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HUB33
Agent: Hepatitis B Virus
(Hepadnavirus)
DEATH
Chronic Sate:
Host: HUMAN
Environmenrtal Factors
Known Factors:
Blood transfusion
Contaminated needles
Unprotected sex.
Unknown Factors:
Time and
Phisiologic
Changes:
Contagious
liver damage.
Illness:
Acute and Chronic diseases.
Recovery:
Acute:
Rest, drinking lots of fluids
Signs and symptoms:
and maintaining adequate
Abdominal pain
nutrition.Avoid fatty foods,
dark urine,
alcohol and antibiotics.
Fever and itching Chronic:
Joint pain
an antiviral medication
Loss of appetite might be recommended to
reduce or reverse liver
Nusea and
vomiting
damage and to prevent long
Weakness
-term complications of
Jaundice
hepatitis B.
DISCERNIBLE
EARLY
PATHOGENESIS
PRE-PATHOGENESIS
SPECIFIC PROTECTION
PRIMARY PREVENTION
ADVANCE
If liver damage
develops because
of
longstanding
infection,
the
person is said to
have
chronic
hepatitis that may
lead
to cirrhosis or liver
failure.
CONVALESCENCE
DISEASE
PERIOF OF PATHOGENESIS
HEALTH PROMOTION
To evaluate the
effect on health education
of Hepatitis B control and
prevention in reproductive
age women in rural areas to
put forward valuable
recommendations for the
strategies of Hepatitis B
control.
EARLY LESSIONS
Disability:
Swelling and
damage of the
liver.
lll-health, loss of
appetite, nausea,
vomiting, body
aches, mild fever,
and dark urine,
and then
progresses to
development
of jaundice.
Itching.
DISABILITY LIMITATION
REHABILITATION
Treatment is mainly
supportive with treatment of
symptoms (fluids,
antiemetics, rest).
Perform six-monthly
surveillance for HCC by
Itching can be difficult to treat.
hepatic ultrasound and alfaAdvise simple measures (stay cool,
fetoprotein testing in people
wear loose clothing, avoid hot
with significant fibrosis or
baths or showers).
cirrhosis.
Refer immediately
adults who develop
decompensated liver
disease. Referral should be
to a hepatologist or to a
gastroenterologist with an
interest in hepatology.
TERTIARY PREVENTION