Care of Patient With Liver Cirrhosis For Nursing Process Related Nursing Care

Care Study
Nursing Care of the patient with
Cirrhosis
Medical Nursing
Submitted to: Tutor

Mr. Mohamed Anwer Naleef
AUC/Nur/2011/0028
Faculty of Nursing
Aquinas University College, Colombo
Cirrhosis
Medical Nursing
Care Study
Submitted to: Tutor
Submitted by: Mohamed Anwer Naleef

Nursing Student 2011/2014
AUC/Nur/2011/0028
Faculty of Nursing
Aquinas University College
Colombo
Care Study of Patient with
Liver Cirrhosis
Mr. Mohamed Anwer Naleef

Nursing Student 2011/2014
AUC/Nur/2011/03/0028
Faculty of Nursing
Aquinas university College
Srilanka
Acknowledgement
I express my heartfelt gratitude, sincere appreciation
and profound regards to the following people who, gave
guidance, strength, and encouragement in making this case
presentation possible. First of all, Thanks to God, for
granted us the knowledge and skills. To their family,
friends, and classmates, for their consideration and
unending support, emotionally, spiritually and financially.
To their clinical instructor, Madam. for guiding us in the
course of making this case presentation and giving them tips
on how to have a good presentation. Thanks to all Lectures
of Aquinas University and School of Nursing and all
medical personnel and staff members of Hospital Wattala,
A Ward, for sharing ideas, cooperating and giving full effort
in making the case presentation successful Lastly, to our
client and his family for their acceptance and willingness to
share time, effort and giving us the essential information
needed for this case presentation.
Thank you Very Much
Contents:-
1. Objectives of Care Study

2. Introduction
2.1. Liver
2.2. Liver Cirrhosis
3. Anatomy and Physiology of Liver
3.1. Anatomy of Liver
3.2. Physiology of Liver
4. Assessment of the Patient
4.1. Patient History and Physical Assessment
4.2. Care plan of the Patient
4.3. Vital Signs Chart
4.4. Intake and Output Chart
4.5. Medication Chart
4.6. Discharge and Follow Up Care
5. Disease Condition ( Cirrhosis)
5.1. Introduction/Definition
5.2. Etiology
5.3. Pathophysiology
5.4. Clinical Manifestation
5.5. Investigations
5.6. Management
5.7. Complications
5.8. Prognosis
6. Nursing Care of Patient with Cirrhosis
6.1. Nursing Assessment
6.2. Nursing Diagnosis
6.3. Nursing Interventions
7. Summary and Conclusion
8. References
1. Objectives
Short term Objectives:1.

2.
3.
4.
5.
6.
To relieve Pain.
To promote Nutritional Level
To give Health Education.
Provide Comfort.
To prevent complication.
To maintain optimal fluid level.
Long term Objectives:1. To prevent Complications.

2. To give Health Education.
3. To give the knowledge of the health promotion and taking health
decision.
4. To maintain normal nutritional level.
5. To continue Follow up Care.
Section 2
Introduction
2. Introduction
2.1. The Liver

The liver is one of the largest and most complex organs in the body. It stores vital
energy and nutrients, manufactures proteins and enzymes necessary for good
health, protects the body from disease, and
breaks down (or metabolizes) and helps
remove harmful toxins, like alcohol, from
the body. It is one of the most important
organs in the body since it has many
significant functions. A lack or failure to
provide proper care of it may lead to an
abnormality or disorder.
2.2. The Liver Cirrhosis

Cirrhosis is defined histologically by septal fibrosis with nodular parenchymal
regeneration. Only 60% of patients with
alcoholic cirrhosis have signs or
symptoms of liver disease, and most
patients with cirrhosis have no clinical
history of alcoholic hepatitis. Liver
enzyme levels may be relatively normal
in cirrhosis without alcoholic hepatitis.
Concomitant HCV infection is common
in patients with alcoholic liver disease.
The prognosis of alcoholic cirrhosis
depends on whether patients continue to
consume alcohol and whether there are
signs (jaundice, ascites, or gastrointestinal tract bleeding) of chronic liver disease.
The 5-year survival rate for patients who have ascites, jaundice, or hematemesis and
abstain from alcohol is 89% and for those who have signs and continue to consume
alcohol, 34%. Liver transplant is an option for patients with end-stage alcoholic liver
disease if they demonstrate that they can maintain abstinence from alcohol.
3. Anatomy and Physiology of Liver
3.1. Anatomy of Liver

The liver largest organ in the body. It weight between 1.0 2.5kg (2.2 5.5lb) and is
heavers in the male than the female. It is a wedge shaped organ, lying immediately
below the diaphragm in the right hypochondrium and epigastrium.
There are two distinct sources that supply blood to the liver, including the following,
Oxygenated blood flows in from the hepatic artery.
Nutrient rich blood flows in from the hepatic and portal vein.
The liver is described as having right and left lobes, and superior, inferior,
anterior, and posterior surfaces. The right and left main lobes are made up of
thousands of lobules. These lobules are connected to small ducts that connect with
larger ducts to ultimately form the hepatic duct. The hepatic duct transports the bile
produced by the liver cells to gallbladder, and Duodenum.
Blood Supply of the Liver

The liver receives blood from two sources and is an extremely vascular organ.
The hepatic artery, which is a branch of the Coeliac axis from the abdominal
aorta, conveys oxygenated blood to the liver cells.
The Portal Vein conveys venous blood, poor in oxygen but rich in nutrients,
frm the Stomach and Intestines.
Venous drainage from the liver is by the Hepatic Veins which empty into the
inferior vena cava.
Due to its great vascularity, lacerations of the liver are very dangerous and
results in profuse haemorrhage.
3.2. Functions of the Liver
Secretion of bile
Storage of glycogen
Metabolism of fat
Deamination of amino acids
Production of the plasma protein
Storage of vitamins
Storage of irons
Production of clotting factors
Production of heat
Detoxification
Section 4
Assessment of the Patient
4. Assessment of the Patient with Cirrhosis

Data gathering at 2013/02/20 at 10am
Bio Graphic Data
Patient Name
Age
Sex
Address
Civil Status
Religion
Contact No
BHT No
Ward & Room No
Consultant Name
Admission Date
: Mr.
: 58 years
: Male
:
: Divorced
: Buddhist
:
: 30848
:
:
:
Chief Complaint
Inadequate urine output since 3 days
Vomiting 4 times today
Vomits are water and light Brown color content.
Swelling in Abdomen and Scrotum since 10 days
Generalized swelling in the abdomen and Scrotum, Tenderness
when palpating the abdomen and Scrotum.
Poor oral Feeding 10 days (Loss of Appetite)
History of Present Illness

Patient is a 58 years old Mr. Rmale, who was in his usual state of health until
Saturday 16/01/2013, when noticed diffuse Swelling throughout his abdomen and
lower extremities. Swelling increased throughout the Saturday and Sunday. Patient
reports Discomfort, Pain and Tenderness. During this time patient attempted to
reduce the swelling by applying ice to affected region.
On Monday, 18/02/2013, the patient came to the Hospital, for a scheduled
appointment to monitor his Diabetes Mellitus condition. That time he presented with
Abdominal Distention, Diffuse Oedema of the lower extrimities and Swollen of the
Scrotum.
At that time Dr. (VP) admitted him. Since admission, he has been treated with
withdrawing peritoneal Cavity fluid (Paracentesis), NG Insertion, and Administration
of Diuretics ( Lasix 40mg IV), which has reduced the sowelling in his abdomen and
scrotum. Patient reports reduced discomfort, tenderness and pain.
Patient has a history of Diabetes Mellitus since 10 years and Hypertension
since 6 years which was controlled. Today the patients abdomen shows distended and
scrotal edema, Poor oral intake, inadequate urine output, and Vomiting.
Past Medical History

Liver Alcoholic Cirrhosis since two years.
Diabetes Mellitus since 10 years patient on Treatment.
Hypertension since 6 years patient on Treatment.
No Known history of Tuberculosis, Cancer, Coronary Artery Disease, Asthma, and
Anemia
Past Surgical History

Herniotomy in 2008
After surgery Blood transfusion done, Blood Group is O+.
After Surgery no Surgical Complications.
Medication History
Metformin 850mg bd
Furosemide 40mg nocte
Spironolactone 25mg bd
Tolbutamide 500mg bd
Atrovastatin 10mg nocte
Family History
Father, Uncles and Brother died at 57 from Myocardial infarction.

Family history of Diabetes Mellitus on both sides.
His daughter (20years old) in good health.
Does not know where about of his mother.
Social History
Living arrangement
Divorced and lives alone
Residence
Resides in an apartment, No identified harmful
environmental exposure.
Occupation
Retired Bank Manager
Tobacco, Alcohol and other Drug Use
One pack per day smoking history until 2010, 20 years of
Alcohol use until 2012 June.
Diet and Exercise
Patient maintains a low sodium diet and gets moderate
exercise.
Education
He is a University Graduate
Physical Assessment of the Patient

VITAL Signs
Temperature
Pulse Rate
Respiratory Rate
Blood Pressure
Pain
Height
Weight
MBI
98.5 F/Axilla
81/bpm
24/bpm
120/80mmHg
6/10 in pain Scale
174cm
78kg
General Appearance
Patient is appropriately groomed for the environment, is oriented for Place,
Time, and Person, and in No apparent Distress. He appears jaundiced and
underweight.
Head
Head is regular shape, with no apparent lesion, Masses, or Foreign bodies.
Scalp shows no evidence of skin condition or infestation, and exhibited no
tenderness on palpation.
Eyes
Lids are normal, No evidence of discharge, Ptosis or edema.
Direct and consensual reactivity of light.
Visual fields are normal
Left 6/6, Right 6/6
Ears/ Nose/ Throat
External ears and nose are of symmetric regular shape and size.
No scars, lesions, masses or foreign bodies.
No tenderness to palpation of ears and nose.
Nasal mucosa is moist and pink with no discharge.
No allergic Rhinitis.
No Ear discharge.
Sense of smell is good.
Mouth/Dental
Lips, Teeth, and gums all appear healthy with no lesions and ulceration.
Oral mucosa, tonsils, and palate appear healthy. No appear masses, lesions,
foreign bodies or other abnormalities.
Sense of taste is good.
Loss of Appetite in 10 days.
Neck
Neck is symmetric with no any lesions or ulceration.
There is no tenderness to palpation.
Skin
Skin appears is yellow color and there are no apparent rashes lesions or
ulcers.
Face
No scars in the face.
No wrinkles and patient facial appearance is normal.
Respiratory System
Chest is regular shape and size.

Chest girth is 34cm.
There is no apparent use of accessory muscle for normal breathing.
No reported respiratory related symptoms.
No cough, Dyspnoea, and Hemoptysis.
Cardiovascular System
No chest pain.
Patient has a 4 years history of hypertension, patient on treatment, regular
check up and using drugs such as Spironolactone and Frusemide.
Nails are Normal.
No cyanosis
CRFT (capillary refilling time) 2 seconds.
No oedema in the body and other extremities.
Extremities are Warm.
Abdomen
Abdominal walls shows/ appearing distended and round.
Pain in the right upper quadrant of the abdomen, this pain is on and off
occurring an exertion.
Abdominal girth is 65cm.
Umbilicus is protruded.
Tenderness to palpation of Abdomen.
Nutritional Assessment
Patient non vegetarian.

No Anemic signs in the patient.
Vomiting
4times a day, vomits content are water.
No Nausea.
10 days history of Anorexia.
No Heart burn.
Elimination
Bladder
No Incontinence
4days history of Urinary Retention.
No Hematuria.
No Burning Sensation during pass Urine.
Bowel
No Constipation/diarrhea.
Patient taking Lactulose 30cc use in Ward.
No Malena
No difficulty passing Stool.
Neurological Assessment
No Syncope
No Confusion
Headache on the Morning
No Convulsive signs.
Musculoskeletal Assessment
No Numbness
No Myalgia
No Fracture/injury
Patient complain of Difficulty in Walking because abdominal distention, and
Dyspnoea on exertion.
Psychiatric Assessment
Patient Alert, and Oriented to Time, Place, and Person.
Patient Anxiety because of Disease Condition.
Drug Treatment
Klean prep Enema bd Per Oral

IV Levofloxacin 500mg daily
IV Pantocid 40mg bd
Lactulose 30cc tds Per Oral
IV Metranindazole 200mg tds
IV Hartman 50cc/hr
Metformin 850mg bd Per Oral
Furosemide 40mg bd Per Oral
Spironolactone 25mg bd Per Oral
Tolbutamide 500mg bd Per Oral
Atorvatatin 10mg nocte Per Oral
Section 5
Cirrhosis
6. Disease Condition ( Cirrhosis)

6.1. Introduction/Definition
6.2. Etiology
6.4. Clinical Manifestation
6.5. Investigations
6.6. Management
6.7. Complications
6.8. Prognosis
5.1. Cirrhosis
Cirrhosis is a complication of many liver diseases that is characterized by
abnormal structure and function of the liver.
The diseases that lead to cirrhosis do so because they injure and kill liver cells,
and the inflammation and repair that is associated with the dying liver cells
causes scar tissue to form.
The liver cells that do not die multiple in an attempt to replace the cells that
have died. This results in clusters of newly formed liver cells (regenerative
nodules) within the scar tissue.
There are many causes of cirrhosis; they include chemicals, viruses, toxic
metals and autoimmune liver disease in which the bodys immune system
attacks the liver.
5.2. Etiology
Alcohol
Is a very common cause of cirrhosis. The development of cirrhosis
depends upon the amount and regularity of alcohol intake. Chronic, high level
of alcohol consumption injures liver cells. Alcohol cause a range of liver
diseases; from simple and uncomplicated fatty liver, to the more serious fatty
liver with inflammation, to cirrhosis.
Chronic viral hepatitis
Is a condition where hepatitis B or hepatitis C virus infects the liver for
years. Most patients with viral hepatitis will not develop chronic hepatitis and
cirrhosis. For example, the majority of the patients infected with hepatitis A
recover completely within weeks, without developing chronic infection.
Inherited (genetic) disorders
Result in accumulation in toxic substance in the liver which lead to tissue
damage and cirrhosis. Examples include the abnormal accumulation of the iron
(hemochromatosis) or copper (wilsons disease). In hemochromatosis, patients
inherit a tendency to absorb an excessive amount of iron from food. Over time iron
accumulation in different organs throughout the body causes cirrhosis, arthritis,
heart muscle damage leading to heart failure, and testicular dysfunction causing
loss of sexual drive. Over time copper accumulates in the liver, eyes, and brain.
Cirrhosis, tremor, psychiatric disturbances and other neurological difficulties
occur, if the condition is not treated.
Autoimmune hepatitis
Is a liver disease caused by a an abnormality of the immune system that is
found more commonly in women. The abnormal immune activity in autoimmune
hepatitis causes progressive inflammation and distruction of liver cells
(hepatocytes), leading ultimately to cirrhosis.
Primary biliary cirrhosis (PBC)
Is a liver disease caused by an abnormality of the immune system that is
found predominantly in women. The abnormal immunity in PBC causes
chronic inflammation and destruction of the small bile ducts within the liver. In
PBC, the destruction of the small bile ducts blocks the normal flow of the bile
into the intestine. As the inflammation continues to destroy more of the bile
ducts, it also spread to destroy nearby liver cells. As the destruction of the
hepatocytes proceeds, scar tissue (fibrosis) forms and spreads throughout the
areas of destruction.
Non alcoholic fatty liver disease (NAFLD)
Refers to a wide spectrum of liver diseases that, like alcoholic liver
disease, ranges from simple steatosis, to nonalcoholic steatohepatitis (NASH),
to cirrhosis. All stages of NAFLD have in common the accumulation of fat in
liver cells. NAFLD is associated with the metabolic syndrome and diabetes
mellitus type 2. Obesity is the most important cause of insulin resistance,
metabolic syndrome, and type 2 diabetes mellitus.
Cryptogenic cirrhosis (cirrhosis due to unidentified cause)

Is a common reason for liver transplantation. Because Cryptogenic
cirrhosis is due to NASH (nonalcoholic steatohepatitis) caused by long
standing obesity, type 2 DM and insulin resistance.
Primary sclerosing cholangitis (PSC)
Is an infection and inflammation of the common bile duct and is an
uncommon disease found frequently in patients with ulcerative colitis. In PSC,
the large bile ducts outside of the liver become inflamed, narrowed and
obstructed. Obstruction to the flow of bile leads to infections of the bile ducts
and jaundice and eventually causes cirrhosis.
Infants can be born without bile ducts (biliary atresia)
Its ultimately develop cirrhosis. Other infants are born lacking vital
enzymes for controlling sugars that leads to the accumulation of sugars and
cirrhosis. On rare occasions, the absence of a specific enzyme can cause
cirrhosis and scarring of the lung (alpha 1 antitrypsin deficiency).
Less common cause of cirrhosis include unusual reactions to some drugs and
prolonged exposure to toxins, as well as chronic heart failure (cardiac
cirrhosis).
Liver cirrhosis occurs when the

regenerative capacity of the liver is
overwhelmed by alcohol consumption,
drug or chemical damage, long-term
infection brought upon by infection or
extra hepatic bile obstruction.
Numerous infiltrating inflammatory
cells stimulate fibrosis in response to such massive destruction. It will then
result in an ever increasing scarring until sheets of fibrous repair tissue from
throughout the liver. These are diffusely distributed thereby isolating areas of
the liver that still retains their regenerative capacity.
These detached areas are called nodules and are readily apparent in the
livers surface. This condition of modularity and fibrosis is called cirrhosis.
Cirrhosis is a non-specific end-stage disease towards which various pathologic
consequences converge. The differing degrees of functional loss of the
hepatocytes results in variable signs and symptoms. In certain instances, the
liver is able to compensate for necrosis that none or minimal symptoms appear.
This situation leads to an unnoticed and unresolved destruction of hepatocytes
until adequate function can no longer be maintained and reserves are
completely depleted leading to liver failure.
Liver cirrhosis is defined by two principal factors: Portal Hypertension and
Hepatic Dysfunction.
Portal hypertension is the result of restricted flow of blood through the liver to
the hepatic veins and then to the inferior vena cava. The resulting portal
congestion increases portal pressures and decreases blood flow to the liver.
With reduced blood flowing to the cirrhotic liver, the hepatocytes have minimal
accessed to blood, severely hampering their capacity to detoxify harmful
chemicals.
As a result, toxins become more concentrated in the blood producing damaging
effects particularly the production of ammonia (from amino acid breakdown).
Instead of being excreted, ammonia stays in the blood causing hepatic
encephalopathy and a noticeable foul breath.
Furthermore, the hepatocytes continue to die leading to a progressive
deterioration of the livers regulatory capabilities resulting in hypocoagulation
and hypoalbuminemia.
Congestion in the hepatic portal system causes blood to be diverted to the

collateral vessels forcing them to accommodate larger volumes. This
engorgement causes the veins to bulge producing easily visible hemorrhoids.
Another consequence of this diversion is the dilation of the thin-walled
esophagus causing esophageal varices. Esophageal varices are subjected to
trauma as food is swallowed and expose to gastric reflux. This poses a
potential threat of rupture and bleeding.
When the varices rupture it is usually asymptomatic and sudden, bringing forth
a large scale blood loss. Compounded by a prolonged bleeding time,
esophageal varices is a very serious complication of liver cirrhosis.
Portal hypertension in liver cirrhosis also causes ascites (accumulation of fluid
in the peritoneal cavity) and splenomegaly. Ascites is caused by hampered
albumin production, the osmotic pressure decreases reducing the return of
fluid to the blood from the tissues. It results in a significant and pronounced
abdominal distention, compressing the abdomen and compromising breathing.
Development of Liver Alcoholic Cirrhosis
Normal liver
Columns of hepatocytes 12
cells thick radiate from the portal
tracts (PT) to the central veins. The
portal tract contains a normal intra
lobular bile duct branch of the
hepatic artery and portal venous
radical
Bridging fibrosis (stained pink, arrows)

spreading out around the hepatic vein and
single liver cells (pericellular), and linking
adjacent portal tracts and hepatic veins.
A cirrhotic liver
The liver architecture is

disrupted. The normal arrangement
of portal tracts and hepatic veins is
now lost and nodules of proliferating
hepatocytes are broken up by strands
of pink/orange-staining fibrous tissue
(arrows) forming cirrhotic nodules
(CN).
5.4. Signs and Symptoms of Cirrhosis
Many people with cirrhosis have no symptoms during the early phases of the
disease. Symptoms are caused by either of 2 problems:
Gradual failure of the liver to carry out its natural functions.
Distortion of the livers usual shape and size because of scarring.
The most common symptoms of cirrhosis are as follows:
Tiredness (Fatigue) or even exhaustion.

weakness
Nausea, Vomiting
Loss of Appetite leading to weight loss
Loss of sex drive
Signs and Symptoms may not appear until complications of cirrhosis set in.
Many people do not know they have cirrhosis until they have a complication:
Jaundice Yellowing of the skin and eyes from deposition of bilirubin in
these tissues. Bilirubin is a product of the breakdown of old blood cells
in the liver.
Fever
Diarrhea
Itching from deposition in the skin of products of the breakdown of
bile.
Abdominal pain and Ascitis from enlargement of the liver or formation
of gallstones.
Weight gain from fluid retention
Swelling in ankles and legs (Edema) from fluid retention
Difficulty breathing from fluid retention
Sensitivity to medications due to impairment of the livers ability to
filter medications from blood.
Confusion, Delirium, Personality changes, or Hallucinations
(Encephalopathy) from buildup of drugs or toxins in the blood, which
then affect the brain.
Extreme sleepiness, Difficulty awakening, or Coma other symptoms of
Encephalopathy
Bleeding from Gums or Nose Due to impaired production of the
Clotting Factors.
Easy bruising due to impaired production of the Clotting Factors.

Blood Vomit or Feces due to bleeding of varicose veins caused by
liver congestion.
Hemorrhoids varicose veins in Rectum due to liver congestion.
Loss of Muscle Mass (wasting)
In women, abnormal menstrual periods Due to impairment of
hormone production and metabolism.
In men, enlargement of the breasts (Gynecomastia), Scrotal swelling or
small testes Due to impairment in hormone production and
metabolism.
5.5. Diagnostic Tests (Investigations)

The single best test for diagnosing cirrhosis is biopsy of the liver. Liver
biopsies, however, carry a small risk for serious complications, and, therefore, biopsy
often is reserved for those patients in whom the diagnosis of the type of liver disease
or the presence of cirrhosis is not clear. The possibility of cirrhosis may be suggested
by the history, physical examination, or routine testing. If cirrhosis is present, other
tests can be used to determine the severity of the cirrhosis and the presence of
complications. Tests also may be used to diagnose the underlying disease that is
causing the cirrhosis. The following are how to diagnose and evaluate cirrhosis:
In taking a patient's history, the physician may uncover a history of excessive
and prolonged intake of alcohol, a history of intravenous drug abuse, or a
history of hepatitis. These pieces of information suggest the possibility of liver
disease and cirrhosis.
Patients who are known to have chronic viral hepatitis B or C have a higher
probability of having cirrhosis.
Some patients with cirrhosis have enlarged livers and/or spleens. A doctor can
often feel (palpate) the lower edge of an enlarged liver below the right rib cage
and feel the tip of the enlarged spleen below the left rib cage. A cirrhotic liver
also feels firmer and more irregular than a normal liver.
Jaundice (yellowness of the skin and of the whites of the eyes due to elevated
bilirubin in the blood) is common among patients with cirrhosis, but jaundice
can occur in patients with liver diseases without cirrhosis and other conditions
such as hemolysis (excessive break down of red blood cells).
Swelling of the abdomen (ascites) and/or the lower extremities (edema) due to
retention of fluid is common among patients with cirrhosis, although other
diseases can cause them commonly, for example, congestive heart failure.
Patients with abnormal copper deposits in their eyes or certain types of
neurologic disease may have Wilson's disease, a genetic disease in which there
is abnormal handling and accumulation of copper throughout the body,
including the liver, which can lead to cirrhosis.
Esophageal varices may be found unexpectedly during upper endoscopy
(EGD), strongly suggests cirrhosis.
Computerized tomography (CT or CAT) or magnetic resonance imaging (MRI)
scans and ultrasound examinations of the abdomen done for reasons other than
evaluating the possibility of liver disease may unexpectedly detect enlarged
livers, abnormally nodular livers, enlarged spleens, and fluid in the abdomen,
which suggest cirrhosis.
Advanced cirrhosis leads to a reduced level of albumin in the blood and
reduced blood clotting factors due to the loss of the liver's ability to produce
these proteins. Thus, reduced levels of albumin in the blood or abnormal
bleeding suggest cirrhosis.
Abnormal elevation of liver enzymes in the blood (such as ALT and AST) that
are obtained routinely as part of yearly health examinations suggests
inflammation or injury to the liver from many causes as well as cirrhosis.
Patients with elevated levels of iron in their blood may have hemochromatosis,
a genetic disease of the liver in which iron is handled abnormally and which
leads to cirrhosis.
Auto-antibodies (antinuclear antibody, anti-smooth muscle antibody and antimitochondrial antibody) sometimes are detected in the blood and may be a clue
to the presence of autoimmune hepatitis or primary biliary cirrhosis, both of
which can lead to cirrhosis.
Liver cancer (hepatocellular carcinoma) may be detected by CT and MRI
scans or ultrasound of the abdomen. Liver cancer most commonly develops in
individuals with underlying cirrhosis.
If there is an accumulation of fluid in the abdomen, a sample of the fluid can be
removed using a long needle. The fluid then can be examined and tested. The
results of testing may suggest the presence of cirrhosis as the cause of the fluid.
5.6. Management
Treatment of cirrhosis includes
1). Preventing further damage to the liver.
2) .Treating the complications of cirrhosis.
3). Preventing liver cancer or detecting it early.
4). Liver transplantation.
1. Preventing further damage to the liver

Consume a balanced diet and one multivitamin daily. Patients with PBC
(Primary Biliary Cirrhosis) with impaired absorption of fat soluble vitamins
may need additional vitamins D and K.
Avoid drugs (including alcohol) that cause liver damage. All patients with
cirrhosis should avoid alcohol. Most patients with alcohol induced cirrhosis
experience an improvement in liver function with abstinence from alcohol.
Even patients with chronic hepatitis B and C can substantially reduce liver
damage and slow the progression towards cirrhosis with abstinence from
alcohol.
Avoid no steroidal anti-inflammatory drugs (NSAIDs, for example, ibuprofen).
Patients with cirrhosis can experience worsening of liver and kidney function
with NSAIDs.
Eradicate hepatitis B and hepatitis C virus by using anti-viral medications. Not
all patients with cirrhosis due to chronic viral hepatitis are candidates for drug
treatment. Some patients may experience serious deterioration in liver function
and/or intolerable side effects during treatment.
Suppress the immune system with drugs such as prednisone and azathioprine
(Imuran) to decrease inflammation of the liver in autoimmune hepatitis.
Immunize patients with cirrhosis against infection with hepatitis A and B to
prevent a serious deterioration in liver function. There are currently no
vaccines available for immunizing against hepatitis C.
2. Treating complications of Cirrhosis

Edema and ascites.
Retention of salt and water can lead to swelling of the ankles and legs
(edema) or abdomen (ascites) in patients with cirrhosis. Doctors often
advise patients with cirrhosis to restrict dietary salt (sodium) and fluid
to decrease edema and ascites. The amount of salt in the diet usually is
restricted to 2 grams per day and fluid to 1.2 liters per day. In most
patients with cirrhosis, however, salt and fluid restriction is not enough,
and diuretics have to be added.
Diuretics are medications that work in the kidneys to promote the
elimination of salt and water into the urine. A combination of the
diuretics spironolactone (Aldactone) and furosemide (Lasix) can reduce
or eliminate the edema and ascites in most patients. During treatment
with diuretics, it is important to monitor the function of the kidneys by
measuring blood levels of blood urea nitrogen (BUN) and creatinine to
determine if too much diuretic is being used. Too much diuretic can lead
to kidney dysfunction that is reflected in elevations of the BUN and
creatinine levels in the blood.
Sometimes, when the diuretics do not work (in which case the ascites is
said to be refractory), a long needle or catheter is used to draw out the
ascitic fluid directly from the abdomen, a procedure called abdominal
paracentesis. It is common to withdraw large amounts (liters) of fluid
from the abdomen when the ascites is causing painful abdominal
distension and/or difficulty breathing because it limits the movement of
the diaphragms.
Another treatment for refractory ascites is a procedure called
transjugular intravenous portosystemic shunting (TIPS, see below).
Bleeding from varices.

If large varices develop in the esophagus or upper stomach, patients
with cirrhosis are at risk for serious bleeding due to rupture of these varices.
Once varices have bleed, they tend to rebleed and the probability that a patient
will die from each bleeding episode is high . Therefore, treatment is necessary
to prevent the first (initial) bleeding episode as well as rebleeding.
Treatments include medications and procedures to decrease the
pressure in the portal vein, and procedures to destroy the varices.
Propranolol (Inderal)
A beta blocker is effective in lowering pressure in the portal vein
and is used to prevent initial bleeding and rebleeding from varices in
patients with cirrhosis.
Octreotide (Sandostatin)
Also decreases portal vein pressure and has been used to treat
variceal bleeding.
During upper endoscopy (EGD)
Sclerotherapy or band ligation can be performed to obliterate
varices and stop active bleeding and prevent rebleeding.
Transjugular intrahepatic portosystemic shunt (TIPS)
Is a non-surgical, radiolotic procedure to decrease the pressure
in the portal vein. TIPS are performed by a radiologist who inserts a
stent (tube) through a neck vein, down the inferior vena cava and into
the hepatic vein within the liver. The stent then is placed so that one end
is in the high pressure portal vein and the other end is in the low
pressure hepatic vein. This tube shunts blood around the liver and by so
doing lowers the pressure in the portal vein and varices and prevents
bleeding from the varices.
A surgical operation to create a shunt (passage)
From the high-pressure portal vein to veins with lower pressure
can lower blood flow and pressure in the portal vein and prevent varices
from bleeding.
Hepatic encephalopathy.
Patients with an abnormal sleep cycle, impaired thinking, odd behavior,
or other signs of hepatic encephalopathy usually should be treated with a low
protein diet and oral lactulose. Dietary protein is restricted because it is a
source of toxic compounds that cause hepatic encephalopathy. Lactulose,
which is a liquid, traps toxic compounds in the colon so they cannot be
absorbed into the blood stream, and causes encephalopathy. (Lactulose is a
laxative and the adequacy of treatment can be judged by loosening or
increasing frequency of stools).
Hypersplenism.
The filtration of blood by an enlarged spleen usually results in only mild
reductions of red blood cells (anemia), white blood cells (leukopenia) and
platelets (thrombocytopenia) that do not require treatment. Severe anemia,
however, may require blood transfusions or treatment with erythropoietin
hormone that stimulate the production of red blood cells.
No approved medication is available yet to increase the number of
platelets. As a necessary precaution, patients with low platelets should not use
aspirin or other no steroidal anti-inflammatory drugs (NSAIDS) since these
drugs can hinder the function of platelets. If a low number of platelets are
associated with significant bleeding, transfusions of platelets usually should be
given. Surgical removal of the spleen (splenectomy) should be avoided, if
possible, due to the risk of excessive bleeding during the operation.
Spontaneous bacterial peritonitis (SBP).
Patients suspected of having spontaneous bacterial peritonitis usually
will undergo paracentesis. Fluid that is removed is examined for white blood
cells and cultured for bacteria. Blood and urine samples also are often
obtained for culturing because many patients with spontaneous bacterial
peritonitis also will have infection in their blood and urine. In the infection may
have begun in the blood and the urine and spread to the ascitic fluid to cause
spontaneous bacterial peritonitis. Most patients with spontaneous bacterial
peritonitis are hospitalized and treated with intravenous antibiotics such as
cefotaxime.
In some patients oral antibiotics can be prescribed to prevent

spontaneous bacterial peritonitis. Not all patients with cirrhosis and ascites
should be treated with antibiotics to prevent spontaneous bacterial peritonitis,
but some patients are at high risk for developing spontaneous bacterial
peritonitis and warrant preventive treatment.
Patients with cirrhosis who are hospitalized for bleeding varices have a
high risk of developing spontaneous bacterial peritonitis and should be
started on antibiotics early during the hospitalization to prevent
spontaneous bacterial peritonitis.
Patients with recurring episodes of spontaneous bacterial peritonitis.
Patients with low protein levels in the ascitic fluid (Ascitic fluid with low
levels of protein is more likely to become infected.)
3. Prevention and early detection of liver cancer

Several types of liver disease that cause cirrhosis (such as hepatitis B
and C) are associated with a particularly high incidence of liver cancer. It
would be useful to screen for liver cancer in patients with cirrhosis, as early
surgical treatment or transplantation of the liver can cure the patient of
cancer. The difficulty is that the methods available for screening are only
partially effective, identifying at best only 50% of patients at a curable stage of
their cancer.
4. Liver transplantation
Cirrhosis is irreversible. Many patients' liver function will gradually
worsen despite treatment and complications of cirrhosis will increase and
become difficult to treat. Therefore, when cirrhosis is far advanced, liver
transplantation often is the only option for treatment. Recent advances in
surgical transplantation and medications to prevent infection and rejection of
the transplanted liver have greatly improved survival after transplantation.
5.7. Complications of Cirrhosis

The complications of cirrhosis are Edema and ascitis,
spontaneous bacterial peritonitis, Bleeding from esophageal vertices, Hepatic
encephalopathy, Hepatorenal syndrome, hepatopulmonary syndrome, Hypersplenism,
and Liver cancer
1.
Edema and ascites
As cirrhosis of the liver becomes severe, signals are sent to the kidneys to retain salt
and water in the body. The excess salt and water first accumulates in the tissue
beneath the skin of the ankles and legs because of the effect of gravity when standing
or sitting. This accumulation of fluid is called edema or pitting edema. As cirrhosis
worsens and more salt and water are retained, fluid also may accumulate in the
abdominal cavity between the abdominal wall and the abdominal organs. This
accumulation of fluid (called ascites) causes swelling of the abdomen, abdominal
discomfort, and increased weight.
2.
Spontaneous bacterial peritonitis (SBP)
Fluid in the abdominal cavity (ascites) is the perfect place for bacteria to grow.
Normally, the abdominal cavity contains a very small amount of fluid that is able to
resist infection well, and bacteria that enter the abdomen (usually from the intestine)
are killed or find their way into the portal vein and to the liver where they are killed.
In cirrhosis, the fluid that collects in the abdomen is unable to resist infection
normally. In addition, more bacteria find their way from the intestine into the ascites.
Therefore, infection within the abdomen and the ascites, referred to as spontaneous
bacterial peritonitis or SBP, is likely to occur. SBP is a life- threatening complication.
Some patients with SBP have no symptoms, while others have fever, chills, abdominal
pain and tenderness, diarrhea, and worsening ascites.
3.
Bleeding from esophageal varices
In the cirrhotic liver, the scar tissue blocks the flow of blood returning to the heart
from the intestines and raises the pressure in the portal vein (portal hypertension).
When pressure in the portal vein becomes high enough, it causes blood to flow around
the liver through veins with lower pressure to reach the heart. The most common veins
through which blood bypasses the liver are the veins lining the lower part of the
esophagus and the upper part of the stomach.
As a result of the increased flow of blood and the resulting increase in pressure, the
veins in the lower esophagus and upper stomach expand and then are referred to as
esophageal and gastric varices.
4.
Hepatic encephalopathy
Some of the protein in food that escapes digestion and absorption is used by bacteria
that are normally present in the intestine. While using the protein for their own
purposes, the bacteria make substances that they release into the intestine. These
substances then can be absorbed into the body. Some of these substances, for example,
ammonia, can have toxic effects on the brain. Ordinarily, these toxic substances are
carried from the intestine in the portal vein to the liver where they are removed from
the blood and detoxified.
When the toxic substances accumulate sufficiently in the blood, the function of the
brain is impaired, a condition called hepatic encephalopathy. Sleeping during the day
rather than at night (reversal of the normal sleep pattern) is among the earliest
symptoms of hepatic encephalopathy. Other symptoms include irritability, inability to
concentrate or perform calculations, loss of memory, confusion, or depressed levels of
consciousness. Ultimately, severe hepatic encephalopathy causes coma and death.
5.
Hepatorenal syndrome
Patients with worsening cirrhosis can develop hepatorenal syndrome. This syndrome
is a serious complication in which the function of the kidneys is reduced. It is a
functional problem in the kidneys, meaning there is no physical damage to the
kidneys. Instead, the reduced function is due to changes in the way the blood flows
through the kidneys themselves. The hepatorenal syndrome is defined as progressive
failure of the kidneys to clear substances from the blood and produce adequate
amounts of urine while other important functions of the kidney, such as retention of
salt, are maintained.
6.
Hepatopulmonary syndrome
Rarely, some patients with advanced cirrhosis can develop hepatopulmonary

syndrome. These patients can experience difficulty breathing because certain
hormones released in advanced cirrhosis cause the lungs to function abnormally.
7.
Hypersplenism
The spleen normally acts as a filter to remove older red blood cells, white blood cells,
and platelets (small particles that are important for the clotting of blood.). The blood
that drains from the spleen joins the blood in the portal vein from the intestines. As the
pressure in the portal vein rises in cirrhosis, it increasingly blocks the flow of blood
from the spleen. The blood "backs-up," accumulating in the spleen, and the spleen
swells in size, a condition referred to as splenomegaly. Sometimes, the spleen is so
enlarged that it causes abdominal pain.
8.
Liver cancer (hepatocellular carcinoma)
Cirrhosis due to any cause increases the risk of primary liver cancer (hepatocellular
carcinoma). Primary refers to the fact that the tumor originates in the liver. A
secondary liver cancer is one that originates elsewhere in the body and spreads
(metastasizes) to the liver.
The most common symptoms and signs of primary liver cancer are abdominal pain
and swelling, an enlarged liver, weight loss, and fever. In addition, liver cancers can
produce and release a number of substances, including ones that cause an increased
in red blood cell count (erythrocytosis), low blood sugar (hypoglycemia), and high
blood calcium (hypercalcemia ).
5.8. Prognosis of Cirrhosis

The overall prognosis in cirrhosis is poor. Many patients present with
advanced disease and/or serious complications that carry a high mortality.
Overall, only 25% of patients survive 5 years from diagnosis but, where liver
function is good, 50% survive for 5 years and 25% for up to 10 years.
The prognosis is more favorable when the underlying cause of the cirrhosis
can be corrected, as in alcohol misuse, haemochromatosis and Wilsons
disease.
Laboratory tests give only a rough guide to prognosis in individual patients.
Deteriorating liver function,as evidenced by jaundice, ascites or
encephalopathy, indicates a poor prognosis unless a treatable cause such as
infection is found.
Increasing bilirubin, falling albumin, marked hyponatraemia not due to
diuretic therapy, and a prolonged prothrombin time are all bad prognostic
features.
Section 6
Nursing Care of Patient with

Cirrhosis
7. Nursing Care of Patient with Cirrhosis

Nursing Priorities:1.
2.
3.
4.
Maintain adequate nutrition.

Prevent complications.
Enhance self-concept and acceptance of situation.
Provide information about disease process, prognosis, potential
complications, and treatment needs.
Discharge Goals:1.
2.
3.
4.
Nutritional intake adequate for individual needs.

Complications prevented or minimized.
Deals effectively with current reality.
Disease process, prognosis, potential complications, and
therapeutic regimen understood.
5. Plan in place to meet needs after discharge.
7.1.
Nursing Assessment
Monitor for signs and symptoms.
Fatigue
Weight loss, abdominal pain, and distention
Pruritus (severe itching of skin)
Confusion or difficulty thinking (due to the build-up of waste
products in the blood and brain that the liver is unable to get rid of).
Gastrointestinal bleeding (enlarged veins [varices] develop and
burst, causing vomiting and passing of blood in bowel movements)
Ascites (bloating or swelling due to fluid build-up in abdomen and
legs)
Jaundice (yellowing of skin) and icterus (yellowing of the eyes)
Petechiae (round, pinpoint, and red-purple lesions), ecchymosis
(large yellow and purple blue bruises), nose bleeds, hematemesis,
melena (decreased synthesis of prothrombin and deteriorating
hepatic function)
Palmar erythema (redness and warmth of the palms of the hands)
Spider angiomas (red lesions vascular in nature with branches
radiating onthe nose, cheeks, upper thorax, and shoulders)
Dependent peripheral edema of extremities and sacrum
Personality and mentation changes, emotional lability, euphoria,
and sometimes depression.
Asterixis (liver flapping tremor) is a coarse tremor characterized by
rapid,nonrhythmic extension and flexion of the wrists and fingers
Fetor hepaticus (liver breath) is a fruity or musty odor.
Assess/Monitor
ACTIVITY/REST
Weakness
Fatigue, exhaustion
CIRCULATION
History of or recent onset of heart failure (HF), pericarditis, rheumatic heart
disease, or cancer, causing liver impairment leading to failure
Easy bruising, nosebleeds, bleeding gums
ELIMINATION
Flatulence
Diarrhea or constipation
Gradual abdominal enlargement
FOOD/FLUID
Anorexia
Food intolerance, ingestion
Nausea, vomiting
Hematemesis
NEUROSENSORY
Significant other (SO)/family may report personality changes, depressed
mentation
PAIN/DISCOMFORT
Abdominal tenderness and right upper quandrant (RUQ) pain
Severe itching
Pins-and-needles sensation, burning pain in extremities (peripheral
neuropathy)
RESPIRATION
Dyspnea
SAFETY
Itching, dryness of the skin (pruritus)
SEXUALITY
Menstrual disorders (women)
Impotence (men)
TEACHING/LEARNING
History of long-term alcohol or injection drug use or abuse, alcoholic liver

disease, use of drugs affecting liver function
History of biliary system disease, hepatitis, exposure to toxins, liver trauma
DISCHARGE PLAN CONSIDERATIONS
May need assistance with self-care and other activities of daily living (ADLs),
homemaking and maintenance tasks
7.2.
Nursing Diagnosis
1. Imbalanced Nutrition: Less than Body Requirements

May be related to:
Inadequate diet; inability to process, digest nutrients
Anorexia, nausea, vomiting, indigestion, early satiety (ascites)
Abnormal bowel function
Possibly evidenced by:
Weight loss, Changes in bowel sounds and functions,
Poor muscle tone, muscle wasting; fatigue
Imbalances in nutritional studies
2. Excess Fluid Volume

May be related to:
Compromised regulatory mechanismsyndrome of inappropriate anti
diuretic hormone (SIADH), decreased plasma proteins, malnutrition,
Excess sodium and fluid intake
Edema, anasarca, weight gain
Intake greater than output, oliguria, changes in urine specific gravity
Dyspnea, adventitious breath sounds, pleural effusion
Blood pressure (BP) changes, altered central venous pressure (CVP)
JVD, positive hepatojugular reflex
Altered electrolyte levels
Change in mental status
3. Risk for impaired Skin Integrity
Risk factors may include:

Altered circulation and metabolic state
Accumulation of bile salts in skin
Poor skin turgor, skeletal prominence, presence of edema, ascites
4. Risk for ineffective Breathing Pattern

Intra-abdominal fluid collection (ascites)
Decreased lung expansion, accumulated secretions
Decreased energy, fatigue
5. Risk for Bleeding

Abnormal blood profile; altered clotting factorsdecreased production
of prothrombin, fibrinogen, and factors VIII, IX, and impaired vitamin K
absorption; and release of thromboplastin, Portal hypertension,
development of esophageal varices
6. Risk for acute Confusion

Alcohol abuse
Inability of liver to detoxify certain enzymes and drugs
7. Self-Esteem [specify]/disturbed Body Image

May be related to:
Biophysical changes, altered physical appearance
Uncertainty of prognosis, changes in role function
Personal vulnerability
Self-destructive behavioralcohol-induced disease
Verbalization of change or restriction in lifestyle
Fear of rejection or reaction by others

Negative feelings about body and abilities
Feelings of helplessness, hopelessness, or powerlessness
8. Deficient Knowledge [Learning Need] regarding condition, prognosis,

treatment, self-care, and discharge needs
May be related to:
Lack of exposure or recall; information misinterpretation
Unfamiliarity with information resources
Questions, request for information, statement of misconception
Inaccurate follow-through of instructions, development of preventable
complications
7.3.
Nursing Interventions
IDEAL
Vital Signs monitored every 4 hours.

Intake and Output monitored every hour.
Monitored and documented Nasogastric tubing output every hour.
Medication given as prescribed by the physician.
Facilitate completion of NPO diet required.
NGT patency checking prior to medication done
Assessment for any alterations in body comfort and report
immediately to the physician.
NGT feeding done and medication.
Assessment for any profuse gum bleeding and note for the color
discharge, include odor.
Education for the significance of medication given
Encouraging the client to do exercise at a minimal level to promote
circulation.
Lifestyle modification: weight reduction (body mass index [BMI]
goal <25), reduction of dietary sodium to less than 2.4 g/day, DASH
diet (i.e., diet high in fruits and vegetables, reduced saturated and
total fat), aerobic physical activity >30 minutes most days of the
week, tobacco avoidance, increased dietary potassium and calcium,
moderation of alcohol consumption.
Use of self BP monitoring. Home measurement device should be
checked regularly for accuracy. Mean self measured BP >135/85 is
generally considered to be hypertensive.
ACTUAL CARE GIVEN

Independent:
Assess for any significant findings on the abdominal size -to provide
a basis of proper and comfortable positioning
Assess for any discomfort related to pain at the right side of the
body- to provide a basis of proper and comfortable positioning.
Monitor intake and output closely (hourly)- to monitor any
improvement or worsening of patients condition
Regulate IVF to ordered flow rate- to prevent overload and under
load of fluid intake.
Provide side rails. - to promote patients safety
Encourage the client to urinate if feeling of voiding is present.- to
alleviate urinary distention
Educated the client and the SO about the significance of urination.to provide information about the significance of voiding in relation
to its underlying condition
Bedside care done-to promote comfort and safety of the clients
condition.
Position the patient in a Fowlers or Semi Fowlers position with
pillows - Relieves pressure on diaphragm. -Observe for
manifestations like crackles or increased respiration.- Identifies fluid
in the lungs
Monitor vital signs every 2 hours- to identify any changes in
patients health status.
Encourage the client to inhale and exhale exercise. - To alleviate
breathing difficulty.
Use light, cool clothing which promotes evaporation. Keep clothing
and bed dry. - Minimizes irritation and itching
Keeping the environment cool.- Minimizes itching
Avoid activities that promote sweating. Minimizes itching
Keep nails short and smooth.- Prevents breaking skin integrity when
scratching
Reposition patient every 2 hour.- Relieves pressure over bony
prominences
Dependent:
Medications were given as prescribed, lactulose 30 ml,
metronidazole 200mg 1 tab TID via NGT. To alleviate clients
condition as prescribed by the physician.- to promote wellness and
alleviate the existing problem.
Instructed the So to maintain Nothing Per Orem Diet (NPO) as
recommended given since Gastrointestinal function are impaired due
to abdominal distention.- to reduce gastric irritation.
Administer Oxygen as ordered. - To alleviate breathing difficulty and
assist the need of air by the client.
8. Summary and Conclusion
The significance of this study promulgates a comprehensive learning, skills and

responsibilities on the said case. It includes a thorough collaborative discussion and
interaction between me, as a student nurse and my client at the Medical Ward at
Hemas Hospital. Different nursing assessment and interventions, both ideal and
actual was presented in order to show a comparison and variability of each procedure
done. Not only on the nursing part was presented, a comprehensive medical and
diagnostic procedures was also compared, both actual and ideal to show the essence
of every care given. During the discussion of anatomy and physiology, and its
pathophysiology related to the condition, the case will thoroughly deviate from the
normal flow of the story and yet further analysis is required since no actual
Pathophysiology was thoroughly discussed to explain the theory presented. All the
essential data required are presented and tabularized in order to ease up the readers
upon reading.
The whole discussion will truly give innovations to the related education and field
studies and will somewhat aid the readers to enlighten their minds about Liver
Cirrhosis.
Thank you.
9. References
Saladin: Anatomy & Physiology: The Unity of Form and Function, Third
Edition, the McGrawHillCompanies
Marilynn E. Doenges, APRN, BC-Retired, Mary Frances Moorhouse, Alice C.
Murr (2010) Nursing Care Plans
Danielle Platt & Mary Moss, Adult Medical and Surgical Nursing
David A. Warrell (Editor), Timothy M. Cox (Editor), John D. Firth (Editor),
Edward J., J R., M.D. Benz, Oxford Textbook of Medicine 4th edition (March
2003), By Oxford Press.
Nicki R. Colledge, Brian R. Walker, Stuart H. Ralston,(2010), Davidsons
Principles and Practice of Medicine, An imprint of Elsevier Limited.
Sondra G. Ferguson, Tracey Goldsmith, Constance J. Hirnle, Carol Ann
Barnett Lammon, Sandra Smith Pennington, Frank Romanelli.(2006), The
Clinical drug Therapy Rationales for Nursing Practice.
Vital Signs Chart

Patient Name:
BHT No:
Ward/Room No:
Mr. R
30848
A
Date
Time
18/02/2013
On
admission
19/02/2013
20/02/2013
21/02/2013
22/02/2013
Temperature
Pulse
Respiration
BP
94.6 F/Axilla
99.2/Axilla
100.4/Axilla
98.6/Axilla
99.8/Axilla
80/bpm
86/bpm
88/bpm
94/bpm
86/bpm
22/bpm
24/bpm
26/bpm
24/bpm
22/bpm
130/70mmHg
10am
02pm
06pm
10pm
130/70 mmHg
02am
06am
10am
02pm
06pm
10pm
96.4/Axilla
95.4/Axilla
94.8/Axilla
96.4/Axilla
98.6/Axilla
80/bpm
84/bpm
82/bpm
82/bpm
86/bpm
22/bpm
24/bpm
28/bpm
26/bpm
24/bpm
120/85 mmHg
130/80 mmHg
120/80 mmHg
02am
06am
10am
02pm
06pm
10pm
98.9/Axilla
99.1/Axilla
98.2/Axilla
98.6/Axilla
92.4/Axilla
98.2/Axilla
84/bpm
88/bpm
84/bpm
86/bpm
82/bpm
80/bpm
22/bpm
26/bpm
24/bpm
20/bpm
24/bpm
20/bpm
02am
06am
10am
02pm
06pm
10pm
92.4/Axilla
98.6/Axilla
96.6/Axilla
98.4/Axilla
97.8/Axilla
88/bpm
94/bpm
84/bpm
86/bpm
82/bpm
22/bpm
26/bpm
24/bpm
20/bpm
24/bpm
02am
06am
10am
02pm
06pm
10pm
98.8/Axilla
98.9/Axilla
101/Axilla
99/Axilla
98.2/Axilla
84/bpm
80/bpm
88/bpm
86/bpm
82/bpm
24/bpm
26/bpm
26/bpm
24/bpm
20/bpm
Remarks
120/80mmHg
130/80 mmHg
120/80 mmHg
96% on air
98% on air
130/80 mmHg
135/65 mmHg
99% on air
130/90 mmHg
120/80 mmHg
120/85 mmHg
99% with O2
98% with O2
99% with O2
96%with O2
120/80 mmHg
130/80 mmHg
140/70 mmHg
98% on air
96% on air
Intake and Output Chart

Patient Name:
BHT No:
Ward/Room No:
Date
Mr. R
30848
A
Time
Oral
IV Fluids
NG Feed
Total
Urine
Other
Total
18/2/2014 1pm-7pm
120ml 300ml
420ml
200ml
200ml
7pm-7am
140ml 600ml
1260ml 250ml
450ml
Total intake :1260ml

Total output:450ml
19/2/2014 Time
Oral
IV Fluids
NG Feed
Total
Urine
Other
Total
7am-1pm
180ml 300ml
480ml
150ml
150ml
1pm-7pm
200ml 300ml
980ml
170ml
330ml
7pm-7am
300ml 600ml
1880ml 200ml
530ml

Total output:530ml
20/2/2014 Time
Oral
IV Fluids
NG Feed
Total
Urine
Other
Total
7am-1pm
250ml 300ml
550ml
180ml
180ml
1pm-7pm
200ml 300ml
1050ml 200ml
380ml
7pm-7am
200ml 600ml
2050ml 300ml
680ml

Total output:680ml
200ml
Intake and Output Chart

Patient Name:
BHT No:
Ward/Room No:
Mr. R
30848
A
21/2/2014 Time
Oral
IV Fluids
NG Feed
Total
Urine
Other
Total
7am-1pm
100ml 120ml
300ml
420ml
200ml
200ml
1pm-7pm
50ml
120ml
300ml
890ml
250ml
450ml
240ml
200ml
1330ml 270ml
720ml
NG Feed
Total
Urine
7pm-7am

Total output:720ml
22/2/2014 Time
Oral
IV Fluids
Other
Total
7am-1pm
100ml 120ml
200ml
420ml
350ml
350ml
1pm-7pm
100ml 120ml
200ml
840ml
300ml
650ml
7pm-7am
50ml
200ml
1330ml 350ml
240ml

Total output:1000ml
1000ml
Diabetic Chart
Patient Name:
BHT No:
Ward/Room No:
Mr. R
30848
A
Date
Time
RBS Value
Medication
18/02/2014
6am
180 mg/dl
S. Insulin 15units SC given
19/02/2014
6am
125 mg/dl
12nn
110 mg/dl
6pm
117 mg/dl
6am
132 mg/dl
12nn
128 mg/dl
6pm
118 mg/dl
6am
96 mg/dl
12nn
84 mg/dl
6pm
70 mg/dl
6am
90 mg/dl
12nn
84 mg/dl
6pm
99 mg/dl
6am
120 mg/dl
20/02/2014
21/02/2014
22/02/2014
23/02/2014
Investigations
N
o
Investigation Name
1.
2.
Normal
Value
18/02/
2013
Full Blood Count

WBC
Nutrophills
Lymphocytes
Monocytes
Eosinophills
Basophills
RBC
HGB
PCV
MCV
MCH
MCHC
RDW
Platelet Count
4000-11000 cumm
40-75%
20-40%
2-8%
1-6%
0-3%
4-6%
11.5-15.5g/dl
36-46%
83-101FL
27.5-32Pg
31.5-35g/dl
11.6-14.8%
150,000-450,000cumm
8000 cumm
68%
20%
6%
4%
2%
6%
12.7g/dl
40%
88.1FL
32.4Pg
24.5g/dl
10.9%
198,0000cumm
Serum Electrolytes
Sodium (Na+)
Potassium (K+)
Chloride (Cl-)
137-145mmol/L
3.5-5.1mmol/L
98-105mmol/L
19/02/
2013
20/02/
2013
21/02/
2013
6500 cumm
57%
38%
6%
5%
0%
4.25%
12.8g/dl
37.5%
88.1FL
29.8Pg
33.8g/dl
14.9%
257,000cumm
141 mmol/L
3.5 mmol/L
99 mmol/L
22/02/
2013
7800 cumm
64%
38%
6%
0%
0%
2.95%
10.4g/dl
30.5%
10.3FL
35.2Pg
34.1g/dl
18.8%
234,000cumm
136 mmol/L
3.2 mmol/L
98 mmol/L
3.
Serum Creatinine
Male 0.5-1.5mg/dl
Female 0.6-1.2mg/dl
1.1mg/dl
0.9mg/dl
1.1mg/dl
4.
CRP
0-6 mg/L
48.5mg/L
42.5mg/L
34.2mg/L
5.
Liver Profile
Total Protein
Albumin
Globulin
A/G Ratio
Total Bilirubin
Alkaline Phosphatese
ALT/SGPT
AST/SGOT
GAMMA GT
6.4-8.2g/dl
3.4-5.0g/dl
2.5-3.5g/dl
1:1
0.2-1.2g/dl
50-136U/L
30-65U/L
15-37U/L
15-85U/L
7.7 g/dl
3.1 g/dl
5.2 g/dl
1.1
10.58 g/dl
75 U/L
32 U/L
18 U/L
49 U/L
7.6 g/dl
2.5 g/dl
5.1 g/dl
0.5
13.78 g/dl
122 U/L
33 U/L
60 U/L
40 U/L
7.8 g/dl
2.2 g/dl
5.8 g/dl
1.1
10.2 g/dl
120 U/L
34 U/L
54 U/L
76 U/L
Renal Profile
Sodium
Potassium
Chloride
Urea
S. Creatinine
Calcium
Phosphorus
Uric acid
137-145mmol/L
3.5-5.1mmol/L
98-105mmol/L
5-40mg/dl
0.5-1.5mg.dl
8.5-10.1mg/dl
2.5-4.5mg/dl
3.5-8.35mg/dl
6.
134 mmol/L
4.7 mmol/L
96 mmol/L
23.54 mg/dl
0.8 mg/dl
8.9 mg/dl
3.3 mg/dl
2.8 mg/dl
28.4mg/L
7.2 g/dl
3.0 g/dl
5.4 g/dl
0.5
9.4 g/dl
122 U/L
38 U/L
42 U/L
80 U/L
7.
ESR
8.
Lipid Profile
Total Cholesterol
Triglyceride
HDL
LDL
VLDL
CHOL/HDL ratio
9.
Prothrombin Time
Control
INR
0-20mm (1st Hour)
28mm
>200mg/dl
>150mg/dl
>40mg/dl
>129mg/dl
212 mg/dl
132 mg/dl
43 mg/dl
142.6 mg/dl
26.40
4.9
39seconds
13seconds
3.1
Ultra Sound Scan Abdomen are Normal

Chest X ray, CT Scan Reports are Normal View.
24mm
22mm
215 mg/dl
126 mg/dl
56 mg/dl
140 mg/dl
25.5
4.7
36seconds
15seconds
2.8
Medication Chart
No
Drug Name
1.
Generic Name
Route
Action & Indications
Levofloxacin
Oral
Drugs
Levofloxacin
Levaquin
Classification
Antibiotic
(Fluoroquinolone)
Side Effects
Nursing Considerations
Action
IV
Trade Name
Contra Indications
Involve the inhabitation

of bacterial action and
fights bacterial in the
body.
Indications
Bacterial
infection of the
Skin, Sinuses,
Kidneys, Liver,
Bladder or
Prostate.
Bronchitis
Pneumonia
Anthrax or plaque
Hypersensitivity
of Levofloxacin
Pregnancy
Breast feeding
Diarrhea
Abdominal
pain/cramps
Agitation
Confusion
Fever
Redness &
Swelling of
skin
Burning on
the skin
Skin rash
itching
Hypersensitivity of
Levofloxacin
History of muscle
disorders
(myasthenia
gravis)
No
2.
Drug Name
Generic Name
Route
Indications
Trade Name
Classification
Proton Pump
inhibitor
Side Effects
Action
Oral
IV Drug Suppress gastric acid
Pantaprazole
production
Pantacid
Pantodac
Contra Indications
Sahort term
treatment of
erosive
oesophagitis
associated with
GERD
Duodenal Ulcer
Prophylaxis of
NSAID associated
gastric or
duodenal ulcer
Hypersensitivity
of Pantaprazole.
Allergic
reactions
Constipation
Dry mouth
myalgia
Thrombocyt
openia
Generalized
oedema
Depression
Vertigo
pruritis
Assess for side

effects.
Educate patient
about side effects.
Drug Name
3.
Route
Generic Name
Metranidazol Oral
drug,
e
Injec,
Classification
Antimicrobial
Drug
Side Effects
Action
High action against
anaerobic bacteria and
protozoa to killing
Trade Name
Flagyle
Metranidazol
e
Contra Indications
Indications
Anaerobic
infection
Leg ulcers &
Pressure sores
Bacterial
vaginosis
Pelvic
inflammatory
disease
Acute ulcerative
gingivitis
Acute oral
infections
Surgical
prophylaxis
Hepatic
impairment
Hepatic
encephalopathy
Pregnancy
Breast feeding
Nausea,
Vomiting
Taste
disturbance
Oral
mucositis
Drowsiness
Dizziness
Headache
Ataxia
Psychotic
disorders
Thrombocyt
openia
Myalgia
Visual
disturbance
Pruritis
Erythema
Assess for contra

indications
Educate patient
about side effects
Drug Name
4
Route
Generic Name
Metformin
Hydrochlorid
e
Classification
Biguanides
Side Effects
Action
Oral
Drugs
It exerts its effect mainly

decreasing
gluconeogenesis and by
increasing peripheral
utilization of glucose.
Trade Name
Glycomet
Metformin
Glymet
Contra Indications
Indications
Diabetes Mellitus
Poly Cystic Ovary
syndrome
Renal impairment
Ketoacidosis
Sepsis
Respiratory
failure
Hepatic
impairment
Pregnancy
Breast feeding
Anorexia
Nausea,
vomiting
Diarrhea
Abdominal
pain
Metallic
taste
Lactic
acidosis
Erythema
Pruritis
Urticaria
Decrease Vit
B12
absorption
Assess the contra

indication before
administering
drug
Educate patient
about side effect
Assess Blood
Glucose level for
continuously
taking patients.
Drug Name
5.
Route
Generic Name
Frusemide
Trade Name
Lasix
Frusemide
Side Effects
Action
Oral,
inje
Loop diuretics inhibits

reabsorption from the
ascending limb of the
loop of Henle in the
renal tubule and are
powerful diuretic
Indications
Classification
Loop Diuretic
Contra Indications
Oedema
Oliguria due to
renal failure
Pulmonary
oedema
Chronic heart
failure
Liver cirrhosis
Renal failure
Anuria
Hyponatrem
ia
Hypokalemi
a
Hypomagnes
imia
Hypochlorae
mic
alkalosia
Increase
calcium
exertion
Hypotension
GI
disturbance
Hyperglyce
mia
pancreatitis
Administer in
night
Educate patient
about polyuria
Drug Name
6.
Route
Generic Name
Spiranolacto
ne
Trade Name
Classification
Potassium
sparing
Diuretics
Aldosterone
Antagonists
Side Effects
Action
Oral
drug
Antagonizing the
Aldosterone
Indications
Aldactone
Spiranolacto
ne
Contra Indications
Oedema
Ascitis in cirrhosis
Malignant
Cirrhosis
Nephritic
Syndrome
CHF
Primary
hyperaldoesteroni
sm
Hyperglycemia
Hyponatremia
Addisons disease
GI
disturbances
Impotence
Gynaecomes
tia
Menstrual
irregulation
s
Lethargy
Headache
Confusion
Rashes
Hyperkalemi
a
Hyponatrem
ia
Osteomalaci
a
As with potassium
sparing diuretics,
potassium
supplements must
not be given with
aldosterone
antagonists.
Monitor serum
Electrolyte level.
Assess for GI
disturbances.
Drug Name
7.
Generic Name
Tolbutamide
Trade Name
Route
Oral
drug
The act by increasing

insulin release from the
beta cells in the
pancrease
Indications
Sulphonylure
as
Side Effects
Action
Tolbutamide
Classification
Contra Indications
Type 2 Diabetes
Mellitus
Hepatic & Renal

impairment
Prophyria
Breast feeding
Ketoacidosis
Headache
Tinnitus
Nausea,
Vomiting
Diarrhea
Hypoglycemi
a
Fever
Jaundice
Photosensiti
vity
Thrombocyt
openia
Agranulocyt
osis
Anemia
Assess patient for

hypoglycemia
Before use of this
drug patient
assess for RBS
Drug Name
8.
Route
Generic Name
Atorvastatin
Trade Name
Atorva
Atacor
Atrovastatin
Classification
Statin
Contra Indications
Side Effects
Action
Oral
drug
Statins are lowering and

regulating LDL
cholesterol
concentration
Indications
Primary hyper
cholesterolaemia
Heterozygous
familial hyper
cholesterolaemia
Homozygous
familial hyper
cholesterolaemia
Prevention of
cardiovascular
events in patient
with Type 2 DM.
Pregnancy
Breast Feeding
Chest pain
Angina
Insomnia
Dizziness
Hypoaesthes
ia
Arthralgia
Back pain
Headache
Altered liver
function test
Abdominal
pain
Flatulence
Constipation
Nausea &
vomiting
Hypersensiti
ve reaction.
Assess for liver

function
Encourage patient
to take in night
time.
Educate patient
about side effects.
Care Plan of the Patient
Nursing Assessment
20/02/2013,
Wednesday, 8.30am.
Subjective Data
Mr W A P Ranjith,
58years
Verbalized I have
1. Abdominal Distention
and Generalized
swelling of Abdomen
and Scrotum since two
weeks
2. Abdominal Pain
Right Upper Quadrent
site, On & Off type pain,
Pain on Exertion, and No
Radiation in other site.
Nursing diagnosis
Goal
1. Pain related to
Abdominal Distention
Reduce the
Pain
Planning
Nursing Intervention
1. Assess the Pain noting

location, Characteristics,
intensity and Radiation.
1. Assessed pain for location,

characteristic, intensity, and
radiation.
2. Position the patient.
2. Positionate the patient for

Semi Fowlers position.
3. Provide comfort
measures such as mouth
care, back care and
repositioning.
4. Encourage the patient to
use of the relaxation
technique.
5. Monitor Vital Signs.
6. Provide Diversitional
Activities.
3. Loss of Appetite sine

one Week
7. Administer Analgesics as
Prescribed.
4. Vomiting 4 times its

watery contents.
8. Administer IV fluids as
Prescribed.
3. Provide comfort measures

such as mouth care and
Repositioning.
4. Educated patient for some
relaxation techniques.
5. Monitored and charted the
Vital Signs.
6. Provided some Divertional
Activities such as TV, Radio,
and Talked with patient.
7. Introduced Hospital
Environment, and ward
Staff.
Evaluation
After Nursing
interventions
patient
verbalized feel
comfortable.
9. Monitor Intake & Output

Chart.
5. Decreased level of
Urine output since 3
days
9. Administer Analgesics As
Prescribed.
Morphine SC
Objective Data
1. Patient General
Appearance is good.
10. Administered the

Intravenous Fluid as
Prescribed.
Hartman 50cc/hr.
2. Patient oriented and

Alert for Time, Person,
and Place.
3. Patient Vision Normal
for L 6/6, R 6/6
4. Skin color is Yellow
color and Poor skin
Turgor.
5. Extremities are Warm.
6. Patient not complain
for Oedema and
Dysponea
7. CRFT <2 seconds
8. Patient's Abdomen is
Distended and
8. Monitored and Charted

the intake and output.
2. Risk for ineffective

breathing pattern
related to intra
abdominal fluid
collection.
Maintain
Effective
breathing
Pattern
1. Monitor Respiration rate

depth and effort.
1. Monitored Respiratory
rate, depth and effort.
2. Auscultate breathe sound

and Crackles.
2. Auscultated the breathing

sound and crackles sound.
3. Monitored and charted

vital signs.
4. Keep patient head of bed

elevated position client on
side.
4. Kept patient head elevated

position.
5. Encourage patient
frequent repositioning.
6. Encourage patient to
deep breathing and
coughing exercises.
5. Encouraged patient for

repositioning.
6. Educated the patient for
Deep breathing and
coughing exercises.
After Nursing
interventions
patient
breathing
pattern is normal
Tenderness when
palpating abdomen.
9. abdominal Girth of the
Patient is 60cm
10. Decreased Urinary
output, today 150ml in
7am to 1pm.
7. Assess the patient

coughing and coughing out
secretion.
8. Introduce the
Physiotherapist for chest
exercises as prescribed.
7. Assessed patient coughing

Secretion its light yellow
color.
9. Monitor ABG and SPO2

level if needed.
9. Prepared and send for the

patient for the Radiology
Department for Paracentesis
Procedure.
10 Administer Supplement
O2 therapy.
11. Patient in R side 18G

IV Cannula and cannula
site normal.
8. Administered oxygen via

the face mask.
11. Prepared patient for the

Paracentesis Procedure.
12. Patient Weight is

78kg.
13. Vital Signs
Temperature 98.2
F/Axi
Pulse 84/bpm
Respiration 24/bpm
BP 120/80mmHg
SPO2 98% on air
14. Blood Investigations
Results.
* ESR 24mm (1st Hour)
* Full Blood Count
3. Imbalanced
Nutrition less than
body Requirements
related to Anorexia,
Nausea and Vomiting.
Maintain
normal
Nutrition
level
1. Assess and Evaluate

client's risk for
malnutrition.
2. Assess patient like and
dislike food and drink.
3. Administer patient like
food, in small amount
frequent interval.
increase oral intake.
1. Assessed and Evaluate

client's risk for malnutrition.
2. Administered patient like
food, in small amount
frequent interval.
3. Encouraged patient to
4. Advised the patient for
limit the high salt food as
canned soups and
vegetables.
After nursing
patent get
normal diet
WBC 6500cumm
RBC 4.25%
Hb% 12.8g/dl
PCV 37.5%
Plt Count 257,000
cumm
* Renal Profile
Na+ 134mmol/L
K+4.7mmol/L
Cl- 96mmol/L
Urea 23.54mg/dl
S. Creatinine 0.8mg/dl
Ca + 8.9mg/dl
Uric acid 2.8mg/dl
5. Assess and encourage

client eat, explain reasons
for the types of diet.
6. Advice the patient for
limit the high salt food as
canned soups and
vegetables.
7. Restrict intake of caffeine
and gas producing or spicy
and excessive hot or cold
foods.
8. Encourage and provide
frequent mouth care
especially before meals.
9. Administer Nutritional
Supplements as prescribed.
Prescribed.
12. Maintain Intake and
output.
5. Restricted intake of
caffeine and gas producing
or spicy and excessive hot or
cold foods.
6. Provided mouth care for
before meals.
7. Administered Nutritional
Foods such as Soup, juice
and Milk.
8. Administered Iv fluids
Hartmann 50cc/hr as
prescribed.
9. Monitor and Charted Vital
Signs.
10. Monitor and charted
intake and output chart.
4. Risk for fluid

volume deficit related
to vomiting and less
Urine Output
Maintain
normal
optimal
body fluids
1. Assess patient fluid status

and skin turgor.
2. Monitor intake and
output chart.
3. Daily weight measuring
and compare periodic
weight, as needed.
prescribed.
5. Assess and Record Vital
Signs.
6. Assess Skin color, Mucous
Membrane and CRFT.
7. Check the patient
Abdomen for Ascitis,
Oedema formation and
Measure Abdominal girth
as needed.
1. Assessed patient for fluid

status and skin turgor its
poor skin turgor.
the intake and output chart.
3. Prescribed IV fluid
administered in 50cc/hr.
Vital Signs.
5. Assessed patient CRFT <2
seconds.
6. Assessed patient ascitis
and Abdominal girth after
Paracentesis 40cm.
increase oral intake as
frequently small interval.
Ambulate and try passing
urine.
After Nursing
interventions
reduced vomiting
and patient have
normal Vital
Signs
frequently try to passing
urine.
10. Educate warn the
patient for risk of fluid
collection in the body and
its complications.
9. Administered Prescribed
medication, such as Antacid
(Pantocid 40mg), Diuretics
( Lasix 40mg, &
Spironolactone).
11. Administer Medication

as Prescribed, such as
Antiemetic, Antacid, and
Diuretics.
5. Risk for impaired

skin integrity related
to Poor skin turgor
and Accumulation of
bile in the Skin it
Evidenced by yellow
color skin
Maintain
skin
integrity in
normal
level
1. Assess patient skin color

and skin turgor.
2. Inspect patient skin
surface and pressure points
routinely.
3. Gently massage bony
prominences or areas and
Pressure point areas.
4. Provide bed bad and use
emollient lotion and limit
use of soap bathing.
1. Assessed patient skin color Maintained

and skin turgor.
normal skin
turgor
2. Inspected patient skin
surface and pressure points
routinely for bedsores.
3. Administered pressure
point massages.
4. Administer emollient
lotion is back, thigh and
ankle such as baby cream
and Vaseline.
5. Administer Morning and

Evening care to maintain
normal level of skin.
5. Provide morning and

evening care for maintain
normal skin care and
provide comfort.
regular schedule while on
bed or chair and active or
passive range of motion
exercises.

active and passive range of
motion.
7. Elevate the edematous

lower part if patient feel
comfort.
8. Keep linen dry and free of
wrinkles.
9. Position change the
patient 4 hourly as needed.
maintain Personal Hygiene
and perineal care following
urination and bowel
opening.
7. Elevated patient
edematous leg part to
reduce edema.
8. Changed bed linen, kept
linen dry and free from
wrinkles.
9. Frequently change
position for prevent bed
sores.
personal hygiene and
perineal care following
urination and bowel
opening.
Nursing Assessment
21/02/2013 7.30am
Thursday
Subject Data
Mr. W A P Ranjith
Verbalized I have
1. Vomiting 3times in
morning its red color
with mixed watery.
Nursing Diagnosis
1. Risk for bleeding
related to development
of esophageal varices
it evidenced by
vomiting with blood.
Goal
Planning
Maintain
1. Assess for signs and
homeostasis
symptoms of GI bleeding.
with absence of
GI bleeding
2. Reassure the patient &
assess Vomitus for blood
stain.
1. Assessed for signs and

symptoms of GI bleeding.
2. Reassured the patient and
assed vomitus slight blood
stain in vomitus.
3. Provide psychology
support.
3. Position the patient in semi

fowlers position.
4. Position the patient.
4. Assessed & Charted Vital

signs.
2. Difficulty breathing
since morning 6am
5. Assess & Monitor the

Vital Signs.
3. Abdominal distention
and Discomfort
6. Assess for level of

Consciousness.
4. B/L Legs below Knee

Oedema
7. Assess for patient Full

Blood Count report as
prescribed.
5. Assessed patient LOC for

patient Conscious and
Rationale.
6. Assessed patient Blood
tests such as S. creatinine,
CRP, LFT, PT/INR, ESR, Lipid
Profile, and FBS.

increase oral intake and
avoid irritable food in mouth.
9. Educate patient for

avoid irritable food in

Evaluation
After nursing
intervention
reduced
vomiting
mouth.
Objective Data
10. Monitor & Maintain
1. Patient General
Appearance ill looking.
11. Administer IV fluid as

prescribed.
2. Patient Restlessness.
3. Skin color is yellow
color and poor skin
turgor
12. Administer
medication as prescribed
such as Antiemetic &
Vitamins.
9. Administered prescribed IV
Fluid in 20ml/hr.
stat dose Antiemetic
Doperidone 10mg stat.
11. Administered prescribed
stool softeners Lactulose 30cc
tds.
4. patient Weight 78kg

13. Administer stool
softeners to reduce
bleeding with rectum.
5. Patient Vision ability

normal R 6/6, L 6/6.
6. Patient on 18G IV
Cannula in R hand,
cannula site normal, no
signs of infection.
7. IV fluid progress in
20ml/hr
8. Patient in Difficulty in
Breathing, chest depth is
increase.
9. Abdomen distended ,
Abdominal girth 60cm
2. Excess fluid volume

related to
accumulation of fluid
in the body, evidenced
by B/L leg edema and
decreased urine
output.
Maintain
optimal body
fluid.
1. Assess patient for signs

of fluid overload.
2. Elevate the edematous
part.
3. Monitor Vital Signs 4
hourly.
4. Measure the patient
Weight daily as needed.
5. Assess for urinary
catheter patency and
1. Assessed patient for signs

of fluid over load as ascitis,
and Leg edema.
2. Elevated patient
edematous part such as both
legs to reduce edema.
3. Monitor and Charted Vital
signs.
4. Assessed patient urinary
catheter patency, no signs of
Slightly leg
Edema
reduced
urine flow.
heamaturia and infection.
6. Assess NG tube
position and administer
Fluid prescribed time
interval.
5. Assessed NG tube position

and Administered liquid food
prescribed interval.
take rest.
6. Encouraged patient to take

rest.

Exercise of Extremities.
7. Educated patient for Leg

Exercises.
9. Monitor Serum
Electrolyte level as
needed
8. Avoided sodium and

potassium contain foods.
10. CRFT < 2 Seconds

11. Patient on NG tube is
inserted yesterday night.
12. Patient on Urinary
Catheter is inserted
yesterday night.
13. Urinary catheter
normally drains and
urine color is dark color
and odor.
14. Patient
Psychologically confused
and worried about his
disease.
15. Vital Signs
Temper 98.6 F/Axilla
Pulse 90/bpm
Resp 26/bpm
BP 130/90mmHg
SpO2 98% with O2
16. Blood Investigation
Results
* S. Creatinine 1.1mg/dl
9. Administered salt free diet.

Avoid & Restrict Sodium
and Potassium contain
diet as indicated.
11. Administer salt free
diet and juice.
12. Administer Diuretic
as prescribed.

Diuretic.
*Frusimide 40mg bd
*Spironolactone mane
* CRP 34.2mg/L
3. Altered breathing
pattern related to
decreased lung
expansion and
accumulated secretion
it shows defaulting
Maintain
normal
breathing
pattern
1. Reassure the patient.
1. Reassured the patient.
2. Provide psychological
support.
2. Provided psychological
support and talked with
patient friendly.
3. Position the patient in semi
fowlers position to reduce
difficulty breathing and
abdominal distention.
3. Position the patient in

semi fowlers position.
4. Administer Oxygen as
needed.
5. Monitor Vital signs
especially patient
respiratory rate and
depth.
4. Administered Oxygen via

the face mask.
vital signs.
6. Assess patient
respiratory pattern for
using accessory muscle
for respiration.
6. Assessed Respiratory rate,

depth and pattern for using
accessory muscle for
breathing.
7. Maintain a calm
attitude environment.
7. Arranged calm and quiet

environment.
8. Encourage patient
deep breathing exercises.

deep breathing exercises.
9. Encourage patient for

express feeling.
10. Administer
Nebulization as

express feelings.
10. Administer Prescribed
medications.
After
Nursing
interventions
patient
breathing
pattern is
normal it
shown
normal
respiratory
rate and
depth.
prescribed.
11. Administer
Medication as prescribed.
4. Disturbed body
image related to
altered physical
appearance.
Understanding
changes &
acceptance of
self in the
present
situation.
support.
2. Provided the psychological

support to the patient.
3. Discuss with patient

situation and encourage
verbalization of fears and
concerns.
3. Discussed with patient

situation and encouraged
verbalized of fears and
concerns.
4. Explain relationship
between nature of
disease and symptoms.
4. Explained relationship
between nature of disease
and symptoms.
5. Support and encourage 5. Supported and Encouraged

client, provide care with
client, provided care with a
a positive friendly
positive friendly attitude.
attitude.
6. Encouraged and Explained
6. Encourage relation to
family members to
understanding patient
understanding patient
situation and participate situation and participate
in care.
patient care.
7. Assist client to cope
8. Assessed client to cope with
Patient
normally
adjusted his
condition.
with change in
appearance, suggest
suitable clothing.
8. Introduce counselor
for Divert patient
worried mind.
9. Keep and observation
of patient in out of bed.
10. Educate the patient
about effect of Alcohol
consumption.
5. Acute confusion
related to disease
condition.
Maintain usual
level of
Consciousness
1. Observe patient for

changes in behavior,
drowsiness, slowing or
slurring speech and
confusion.
support and talk with
friendly.
3. Keep the patient rest
and evaluate sleep and
rest schedule.
4. Maintain a pleasant,
changes in appearance,
suggested suitable clothing.
9. Introduced Psychological
Counselor to divert patient
and family worried mind.
10. Kept and Observed
patient out of bed.
11. Educated patient for
effect of Alcohol and Smoking
consumption.
1. Observed patient for

behavioral changes, patient
in drowsy and slight
restlessness.
support and talked with
friendly.
3. Kept patient rest and
scheduled sleep time.
4. Maintained a pleasant,
quiet environment and
Patient
diverted in
his disease
condition
and satisfied
his nature of
disease.
quiet environment and

approach slow, calm
manner.
5. Discuss with patient in
current situation and
future expectation of
disease and treatment
method.
approach slow, calm manner.

5. Discussed patient current
situation and future
expectation of disease and
treatment method.
6. Provided Safety measures,
such as bed in low position,
and pt side rails every time.
6. Identify and provide

for safety needs, such as
bed in low position and
put side rails.

vital signs.
7. Monitor vital signs.
8. Administered IV fluids and

Nutritional Food
supplements.
8. Administer IV fluids
and Nutritional food
supplements.
9. Provide continuity of
care for morning care,
evening care and mouth
care.
9. Provided continuity care of

Morning, Evening and Mouth
Care.
10. Administered Medication
in correct interval.
express feelings.
Assessment
Nursing Diagnosis
Goal
Planning
22/02/2013,
02.00pm
1. Hyperthermia
related to infective
process
Maintain
normal body
temperature
1. Monitor QHT
1. Monitored and maintained

the QHT chart.
Subjective Data
2. Assess the patient for

chills and diaphoresis.
Mr. Ranjith verbalized
3. Monitor Vital signs.
1. Fever since morning
4. Monitor and Adjust the

room temperature .
2. Nausea
3. Tiredness due to
3days
4. Loss of Appetite due
to 10 days
5. Apply Tepid sponge

bath if needed.
6. Provide cooling blanket
as needed.
7. Administer Antipyretic
as prescribed.
Objective Data
1. Patient general
appearance is lethargy
and weakness.
2. Patient Conscious,
Rationale, and Alert to
2. Assessed patient chills and

diaphoresis.
3. Monitor and charted Vital
signs.
4. Adjusted the room
temperature in 67'C
Antipyretic.
Paracetamol 1g
Antibiotic.
IV Levofloxacin 500mg
8. Administer Antibiotic as
prescribed.
7. Administered Slight cool
orange juice.
9. Administer Cool drink.
8. Raised patient bed rails to
10. Raise the bed side rails prevent falling.
of all time.
Evaluation
After nursing
interventions
patient body
temperature
was reduced
in
98.5F/axilla
Time, Person, and

Place.
11. Monitor intake and

output chart.
3. Patient vision ability

normal.
4. Skin color is normal,
no itching and rashes.
9. Monitor and charted the


prescribed.

IV fluids.
13. Provide high calorie

diet as needed.

signs of Hypothermia.
5. No Allergies.
for signs for Hypothermia.
6.Patient sad mood and

worried about his
disease condition.
7. No respiratory
problem in patient.
8. Reduced extremities
edema.
9. CRFT <2 seconds.
10. Nails and
Extremities normal.
11. Peripheries are
warm.
12. Patient in 18G IV
cannula in L hand.
2. Imbalanced
Nutrition less than
body requirement
related to Loss of
appetite and Nausea
Maintain
normal body
nutrition level
1. Assess the patient for

like and dislike.
1. Assessed the patient like

and dislike food.
2. Administer the like food

or drink the patient.
2. Administered fluid via the

NG tube 3 hourly such as drink
and soup.
3. Administer the fluid

drink 3 hourly.
3. Educated the patient about

nutritional supplement.
4. Administer Nutritional
supplements and IV fluid
as prescribed.
prescribed.
5. Maintain intake and

output chart.
5. Maintained intake and

output chart.
6. Educated the family about
Patient
satisfied
about
nursing care
about nutritional Diabetic importance nutrition.

diet.
6. Assessed and charted vital
7. Educate the family
signs.
members importance of
nutrition.
7. Educated the patient
simple relaxation techniques.
8. Assess the vital signs.
13. Paracentesis
procedure done on
yesterday night, 500ml
peritoneal fluid
removed.
14. Patient feel
comfortably of without
abdominal distention.
Abdominal girth 45cm
15. Urinary Catheter
removed today
morning, no bleeding
from urethra after
removing catheter.
16. Patient urinary
sensation +.
17. Reduced patient
Scrotal edema.
18. Morning Bowel
opened normally, Dark
green color and no
blood stain.
19. No headache and
3. Anxiety related to
disease condition
Reduce Anxiety
support.
support to the patient to
reduce the anxiety.
3. Communicate with
friendly and kindly.
4. Change the patient
position frequently.
5. Encourage the patient
for deep breathing and
exercises.
6. Educate simple
relaxation techniques.
7. Introduce the hospital
staff and environment for
the patient.
4. Communicated with patient

friendly and kindly, to relieve
the anxiety.
5. Frequently changed patient
position.
6. Encouraged the patient for
deep breathing and exercises.
simple relaxation technique.
8. Introduced the hospital
environment and ward staff.
9. Done the some divertional
Patient
satisfied and
gained some
knowledge
about his
disease
condition
Confusion signs.

for express feelings.
20. Patient on NG tube

3 hourly feeding to be
done.
activities to reduce anxiety

such as TV, and Radio.
9. Divert the patient

10. Provided evening care of
anxiety mind such as TV,
the patient to induce comfort.
Radio and other activities.
11. Educated the patient
10. Provide body wash to about his disease condition
increase comfort.
such as causes,
pathophysiology, and
treatment methods.
for about his disease
condition, causes,
12. Before doing every
pathophysiology, and
procedure explained briefly, to
treatment method.
reduce fear.
21. Vital signs

Temperature
102F/Axilla
Pulse 88/bpm
Respiration 26/bpm
BP 130/80mmHg
Spo2 96% on air
21. Blood Investigation
Report Findings
12. Explain the patient for

doing every procedure
decrease fear.
* FBC
WBC 7800cumm
RBC 2.95%
Hb% 10.4g/dl
PCV 30.5%
Plt count
234,000cumm
13. Provided proper

information about drug side
effects and interactions.
13. Provide proper

information about drug
side effects of drugs.
*CRP 28.4mg/L
*Liver Profile
Total Protein 7.2g/dl
4. Deficient knowledge
regarding disease
condition, treatment,
and prognosis.
Increase
patient
knowledge
regarding
1. Educate the patient for

his disease condition, such
as pathophysiology,
clinical manifestation and
1. Educated the patient for his

disease condition such as
pathophysiology ,clinical
manifestation and treatment
Patient
satisfied and
gained some
knowledge
Albumin 3.0g/dl
Globulin 5.4g/dl
A/G ratio 0.5
Alkaline Phospata
122U/L
ALT(SGPT) 38U/L
AST(SGOT) 42U/L
disease
condition
treatment methods.
modalities.

about causes of cirrhosis.

causes of cirrhosis.
3. Teach the patient

about avoid the risk
factors and worsening
factors.

avoid risk factors and
worsening factors.

about maintain normal
life style.
5.Educate the patient
about warning signs of
severe conditions.
6. Educate the family
members about cirrhosis
disease condition.
for consult the Dietitian.

maintain normal life style
such as regular exercise,
restrict sodium intake, and
dietary management.
worsening signs.
6. Educated the family
members for cirrhosis and
how to care a cirrhosis patient
in home.
about his
disease
condition.
Assessment
Nursing Diagnosis
Goal
Planning
Evaluation
23/02/2013 9.30am
Deficient Knowledge
regarding self and
Home care activities.
Educate self
and home care
interventions.

about self and home care
activities of the cirrhosis.
1.Educated the patient for stop

drinking alcohol if you stop all
alcohol intake, you may slow the
disease and feel better.
Patient
satisfied
the nursing
care.
Patient was
Discharged at 8am
Subjective Data
Mr. Ranjith 58yrs male
patient verbalized "I
am"

about how manage
symptoms in home
setting.
2. Avoid unnecessary medication

that may be harmful to your liver,
such as PCM or your kidneys such
as ibuprofen.
3. Follow up Care.
3. A low sodium diet helps relieve
that fluid retention problem.
1. Feeling good.
2. Loss of Appetite and
loss of food taste
sensation.
4. Eat a balanced diet with

adequate calories and protein.
5. To do regular simple Exercises
to help maintain proper posture.
3. Patient asked about

home care measures
and Drugs side effects.
Objective Data
1. Patient general
appearance is good.
2. Patient conscious ,
rationale and Alert to
Time, Place and Person.
3. Patient happy mood
in his discharge.
4. Skin color is normal
and no itching and
rashes.
6. Maintain optimal nutrition

level forget nutritional diet and
nutritional supplements such as
Vitamins A, B complex, D and K,
its help to reduce the Anemia.
7. Educated the patient deep
breathing and extremity
exercises.
drugs, such as side effects of
drugs.
9. Encouraged patient for proper
elimination habit.
6. Peripheries are warm.
10. Educate the patient about

worsening signs of his disease
condition. and that how to
manage and prevent further
damage.
7. No Extremities
edema.
11. Encouraged the patient

express the feelings.
8. IV cannula removed
and no bleeding in
cannula site.
12. Educated the patients

relatives and family members for
how to care cirrhosis patient in
5. Normal vision View.
home settings.
9.Patient discharge with
NG tube.
10.Urine output is good
and patient urine pass
with sensation.
11. Morning Bowel
opened without
lactulose.
12. Patient ambulate
morning without
restlessness and
discomfort.
13. Vital Signs
Temperature
98.6F/Axilla
Pulse 84/bpm
Respiration 24/bpm
BP 120/85mmHg
13. Encouraged patient for get

adequate food and fluids as
frequent interval.
14. Encouraged the patient to
seek frequent medical follow up
from a physician and take
medicine on time.
15. Visits from a monthly clinics
to monitor the patient progress.
16. At lastly submitted the
patient Diagnosis card Drugs.

Care of Patient With Liver Cirrhosis For Nursing Process Related Nursing Care

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Care of Patient With Liver Cirrhosis For Nursing Process Related Nursing Care

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Care Study

Nursing Care of the patient with

Submitted to: Tutor

Submitted by: Mohamed Anwer Naleef

Care Study of Patient with

Mr. Mohamed Anwer Naleef

Thank you Very Much

1. Objectives of Care Study

Short term Objectives:1.

Long term Objectives:1. To prevent Complications.

2.1. The Liver

2.2. The Liver Cirrhosis

3. Anatomy and Physiology of Liver

3.1. Anatomy of Liver

Blood Supply of the Liver

3.2. Functions of the Liver

Assessment of the Patient

4. Assessment of the Patient with Cirrhosis

Bio Graphic Data

History of Present Illness

Past Medical History

Past Surgical History

Father, Uncles and Brother died at 57 from Myocardial infarction.

Physical Assessment of the Patient

Ears/ Nose/ Throat

Chest is regular shape and size.

Patient non vegetarian.

Klean prep Enema bd Per Oral

6. Disease Condition ( Cirrhosis)

Cryptogenic cirrhosis (cirrhosis due to unidentified cause)

Liver cirrhosis occurs when the

Congestion in the hepatic portal system causes blood to be diverted to the

Development of Liver Alcoholic Cirrhosis

Bridging fibrosis (stained pink, arrows)

The liver architecture is

5.4. Signs and Symptoms of Cirrhosis

Tiredness (Fatigue) or even exhaustion.

Easy bruising due to impaired production of the Clotting Factors.

5.5. Diagnostic Tests (Investigations)

1. Preventing further damage to the liver

2. Treating complications of Cirrhosis

Bleeding from varices.

In some patients oral antibiotics can be prescribed to prevent

Patients with recurring episodes of spontaneous bacterial peritonitis.

3. Prevention and early detection of liver cancer

5.7. Complications of Cirrhosis

Edema and ascites

Spontaneous bacterial peritonitis (SBP)

Bleeding from esophageal varices

Rarely, some patients with advanced cirrhosis can develop hepatopulmonary

Liver cancer (hepatocellular carcinoma)

5.8. Prognosis of Cirrhosis

Nursing Care of Patient with

7. Nursing Care of Patient with Cirrhosis

Maintain adequate nutrition.

Nutritional intake adequate for individual needs.

History of long-term alcohol or injection drug use or abuse, alcoholic liver

1. Imbalanced Nutrition: Less than Body Requirements

2. Excess Fluid Volume

Risk factors may include:

4. Risk for ineffective Breathing Pattern

5. Risk for Bleeding

6. Risk for acute Confusion