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HYPO PARATHYROIDISM-this hormone is – Increased blood calcium

responsible for the regulation for the regulation of – Muscle weakness


calcium and phosphorus levels in the blood. – Osteoporosis
– Calcium and phosphorus levels must maintained – Bone pain and pathological fracture
within in the normal limits. – Increased urinary output and renal calculi
– Bone density is also maintained by the – Nausea and vomiting.
parathormone. – Changes in EKG (shortened QT interval and
signs of the heart block)
SIGNS AND SYMPTOMS:
– Decreased blood calcium MANAGEMENT:
– Increased blood phosphorus – Removal of parathyroid. Post operative
– Neuromuscular hyper excitability manangement,
– Carpopedal spasms – Assessment of the client for respiratoty distress
– Urinary frequency – Maintaining suction , oxygen, and tracheostomy
– Mood changes (depression) set at the bedside
– Dry, scaly skin, and thin hair – Checking for bleeding 1-5 is normal
– Cataracts – Checking the serum calcium level and serum
– Changes in teeth cavities phosphorus
– Seizures – Checking Trousseau’s sign, which is carpopedal
– Changes in EKG prolonged QT intervals and spasms, noted when the bp cuff is inflated on
inverted T waves. the arm, Chvostek’s sign, facial nerve is tapped
with the nurse’s index finger and twishing of the
MANAGEMENT: facial muscle is observed.
– Administration of IV calcium gluconate and long
term of calcium salts.
– Vit. D supplements can be given to increase the ADRENOCOTICAL INSUFFIENCY (ADDISON’S
absorption of calcium preparation. DISEASE)
– Result of long term use of steroid or rapid
cessation of corticosteroids. It also caused by
sepsis, surgical stress or hemorrhage of the
adrenal glands (Waterhouse-Friderichsen
HYPERPARATHYROIDISM- overproduction of Syndrome)
parathormone.
SIGNS AND SYMPTOMS:
SIGNS AND SYMPTOMS: – Weakness
– Decreased blood phosphorus – Bronze like pigmentation of the skin
– Decreased level glucose – Osteoporosis
– Decreased BP – Decreased Serum Potassium and Decreased
– Anorexia Serum Chloride
– Sparse axillary hair – Increased 17-hydroxy corticoids
– Urinary frequency – Decreased eosinophils and Decreased
– Depression Lymphocytes
– Addisonian crises are severe hypotension,
cyanosis, and shock MANAGEMENT:
– Removing part of adrenal gland or decreasing
DIAGNOSIS; dosage of steroid medication
– Evaluation of serum sodium and chloride
– Evaluation of ketosteroid and 17-
hydroxycorticoids
– Adrenal function is evaluated by administering
adrenocoticoid stimulating hormone (ACTH) and
checking changes cortisols level. DIABETES MELLITUS
– Chronic disorder of carbohydrate metabolism,
MANAGEMENT; marked by hyperglycemia and hypoglycaemia.
– Use of IV cortisone and plasma expanders to
maintained BP. SIGNS AND SYMPTOMS:
– IM cortisol form of dexamethasone (Decadron) – Wt. Loss
or oral Prednisone. – Ketonuria, fruity breath
– Polyphagia
ADRENOCORTICAL HYPERSECRETION – Polyuria
( CUSHING’S DISEASE) – Polydipsia
– Prolonged administration of cortisone or due to – Delayed wound healing
hypersection of the adrenal cortex. – Elevated blood glucose, normal 70-110 mg/dl

SIGNS AND SYMPTOMS: DIAGNOSIS:


– Pendulous abdomen – Glucose tolerance test
– Buffalo hump – Fasting blood glucose levels
– Moon faces – Two-hour post prandial
– Hirsutism (facial hair) – Dextrostix
– Ruddy complexion (dark red) – Urine check for glucose, if 240 mg/dl ketonuria
– Increased BP occurs.
– Hyperglycemia
ALERT: Regular Insulin peaks in 90-120 minutes
and NPH insulin peaks in 8-12 hours, the nurse
should instructed the client to draw up the regular
insulin (clear) and then draw up the NPH insulin. ROME
HTPERGLYCEMIA S/SX:
Respiratory Opposite
– Headache Metabolic Equal
– Nausea/vomiting
– Coma Respiratory Acidosis- ph , CO2 ,
– Flushed, dry skin
– Glucose and acetone in urine HCO3

TIP: Metabolic Acidosis- ph , CO2 , HCO3


➢ Hot and dry; blood sugar high- this means DM
skins is hot and he is dehydrated, his blood
glucose level is likely high. Respiratory Alkalosis- ph , CO2 ,
➢ Cold and clammy; need some candy- means DM HCO3
skins is cold and clammy, his glucose level is
low and he needs a glucose.
Respiratory Acidosis- ph , CO2 ,
HYPOGLYCEMIA S/SX: HCO3
– Headache ABG VALUES:
– Irritability
– Disoriented
➢ HCO3= 24-26 meq/ml, BICARB
– Nausea and vomiting ➢ CO2= 35-45 meq/ml, CARBON
– Diaphoresis DIOXIDE
– Pallor
– Weakness ➢ PaO2= 80%-100%, PARTIAL
– Convulsion OXYGEN
– Coma
– Death ➢ SaO2= >95%, SATURATION

SODIUM(Na)= 135-145meq/l
POTASSIUM(K+)= 3.5-5.5meq/l
CALCIUM(Ca++)= 4.5-5.5 meq/l or PROTEIN: 6.2-8.1 g/dl
8.5-10.5 mg/dl ALBUMIN:3.4-5.0 g/dl
CHLORIDE(Cl)= 95-105 meq/l
PHOSPHORUS(ph+)= 2.5-4.5meg/l THERAPEUTIC DRUG LEVEL
MAGNESIUM(Mg++)= 1.4-2.5 meq/l DIGOXIN: 0.5-2.0 mg/dl
Toxic level: 4.8-9-6 meq/l LITHIUM: 0.8-1.5 mg/dl
DILANTIN:10-20 mg/dl
HEMATOLOGY VALUES THEOPHYLLINE:10-20 mg/dl
RBC: 4.5-5.0 million IOP:10-21mmhg
WBC: 5,000-10,000
PLATELET: 200,000-400,000 FHR: 120-160 BPM
Hgb: 40%-48% women, 42%-50% VARIABILITY: 6-10 BPM
men CONTRACTION:
FREQUENCY= 2-5 MINS. APART
DURATION= <90 SEC.
CHEMISTRY VALUES INTENSITY= <100 mm/hg
GLUCOSE: 70-110 mg/dl AMNIOTIC FLUID: 500-1200 ml
SPECIFIC GRAVITY: 1.010-1.030 Nitrozine urine=litmus paper green
BUN: 7-22 mg/dl Amniotic Fluid= litmus paper blue
CREATININE: 0.6-1.35 mg/dl AVA: umbilical
Arteries- 2, carry deoxygenated blood
LDH:100-190 U/L Vein- 1, carry oxygenated blood
CPK: 21-232 U/L
URIC ACID: 3.5 -7.5 mg/dl Coumadin (sodium warfarin) PT:10-12 sec
Antidote: Vit.K
TRIGLYCERIDE: 40-50 mg/dl
TOTAL CHOLESTEROL: 40-50 mg/dl Heparin PTT 30-45 sec
BILIRUBIN:<1.0 mg/dl Antidote: Protamine Sulfate
RULE OF NINE FOR BURN 5 Ps Fracture and Compartment S.
Head- 9% Pain
Arms- 18% Pallor
Back- 18% Pulselessness
Leg- 36% Paresthesia
Genitalia- 1% Polar(cold)

Diets WASTE MANAGEMENT COLOR CODING


Renal Diets: BLACK- non-infectious, DRY
-high Calorie GREEN- non-infectious, WET
-high Carbo YELLOW- Dry & Wet Chemical & other potentiall
-low Protein infection,pathologic chemical waste
-low Potassium ORANGE- with TREFOIL SIGN, Radioactive sign
-low sodium
-fluid restriction CODING IN DISASTER MANAGEMENT
RED-IMMEDIATE, life threatening but survivable
with minimal intervention
Gout Diets YELLOW-DELAYED,significant injuries but can wait
-low Purine hours without life threat to life
-omit Poultry GREEN-MINIMAL, walking wounded
BLACK-EXPECTANT, extensive injury with unlikely
TX for Spider bites: survival
R-est
I-ce MIDWIFE- 1st level health worker, 1:5000
C-ompression population
E-levate extremity PUBLIC HEALTH NURSE- 2nd level health worker,
1:10,000 population
TX for SCA: PHYSICIAN (MHO)- 3rd level health worker,
H-eat 1:20,000 population
H-ydration
O-xygen Cerebral Edema- mannitol, corticosteroid
P-ain Meds. Increase Intracranial Presure- mannitol
Febrile Seizure- Phenobarbital
Partial Seizure- Carbamazipine
Myoclonic Seizure- Valproic acid
Status Epilepticus- Diazepam(Valium), Phenytoin
Hepatic encephalopathy- Neomycin

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