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  • Pathophysiology of Prostate Cancer Risk Factors

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    mkho
    216 vizualizări3 pagini
    Prostate cancer develops through a multi-step process starting with initiation by carcinogens interacting with DNA to cause mutations. The cancer then progresses through promotion of abnormal cell growth and replication, eventually forming precancerous and invasive lesions. If left untreated, the cancer may metastasize from the prostate to other areas like the testes, and treatment aims to stop metastasis and progression through medication or surgical removal of affected tissues.

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    Prostate Cancer Pa Tho

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    Prostate cancer develops through a multi-step process starting with initiation by carcinogens interacting with DNA to cause mutations. The cancer then progresses through promotion of abnormal cell growth and replication, eventually forming precancerous and invasive lesions. If left untreated, the cancer may metastasize from the prostate to other areas like the testes, and treatment aims to stop metastasis and progression through medication or surgical removal of affected tissues.

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    Attribution Non-Commercial (BY-NC)
    Descărcați ca DOC, PDF, TXT sau citiți online pe Scribd
    Indicator pentru conținut neadecvat
    216 vizualizări3 pagini

    Pathophysiology of Prostate Cancer Risk Factors

    Încărcat de

    mkho
    Prostate cancer develops through a multi-step process starting with initiation by carcinogens interacting with DNA to cause mutations. The cancer then progresses through promotion of abnormal cell growth and replication, eventually forming precancerous and invasive lesions. If left untreated, the cancer may metastasize from the prostate to other areas like the testes, and treatment aims to stop metastasis and progression through medication or surgical removal of affected tissues.

    Drepturi de autor:

    Attribution Non-Commercial (BY-NC)
    Descărcați ca DOC, PDF, TXT sau citiți online pe Scribd
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    din 3

    PATHOPHYSIOLOGY OF PROSTATE CANCER

    Predisposing Factors
    Precipitating Factors -Heredity
    -Diet (Processed foods) -Age
    -Gender (male)

    Phase 1 Excretion
    Enzyme

    Phase II Excretion
    detoxification
    enzymes

    Ultimate Carcinogen

    DNA adduct
    (DNA+Carcinogen)

    Initiation

    Mutation

    DNA Normal DNA


    repair

    Promoters Abnormal DNA and cell replication

    Promotion
    Apoptosis

    Precancerous lesion

    1
    Invasive lesion

    Progression

    Hyperplasia of
    prostate periphery

    Metastasis
    at the testes

    Atrophy of
    the testis
    Adenocarcinoma
    of the testes

    If untreated If treated

    Further Medication
    metastasis regimen / Surgical
    removal of
    adenocarcinoma

    Wellness

    Right orchiectony (scrotal)


    Left orchiectomy
    (inguinal)
    Bicaluatmide 50 mg TID

    2
    3

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