DAFTAR PUSTAKA

Agus, S. dan Yanti, R. 2010. Penyakit Periodontal dan Penyakit

Jantung Koroner (Aterosklerosis). Bandung : UNPAD
Akpan, A. dan Morgan, R. 2002. Oral candidiasis. Postgrad Med J, 78: 455-59.
Andrutis KA., Riggle PJ., Kumamoto CA., dan Tzipori S. 2000. Intestinal lesions
associated with disseminated candidiasis in an experimental animal model.
Journal of Clinical Microbiology. 38(6): 23172323.
Bates, S. dan Rosa, J.M. 2007. Candida albicans Iff11, A secreted protein
required for cell wall structure and virulence. J Infect and Immun, 75(6):
2922- 2928.
Caroline, I. 2003. Periodontitis Sebagai Suatu Faktor Resiko Terjadinya Penyakit
Jantung Koroner. Tidak Diterbitkan. Skripsi. Medan : Fakultas Kedokteran
Gigi Universitas Sumatera Utara.
Constantinides, P. 2004. General Pathobiology. New Yersey: Appleton and Lange.
Cotter, G. dan Kavanagh. 2000. K Adherence mechanisms of C. albicans. British
J of Biomed Scien.
Daniel, W. 2005. Biostatic a Foundation for Analysis in The
Health Science 6th edition. Canada : John Wiley and Sons,
Inc.
Deguchi, J.O., Aikawa, E., Libby, P., Vachon, J.R., Inada, M., Krane,
S.M.,
Whittaker,
P.,
Aikawa,
M.
2005.
Matrix
Metalloproteinase-13/Collagenase-3
Deletion
Promotes
Collagen Accumulation and Organization in Mouse
Atherosclerotic Plaque. Circulation. Vol. 112: 2708-2715.
Dorlan, W.A. 2006. Kamus Kedokteran Dorland. Edisi 29. Jakarta : EGC.
Filler SG., Crislip MA., Mayer CL., dan Edwards JE. 1991.Comparison of
fluconazole and amphotericin B for treatment of disseminated candidiasis
and endophthalmitis in Rabbits. Antimicrobial Agents and Chemotherapy.
35(2): 288292.
Fuberlin.
2012.
C.
albicans
Patogenicity.
http://www.diss.fuberlin.de/diss/servlets/MCRFileNodeServlet/FUDISS_der
ivate_000000001849/03_chap1.pdf;jsessionid=AC33218EA116E17A598F
CDCDB7C50141?hosts=. [22 Januari 2012]
Hannson, G.K. 2005. Mechanisms of Disease:Inflammation,
43
Atherosclerosis, and Coronary
Artery Disease: Review
43

44

Literature. New England Journal of Medicine. Vol. 352: 16851695.

Hashimoto, T. 1991. In vitro study of contact-mediated killing of C. albicans
hyphae by activated murine peritoneal macrophages in a serumfree medium.
Infect. Immun, 59(10): 3555-3561.
J. Fransen J., J. van Cutsem J., R. Vandesteene R., dan P. A. J. Janssen PAJ.1984.
Histopathology of experimental systemic candidosis in guinea-pigs.
Sabouraudia. 22(6): 455469.
Javatilake AY., Samarayanake YH., Samarayanake LP. 2005. An ultrastructural
and a cytochemical study of candidal invasion ofreconstituted human
epithelium. J Oral Pathol Med, 34(4): 240-6.
Jawetz, E., Melnick, J.L. dan Adelberg, E.A. 1995. Mikrobiologi kedokteran.
Jakarta: EGC; 627-629.
Jawetz, M. 2004. Mikrobiologi Kelautan. Edisi 23. Alih Bahasa: Huriwati
Hartanto. Jakarta: EGC.
Jegier, B., Jaszewski, R., Kurnatowski, P., Kuba, K. dan Lelonek, M. 2010.
Mycotic DNA in Non-Atherosclerotic Aortic Wall of Coronay Patients is
Associated with sICAM-1 Expression.Circulation Journal, 74: 749-53.
Karnen B. 1996. Immunologi Dasar edisi Ilmu penyakit Dalam, edisi 2. Jakarta:
Gaya Baru.
Kumamoto CA. 2005. A contact-activated kinase signals C. albicans invasive
growth and biofilm development. Proc Natl Acad Sci USA. 102(15): 557681.
Kurita-Ochiai, T., Yamamoto, M. 2014. Periodontal Pathogens and
Atherosclerosis: Implications oh Inflamation and Oxidative
Modification of LDL. Biomed Research. Matsudo: Nihon
University School of Dentistry.
Kusumaningtyas, E. 2009. Mekanisme Infeksi C. albicans pada permukaan sel.
Jurnal. Lokakarya Nasional Penyakit Zoonis. Balai Penelitian Veteriner.
Bogor.
Libby, P. 2003.The vascular biologi of aterosclerosis. Am J Cardiol, 91(3A):3A6A [on line]. http://
www.harcourthhealth.com/SIMON/Braunwald/chapter30.pdf . [15 April
2015].
Libby, P. 2005. Prevention and treatment of atherosclerosis in Harrisons

45

Principles of Internal Medicine 16th Ed, Vol. II. US: The McGraw-Hill
Companies US. 1430 34.
Lyman CA., Gonzalez C., Schneider M., Lee J., dan Walsh TJ. 1999. Effects of
the hematoregulatory peptide SkandF 107647 alone and in combination with
amphotericin B against disseminated candidiasis in persistently neutropenic
rabbits. Antimicrobial Agents and Chemotherapy. 43(9): 21652169.
MacCallum, D.M., Coste, A., Ischer, F., Jacobsen, M.D., Odds, F.C. dan Sanglard,
D. 2010. Genetic dissection of azole resistance mechanisms in C. albicans
and their validation in a mouse model of disseminated infection. Antimicrob
Agents,54: 147683.
Mayo. 2005. Arteriosclerosis/Artherosclerosis. Mayo Clinic [on line].
http://www.mayoclinic.com/healtharteriosclerosis-atherosclerosis /DS00525
. [ 17 April 2015].
Michael, E.R., dan Lee Ann C. 2011. Pathogens and Atherosclerosis: Update on
the Potensial Constribution of Multiple Infectious Organisms to the
Pathogenesis of Atherosclerosis. Washington USA: Schattauer Thromb
Haemost, 106: 85867.
Miura, N.N., Harada, T. dan Shinohara, H. 2006. Lethal and severe coronary
arteritis in DBA/2 mice induced by fungal pathogen, CAWS, C. albicans
water- soluble fraction. Atherosclerosis, 186(2): 310-20.
Morrell, M., Fraser, V.J. dan Kollef, M.H. 2005. Delaying the empiric treatment of
candida bloodstream infection until positive blood culture results are
obtained: a potential risk factor for hospital mortality. Antimicrob Agents,
49: 36405.
Murwani, S., Ali, M. Dan Muliartha, K. 2006. Diet Aterogenik pada Tikus Putih
(Rattus norvegicus strain Wistar) sebagai Model Hewan Aterosklerosis.
Jurnal Kedokteran Brawijaya, 22(1): 6-9.
Nafilah, R. 2015. Deteksi Lesi Aterosklerosis Koroner pada Model Tikus
Periodontitis. Skripsi. Jember: Fakultas Kedokteran Gigi, Universitas
Jember
Naglik, J.R., Challacombe, S.J. dan Hube, B. 2003. C. albicans secreted aspartyl
proteinases in virulence and pathogenesis. Microbiol Mol Biol Rev, 67(3):
400-28.
Najjar, S. S., Scuteri, A., Lakatta, E. G. 2005. Arterial Aging: Is It
an
Immutable
Cardiovascular
Risk
Factor.
Dallas:
Hypertension. Vol.46: 454-462.
NHLBI (National Hearth Lung and Blood Institute). 2009. At A Glance:
Atherosclerosis. NIH Publication No. 09-7431.

46

Notoatmojo, S. 2005. Metodologi Penelitian Kesehatan Cetakan Ketiga. Jakarta :

Rineka Pustaka.

Nurgeldiyeva, M.J., Hojakuliyev, B.G. dan Muhammedov, M.B. 2014. Correlation

of Atherogenesis with an Infection of C. albicans. Int J Clin Exp Med , 7(8):
2137-43.
Pereira-Cenci, Del Bel C.A.A.,Crielaard,W. Dan Ten Cate J.M. 2008.
Development of Candida-associated denture stomatitis: new insights. J
Appl Oral Sci, 16(2): 86-94.
Polanco A., Mellado E., Castilla C., dan Rodriguez- Tudela JL. 1999. Detection of
Candida albicans in blood by PCR in a rabbit animalmodel of disseminated
candidiasis. Diagnostic Microbiology and Infectious Disease. 34(3): 177
183.
Prasetyo, A., Sadhana, U. dan Miranti. 2004. Profil Lipid dan Ketebalan Dinding
Arteri Abdominalis Tikus Wistar Jantan pada Injeksi Inisial Adrenalin dan
Diet Kuning Telur terhadap Kadar Lipid, Jumlah Sel Busa, dan Ketebalan
Dinding. Jakarta : Media Medika Indonesia.
Price, S.A. dan Wilson, L.M. 2003. Patofisiology : Konsep klinis Proses-Proses
Penyakit, Edisi 6. Jakarta : EGC.
Riskillah, A.G., 2010. Candida albican. Tidak Diterbitkan. Makalah.
Pekanbaru: Faculty of Medicine. University of Riau.
Rose, L.F. dan Mealey, B.L. 2004. Periodontics: Medicine, Surgery, and
Implants. Saint Louis : Elsevier Mosby.
Ross, R. 1999. Atherosclerosis and Inflamatory Disease. Am Heart
J. Vol. 340: 115-126.
Ross, R. 2004. Aterosclerosis and Inflammatory Disease. New England : Med.
Sarvtin, M.T., Parsa, A.M.Z., Kordbacheh, P., Hashemi, S.J., Mahmoudi, M.,
Daie, R., Mosavi, S.A.A., Masoomi, O. dan Hamta, A. 2014. Camparison of
Oral Candida Flora in Patients with Coronary Atherosclerosis and Healthy
People. ZJRMS,16(1): 40-3.
Selwyn AP, Braunwald E. Ischemic heart disease. In: Kasper DL, Fauci AS,
Longo DL, Braunwald E, Hauser SL, Jameson JL, editors. 2005. Harrisons
Principle of Internal Medicine [e-Book]. 16th ed. New York: McGraw-Hill;
p.1434-44
Shi, Y.U. dan Tokunaga, O. 2004. Chlamydia pneumoniae (C. Pneumoniae)

47

infection upregulates atherosclerosis related gene expression in human

umbilical vein endothelial cell (HUVECs). Atherosclerosis, 177(2): 245-53.
Stocker, R. dan John F. Keaney, Jr. 2004. Role of Oxidative Modifications in
Atherosclerosis. Physiol Rev, 84: 1381 478.
Suryadipradja, R.M. 2002. Trombus Intra-Arterial pada Sindrom Koroner Akut,
Peran
Pengobatan
dengan
Antikoagulan.
http://www.interna.or.id/interna/artikel/darurat2002/dar205.html. [ 15 April
2015].
Suryohudoyo, P. 2002. Kapita Selekta Ilmu Kedokteran Molekuler. Jakarta : CV
Sagung Seto.
Tjampakasari, C.R. 2006.
Kedokteran,151: 33-6.

Karakteristik

C.

albicans.

Cermin

Dunia

Umeyama, T. dan Kaneko, A. 2006. Deletion of the CaBIG1 gene reduces -1,6glucan synthesis, filamentation, adhesion, and virulence in C. albicans. J
Infect and Immun, 74(4): 2373-2381.
Vandewouwer, M. dan Collen, D. 2004. Thrombomodulin protein C-EPCR
system: Integrated to reglate coagulation and inflamation. Arterioscler
Thromb Vasc Biol, 24(8): 1374-83.
Waluyo, L. 2004. Mikrobiologi Umum. Universitas Muhammadiyah. Malang
Press. Malang
Whit, T.C. dan Agabian, N. 1995. C. albicans secreted aspartyl proteinases:
Isoenzyme pattern is determined by cell type and levels are determined by
environmental factors. Bacteriol, 177(18): 5215-21.
Wingard JR., Dick JD., Merz WG., Sandford GR., Saral R., dan Burns WH. 1980.
Pathogenicity of Candida tropicalis and Candida albicans after
gastrointestinal inoculation in mice. Infection and Immunity, 29(2): 808-13.
Yan, S., Negre, E. dan Cashel, J.A. 1996. Specific induction of fibronectin binding
activity by hemoglobin in C. albicans grown in defined media. Infect
Immun, 64(8): 2930-5.
Zunt, L.S. 2000. Oral candidiasis: Diagnosis and treatment. The J of Prac Hyg.