeth.
Pressure-induced
. Succedaneous teeth . Impacted teeth . Expanding lesions . Iatrogenic pressure, such as excessive orthodontic movements
Chmeical-induced:
occurs following extensive necrosis of the periodontal ligament with formation of bone onto a denuded area of the root surface.
This condition is most often seen as a complication of luxation and avulsion injuries.
Extracanal invasive:
uncommon form of root resorption.characterized by its cervical location, and invasive nature. There may be no signs or symptoms unless the resorption is associated with pulpal or periodontal infection.
The etiology of invasive cervical resorption is not fully understood. but, predisposing factors like traumatic injuries, orthodontic treatment, and intracoronal bleaching may be associated.
- Surgical exposure and removal of the granulation tissue, filling the defect followed by re-suturing the flap. "apically"
External Resorption
caused by stimuli such as: pulpal and/or sulcular infection
tumors
Internal Resorption The etiology of this type of root resorption is usually trauma. Extreme heat was suggested as a possible cause.
Removal of the inflammed pulpal tissue and obturation of the root canal system is the treatment of choice
The effect of periodontal inflammation on the pulp is controversial. It has been suggested that periodontal disease has no effect on the pulp before it involves the apex. On the other hand, several studies suggested that the effect of periodontal disease on the pulp is degenerative.
Teeth with caries or restorations that also have periodontal disease have more atrophic pulps than teeth with caries or restorations but no periodontal disease.
scaling, curettage as well as periodontal surgery may not induce severe inflammatory changes of the pulp
Pathogenesis
Living pathogens
Pathogenesis
Biofilm
A.a
T.f E.corrodens
C. albicans
Classification
Primary Endo Acute exacerbation of a chronic AP on a tooth with a necrotic pulp to drain through the PDL into sulcus mimicking a periodontal abscess, a deep periodontal pocket or a Grade III furcation in multirooted teeth
Primary Perio
It is the result of progression of chronic periodontitis apically along the root surface with wide generalized pockets.
Primary Endo with secondary Perio When primary endodontic disease remains untreated.
Plaque forms at the gingival margin of the sinus tract and leads to plaque-induced periodontitis in the area
Apical progression of a periodontal pocket continues until the apical tissues are involved via the apical foramen
Concomitant Lesion Concomitant endo-perio lesion is an additional classification that has been proposed to describe the presence of endo and perio disease as two separate and distinct entities
True combined endo/perio disease occurs less frequently than other endo/perio problems
Diagnosis
Inspection
Swelling caused by endodontic infections often occurs in the mucobuccal fold or spreads to the fascial planes.
Swelling associated with periodontal problems is found in the KAG and rarely spreads beyond the mucogingival line.
Palpation
Percussion
A tooth with an endodontic problem usually produces tenderness and pain on percussion and palpation.
Mobility
Ice test
Heat test
Hot gutta-percha applied to the tooth coated with petroleum jelly to prevent sticking to the tooth surface.
If a crown is present, a rotating rubber prophylaxis cup can be run on a dried tooth to create heat.
EPT
Cavity test
Preparation of a test cavity should be done without anesthesia.
A small access preparation is made through a crown or through the enamel to determine whether vitality is present in the pulp.
Probing
Sinus tracing
Aided inspection
Transillumination
dyes
Bite test
Radiographs
Periodontal and endodontic problems can radiographically mimic each other;therefore pulp testing and periodontal probing must be used along with the radiograph.
1ry endo
1ry perio
sequelae
-Necrotic pulp with a chronic AP -Draining sinus tract -Swelling in the mucobuccal fold is pathognomonic.
Dx
-Negative pulp vitality tests -Periodontal probing is within normal limits -sinus tracing
Tx
NSRCT
Peiodontal Tx
Px
Excellent
sequelae
Dx
-Probing "generalized -Pulp vitality tests are negative periodontitis' -plaque and calculus in the -Pulp vitality test results can be pocket mixed -NSRCT -Perio.Tx
Tx
Perio. Tx + RCT
Px
True combined
Concomitant
sequelea
-Pulpal and periodontal pathoses develop independently and unite -Significant periodontalinvolvement
Pulpal lesion separate from the periodontal lesion BUT occurring at the same time
Dx
-Different Diagnostic methods for pulp & periodontium D.D: vertical root fracture perforations resorption -Good conservative NSRCT. - Periodontal therapy can be performed before, during, or immediately after the endodontic treatment. -Hemisection or root resection. -SRCT
Tx
-Good conservative NSRCT. - Periodontal therapy can be performed before, during, or immediately after the endodontic treatment. -Hemisection or root resection. -SRCT
Px
References
- Periodontics: Medicine, Surgery and Implants, 1e Louis F. Rose , Brian Mealey , Robert Genco
Clinical Periodontology and Implant Dentistry, 5e Jan Lindhe, Niklaus P. Lang, Thorkild Karring
Diagnosis, prognosis and decision-making in the treatmentof combined periodontal-endodontic lesions by Ilan Rotstein & James H. S. Simon "2000"
The endo-perio lesion: a criticalappraisal of the disease condition by ILAN ROTSTEIN & JAMES H. SIMON "2006"
!ank Y"