Client-based Pathophysiology

Modifiable:
Diet
Stress
Lifestyle
Smoking
Exercise
Hypertension
Obesity

Non modifiable
Prior stroke
Age
Sex
Family history of stroke
Race
Socio-economic factor

Continuity of risk
factors
Too much intake
of LDL

Increased vasoconstriction due

to lifestyle
Increased
Blood perfusion
Arteriolar
vasoconstricti

FormationIIof fatty
Angiotensin
plaque deposits

Harden and
narrowing of
arteries

Hardening
Adrenal
cortex of fat
becomes
thrombus
II

Occlusion of major
vessels

aldosterone

sodium
reabsorption
water
reabsorption
plasma
volume (ECF)

Increased blood
coagulation

Stroke
(cerebro
vascular
accident

(Incontinenc

(weaknes

peripheral
Plaque ruptures the
resistance
artery

Hypertension

Changes in
arteriolar bed
systemic
vascular
resistance
afterload

Blood supply to
the heart
blood
renal
pressure
perfusion

blood flow to
the organs
blood
pressure

hyponatremia

Juxtaglomerul
ar cells

Angiotensinog
en

Renin

Angiotensin I

Beta receptor
activation
hypovolemi
a
(Visual
disturbances