Documente Academic
Documente Profesional
Documente Cultură
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http://www.library.vcu.
edu/cfapps/tml/oralpath
ology/browse_oral.cfm
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Taurodontism
Developmental dental
anomaly
Elongated large pulp
chambers
Uncommon
Occurs in deciduous and
permanent dentition
Affects molars most often
Crown is clinically normal
No treatment required
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Taurodontism
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Taurodontism
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Taurodontism
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Congenitally Missing Maxillary
Lateral Incisors
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Congenitally Missing
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Congenitally Missing
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Congenitally Missing mandibular
Premolar
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Supernumerary mandibular
Premolar
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Mesiodens
D Nasal Fossa
F MPS
H ANS
I Mesiodens
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Microdontia of the Maxillary
Right Third Molar
Abnormally small teeth
Location
Maxillary lateral incisors
Third molars
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Impaction
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Impactions
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B Microdontia
C Healed extraction site
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Mesioangular Impaction
Mesioangular
Distoangular
Vertical
Horizontal
Transverse
Inverted
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Transverse
Kasle
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Mandibular Tori
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Mandibular Tori
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Max Tori
Etiology: Inherited
Clinically: Nodular
growth, bilateral, multiple
or solitary, lingual surface
of the mandible, middle of
palate
Treatment: Nothing,
surgery only if needed such
as pain or large growth
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Dilaceration
Sharp bend or curve in
the root of a formed
tooth
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Dilaceration Etiology
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Dilaceration
Radiographic appearance
Sharp bend or curve in the root
Treatment
None
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Hypercementosis
Appears like a radiopaque
bulbous enlargement
Caused by :
Excessive cementin formation
Chronic inflammation of the
tooth
Loss of antagonist
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Hypercementosis
Etiology :
Increased or decreased forces
Pagets disease
Hyperpituitarism
Chronic infection in adjacent area
Excessive cementin formation
Chronic inflammation of the tooth
Loss of antagonist
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Hypercementosis
Location
Apical area of one or all the teeth
Treatment none
Problems
May have trouble extracting teeth
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Hypercementosis
Clinically
Undetectable
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External Resorption
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Internal Resorption
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Attrition
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Periapical Abscess
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Lateral or Periodontal Abscess
Also called periodontal Abscess
Results from bacterial infections
Results from inadvertent
embedding of foreign objects
subgingivally
Calculus
Popcorn husks
They obstruct normal sucular
drainage
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Lateral or Periodontal Abscess
Sometimes seen following PD in deep
pockets
Fistula may occur
Treatment
Manage acute signs and symptoms by
getting drainage and remove foreign object
PD to establish drainage
Eliminate pocket
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Periodontal vs Periapical Abscess
Periodontal Abscess Periapical Abscess
Vital Tooth nonvital Tooth
No Caries Caries
Deep Pocket No Pocket
Tooth Mobility No or little Tooth
Mobility
Draining Fistula at Draining Fistula at
lateral aspect of tooth apex
Lateral Radiolucency ApicalRadiolucency
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Periapical Abscess
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Periapical Abscess
Almost one-third (32%) of
the mentally retarded and
developmentally disabled
patients had periapical
radiolucencies that had not
been suspected without
radiographs
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Odontoma
Radiographic Appearance
Irregular mass of calcified
material (toothlike structures)
surrounded by a narrow
radiolucent band
Smooth outer periphery ranging
from radiolucency to
radiopacity
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Odontoma
Compound
Tooth structures are identified
radiographically
Complex
Appears as a radiopaque mass
Cyst involvement may occur
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Odontoma Etiology
Infection
Local trauma
Inherited trait
Mutant gene
Postnatal interference
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Odontoma Treatment
Surgical removal
Prognosis good
Rare recurrence
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Compound Odontoma
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Compound Odontoma
Numerous small toothlike
radiopacities surrounded
by a radiolucent halo
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Compound Odontoma
Dome-shaped radiodense
mass above the crown of the
unerupted canine composed
of numerous small tooth-like
structures
The mass is surrounded by a
radiolucent zone.
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Compound Odontoma
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Complex Odontoma
Radiopaque mass
surrounded by a
radiolucent halo
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Odontoma Complex
Description: Large
irregular shaped
radiodense mass
preventing the eruption of
tooth #30
Note that the
radiodensity of the mass
is the same as the
adjacent teeth.
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Odontoma
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Condensing Ostetitis
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Condensing Ostetitis
Localized area of increased
radiodensity of the alveolar bone
extending from the apical portion of
the distal root into the underlying
bone forming a bulbous appearing
radiodense mass.
Note the faint outline of the root
visible within this bone showing the
increased radiodensity.
Location: Distal root of lower first
molar
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Sclerotic Bone
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Occlusal Trauma
Widening of the
periodontal ligament
spaces
Due to heavy trauma
No alveolar crestal bone
loss
No increased probing
depths
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Occlusal Trauma
Reduced bony support
Increasing tooth mobility
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History:
A healthy 24 year old female
presented at a university dental
screening clinic with
masticatory muscle pain on the left
side
There was no regional
lymphadenopathy or cortical
expansion
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History:
The marginal gingivae were "brick
red" but there was no periodontal
pocketing or bleeding when probing.
Tooth # 3-5 (20) was mobile (grade
2), showing heavy wear facets due to
bruxing.
Mild soreness.
no history of trauma.
The tooth failed to respond to
thermal testing
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Differential Diagnoses
Description: The periapical radiograph
shows a widened periodontal ligament
space around tooth #3-5 (20) with periapical
condensing osteitis.
Differential Diagnoses:
1) Traumatogenic occlusion at #3-5
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Gemination
Clinical view
2 completely or
incompletely separate
crowns
One root or root canal
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Globulomaxillary Cyst
Often pear shaped
Occurs between the
max lateral & canine
Clinical Features
All teeth vital
Adults males &
females equally
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Odontogenic Cysts
Inflammatory Developmental
Radicular Dentigerous Cyst
Paradental
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Cysts
Dentigerous cyst
Radicular or Periapical
Residual
Incisive canal
Globulary maxillary
Nasopalatine
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Dentigerous Cyst
Radiographic Features
Smooth uniocular radiolucency
Larger than 4 mm extending
from the crown to the reduced
epithelium
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Dentigerous Cyst
Also known as
Follicular cyst
Eruptive cyst
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Dentigerous Cyst
Age and Gender Affected
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Dentigerous Cyst
Location
Mandibular 3rd molars- often extending to
and destroying the ramus
Maxillary canine region- compromising the
maxillary sinus
These areas also have a higher incidence of
impactions
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Dentigerous Cyst
Clinical Features
Always associated with the crown of an
imbedded or unerupted tooth
Aggressive lesion causing expansion of
bone and extreme displacement of teeth
Painful
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Dentigerous Cyst
Histologic Characteristics
Stratified squamous epithelium lining the
lumen
Surrounded by a thin connective tissue
wall
Epithelial lining is not keratinized
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Dentigerous Cyst
Large well-defined
unilocular
radiolucency
associated with the
crown of the
unerupted molar
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Dentigerous Cyst
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Eruption Sac
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Radicular
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Radicular or Periapical
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Incisive Canal Cyst or Nasopalatine
duct cyst
Description: Heart-shaped
well-circumscribed
radiolucency of the midline
of the anterior hard palate
Location: Midline of the
anterior maxilla above and
between the apices of
central incisors
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Floor of Nasal Fossa
Periapical Abscess
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Static
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Hutchinson’s Incisors
Are notched incisors or
sometimes called
screwdriver shaped.
The shape of the
central incisors is
altered in 10% to 30%
of children with
congenital syphilis
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Hutchinson’s Incisors
They resemble a screwdriver or may show notching
of the incisal edges.
Maxillary centrals are most frequently involved but
the lateral incisors and the mandibular incisors may
also be affected.
Deciduous dentition is not altered.
The alteration in the shape of the teeth is due to
changes in the tooth germ during the stage of
morphodifferentiation.
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Hutchinson’s Incisors
The molar counter part is called, mulberry molars.
The shape of the 1st permanent molar is altered in
10% to 30% of the patients with congenital syphilis.
The occlusal surface is much narrower than normal
and gives the crown a pinched appearance.
The teeth also show hypoplasia of the enamel
Pflüger molars are similar to mulberry molars
except they do not have hypoplasia present.
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Amelogenesis Imperfecta
Is a developmental anomaly
related to hypocalcification.
It is hereditary not acquired.
Can affect permanent and
deciduous teeth, but sometimes
only the permanent teeth are
affected.
The enamel is affected, not the
dentin or the root.
It the enamel is present, it is
thin, is stained various shades
of yellow and brown and easily
fractures away.
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Amelogenesis Imperfecta
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Amelogenesis Imperfecta
Etiology: Inherited, failure of the ameloblasts to lay
down enamel matrix properly
Clinically: Teeth are pitted, yellow to brown color,
affects all the teeth, both dentitions
Normal pulp and dentin, rough and thin enamel
Treatment: Cosmetic surgery, such as restorations
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