PATHOPHYSIOLOGY

Modifiable Factors: Diet Environment Occupation Alcohol Intake Increased blood flow to the brain Non - Modifiable Factors: Gender Age Hereditary

Compromise of the integrity of cerebral arterioles Weakening of the walls of a blood vessel Formation of CharcotBouchard aneurysm Rupture of the anterior cerebral artery

Intracerebral hemorrhage Inflammation of the Frontal lobe Alteration in the cerebral component Hematoma formation Presence of free blood in the interstitial areas

Increase Intracranial pressure

Thrombus formation CT SCAN and MRI

Cell membrane destruction

Compression of the brain components Stimulates further swelling and inflammatio n

Decrease oxygen supply

Compensatory mechanism

Cellular edema

Vasospasm

Electrolyte imbalance

Acidosis

-projectile vomiting, numbness of extremiities , visual disturbances

Ischemia

Stimulation of vasomotor centers

Scar Formation Alteration in the frontal lobe function Increase systemic pressure

Serum Electrolyte Test Decreased Mg and K ABG(Respiratory alkalosis) changes in the v/s

X RAY, CT scan, MRI

Brocas aphasia Hemiparesis Hemianopsia dysarthria incapable of abstract thinking

Increase BP Without Medical intervention Brain stem herniation With Medical Intervention

Comatose Stupor Worsening of the condition

Management: SURGICAL: craniectomy, craniotomy MEDICAL: Pharmacologic Therapy (citicholine, mannitol, dilatin, captopril, Nitroglycerin, furosemide, remopain, Kalium Durule, MgSO4)

Death