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Periodontology 2000, Vol. 71, 2016, 228–242 © 2016 John Wiley & Sons A/S.

& Sons A/S. Published by John Wiley & Sons Ltd


Printed in Singapore. All rights reserved PERIODONTOLOGY 2000

Management and prevention of


gingival recession
G E O R G E K. M E R I J O H N

Gingival recession, in other words exposure of the root caries. This alone is reason to incorporate practi-
root surface caused by apical displacement of the gin- cal protocols for managing and preventing gingival
gival margin past the cemento–enamel junction (18, recession into daily clinical practice.
54, 91), is highly prevalent worldwide. Depending on The prudent clinician is aligned with the patient in
the population surveyed, the percentage of people the desire to avoid both neglectful oversight and
affected ranges from 30% to 100% (2, 46, 48, 56, 57, inappropriate treatment. Between lies the approach
79, 84). Its prevalence and severity increases with age to personalized, targeted and step-by-step conserva-
(2, 56). In the USA alone, the prevalence of recession tive measures for the management and prevention of
of ≥1 mm in persons ≥30 years of age has been gingival recession. Therefore, the dental practitioner
reported to be 58% and averages 22.3% teeth per per- must consider the esthetic and dental health con-
son (2). In both clinical practice and continuing edu- cerns associated with gingival recession, as well as
cation, dental professionals invest substantial time individual patient factors, when developing a treat-
and effort on treating the esthetic zone. Clinicians are ment strategy. Bearing in mind these various consid-
especially frustrated when gingival recession occurs, erations, the aims of this article were to: (i) illustrate
for example, after orthodontic tooth movement three major factors associated with increased suscep-
(Fig. 1A) and/or after dental restoration margins are tibility to gingival recession; (ii) present practical con-
properly placed in proximity to gingival tissue. Den- cepts regarding the recognition and management of
tists and dental hygienists often ask, ‘How does one risk exposures that can be implemented in a clinical
avoid getting gingival recession in the first place and setting; (iii) review essential data collection and
then after it’s treated, how do we keep it from coming recording for monitoring patients; (iv) introduce
back?’. Dental appearance, compromised by gingival chairside clinical-decision-support tools designed to
recession, is a common patient concern. However, help the clinician focus on triage, evaluation, plan-
this awareness is often limited to those patients with ning and patient communication regarding the pre-
pronounced gingival display and their focus infre- vention and management of gingival recession; and
quently goes beyond the facial aspect of the anterior (v) propose clinical decision-making criteria for when
dentition. Patient-driven concerns about gingival and how to monitor gingival recession, for deciding
recession are also raised if it interferes with comfort when a patient is a candidate for surgical evaluation
(e.g. root sensitivity) and/or function. or referral to a periodontist and, if surgery is the treat-
In addition to esthetic concerns of the patient, an ment of choice, what should be considered as key
unfavorable consequence of gingival recession is the surgical outcome objectives.
exposure of root surfaces to a potentially cariogenic
supragingival microbiota (Fig. 1B). In the USA, the
prevalence of root caries has been reported to be Gingival recession: causes and
55.9% among those ≥75 years of age (38). Of great susceptibility
concern is that the group ≥65 years of age, which rep-
resented 12% of the population in 2000, is expected to The primary causes of gingival recession are plaque-
exceed 20% of the population by 2030 and root caries induced inflammation (79, 81) and mechanical (phys-
is also expected to increase (38). Prevention of gingi- ical) abrasion/removal (27, 36, 41, 44, 46, 56, 76, 79,
val recession is essential in the primary prevention of 83, 89). Occasional causes of recession include

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Management & prevention of gingival recession

A B

Fig. 1. Root exposure and root caries. (A) A 33-year-old tially early-stage/reversible root decay. (B) A 56-year-old
male orthodontic patient who developed gingival recession female patient with progressive gingival recession and clin-
during treatment. The patient presented with thin (deli- ically detectable root caries. Note the thin gingival tissue
cate and fragile) gingival tissue on the canine and first pre- and presenting mucogingival conditions (see Fig. 2). The
molar with no gingival recession before treatment. Oral patient reported that she is a tobacco user, flosses four to
hygiene consisted of: enthusiastic use twice daily of a five times per week and uses a power toothbrush and/or a
power toothbrush or a large-headed medium/firm-bristle manual medium/firm-bristle toothbrush with over-the-
manual toothbrush; once daily use of Waterpikâ on med- counter fluoridated toothpaste twice daily. She periodically
ium pressure aimed at the gum line at the recommended used tooth-whitening strips. Previous periodontal treat-
90° angle; and sporadic flossing. The first premolar ment consisted of routine adult prophylaxis two to three
demonstrates tissue erythema and advanced recession. times per year, oral hygiene instruction, typical dietary
The canine demonstrates moderate recession with poten- counseling and infrequent in-office fluoride application.

thermal and chemical injury (41, 76). Mitigating these tutes a clinically important reduction (5). Teeth with
causes in susceptible patients will decrease the inci- less than 2 mm of keratinized tissue have been
dence and severity of gingival recession. Three major studied in the literature. Lang & Lo } e (52) demon-
factors are associated with increased susceptibility to strated that although tooth surfaces may be kept
gingival recession: (i) thin gingival tissue; (ii) free of clinically detectable plaque, areas with
mucogingival conditions; and/or (iii) a positive his- <2 mm of keratinized gingiva tended to remain
tory of progressive gingival recession and/or inflam- inflamed. However, other clinicians have evaluated
matory periodontal disease(s) in teeth presenting sites with <2 mm of keratinized tissue and con-
with either or both of the first two factors. cluded that these sites do not necessarily develop
Relative to its clinical appearance, gingival tissue gingival recession solely as a result of a narrow
can be broadly categorized as thick, average or thin. width or band of keratinized tissue (28, 29, 31, 32,
Thick gingival tissue appears densely fibrous and 47, 49, 65). It is therefore important to emphasize
noticeably keratinized, whereas thin gingival tissue (1, that surgical therapy is not warranted based solely
12, 22, 50, 67, 77) appears more translucent with less on the presence of thin gingival tissue, gingival
obvious keratinization, as depicted in Fig. 2, an exam- recession, probing depths extending beyond the
ple of a visual guide to enhance chairside patient mucogingival junction and/or the absence or reduc-
communication. When describing gingival tissues to tion of keratinized tissue (31, 32, 54, 60, 63).
patients, it is helpful to refer to thick tissue as protec- Nevertheless, the clinician must consider when
tive and durable and thin tissue as fragile and deli- and if gingival recession is an indicator of progres-
cate. It is important to note that not all teeth will sive loss of periodontal support and future reces-
necessarily develop gingival recession solely as a sion. For example, Serino et al. (81) reported that
result of thin gingival tissue (Fig. 3). teeth with a positive history of progressive gingival
Mucogingival conditions (Fig. 2) are defined as recession have increased susceptibility to additional
deviations from the normal anatomic relationship apical displacement of the soft-tissue margin. They
between the gingival margin and the mucogingival also noted that loss of proximal periodontal support
junction (42). The most common mucogingival con- was associated with gingival recession at the buccal
ditions are: gingival/soft-tissue recession (42); the surface. Yoneyama et al. (94) determined that the
absence or reduction of keratinized tissue; and/or major feature of the progression of destructive peri-
probing depths extending beyond the mucogingival odontal disease with age was gingival recession. Sar-
junction (5). In the section, Parameter on Mucogingi- fati et al. (79) found that gingival bleeding was
val Conditions of the Parameters of Care, the Ameri- significantly associated with the severity of gingival
can Academy of Periodontology identifies reduced recession and concluded that the inflammatory
keratinized tissue as a mucogingival condition but reaction to dental biofilms is the predominant
does not specify numerical criteria for what consti- biologic feature shared by gingival recession and

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Merijohn

Fig. 2. Gingival recession chairside


visual guide.

periodontitis. Albandar & Kingman (2) suggested conditions. Although mitigating the causes of gingi-
that gingival recession occurs primarily as a conse- val recession in susceptible patients will decrease its
quence of periodontal diseases and aggressive use incidence and severity, implementing practical
of mechanical oral hygiene measures. A positive his- management and prevention strategies in a clinical
tory of inflammatory periodontal disease (e.g. pla- setting can be challenging (46, 50, 89, 94). Of prime
que-induced gingivitis and localized chronic importance in implementing such prevention and
periodontitis) can be considered an important fac- treatment strategies is an understanding of the cur-
tor associated with gingival recession, especially for rent knowledge of factors associated with the risk of
teeth with thin gingival tissues and/or mucogingival initiation and progression of gingival recession.

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Management & prevention of gingival recession

A B dental plaque, forceful brushing and tobacco use),


whereas others are not (e.g. age, gender and history
of progressive gingival recession). The term ‘modifi-
able conditions’ is used in this article for those con-
tributing factors and conditions commonly
associated with gingival recession that lend them-
selves to modification through conventional inter-
ventions in a clinical setting. These modifiable
conditions are listed in Table 1. Increasing patient
awareness about gingival recession susceptibility
and modifiable conditions is an indispensable first
step for an effective management and prevention
program. In addition to being examined and
Fig. 3. Conservatively managed and nonsurgically treated advised about findings by their dentists and dental
thin gingival tissue (mandibular left canine tooth): a 17- hygienists, independent self-discovery by patients is
year follow-up. (A) A 39-year-old male patient on a 4-
often a strong motivator for adopting preventive-
month recall interval in 1993 presenting with thin gingival
tissue, narrow band of keratinized tissue and probing oriented lifestyle choices and accepting treatment
depth extending beyond the mucogingival junction. Treat- interventions appropriate for managing and pre-
ment consisted of: coronal scaling with hand instruments venting dental problems.
and coronal polishing; and subgingival hand instrumenta- Online knowledge resources can serve as a practical
tion with curettes for removal of biofilm, plaque and any
means to increase awareness and decrease confusion
clinically detectable calculus. Personal oral hygiene con-
sisted of: twice daily nonforceful sulcular/Bass brushing about dental conditions. My Gum Recession Analyzer
technique with a soft-bristled, narrow-headed manual and Gum Recession FAQ (www.gumtest.com) are two
toothbrush and over-the-counter fluoridated toothpaste; examples of evidence-based online resources (Fig. 4).
flossing once daily with lightly waxed or nonwaxed dental My Gum Recession Analyzer is a self-screening tool that
floss; tooth pick use (blunted and softened tip) once
features those gingival recession susceptibility factors
weekly; and use of an end-tufted brush for buccal cervical
plaque removal. The patient was instructed to use a mag- and modifiable conditions from Table 1 that more
nified lighted mirror when performing procedures. The readily lend themselves to patient self-discovery. It is
patient had an acceptable level of skill and reasonable designed to help the individual: (i) discover if she/he
compliance with the recommended oral hygiene proce- has gum recession; (ii) determine her/his susceptibility
dures. (B) The same tooth in 2010: relatively unchanged
to gum recession; and (iii) identify exposure to any of
thin gingival tissue, 17 years later, is observed.
eight common, but modifiable, risks associated with
gum recession. Gum Recession FAQ addresses fre-
The susceptible patient and factors quently asked questions about gingival recession.
that increase gingival recession risk Anecdotal evidence suggests that use of appropriate
online knowledge resources can improve and stream-
Studies have reported several contributing factors line patient communications and consultations.
and conditions commonly associated with gingival Encouraging patients to perform self-screening and
recession and/or increasing recession (8, 46, 55, 57, increase personal awareness augments the clinician’s
76, 77, 79). Exposure to such factors and conditions
can make susceptible patients particularly vulnera-
ble to gingival recession (4). Although definitive
causality and magnitude-of-effect hierarchy have
yet to be determined for the contributing factors
and conditions associated with gingival recession,
there is some evidence upon which to base recom-
mendations for its management and prevention.
Presently, clinicians can recommend simple lifestyle
interventions and consider various dental proce-
dures that are capable of reducing the risk of
inflammation and/or mechanical injury for tissues
susceptible to gingival recession. Some contributing
factors and conditions are readily modifiable (e.g. Fig. 4. Online gingival recession self-screening.

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Table 1. Susceptibility factors and modifiable conditions

efforts in determining which patients are most Essential data collection and
susceptible to gingival recession. Patients can access
recording
online knowledge resources at their convenience from
smart phones or computer tablets/personal computers.
The surest way to under-treat or over-treat gingival
Clinicians utilize online self-screening tools in a variety
recession is through inadequate records.
of ways based on personal preferences. For example,
Evaluation of a patient’s periodontal status requires
patients can be instructed to use online tools outside
obtaining relevant medical and dental histories and
the practice setting before appointments, in the prac-
conducting thorough clinical and radiographic exam-
tice reception room or in the dental operatory during
inations with evaluation of extra-oral and intraoral
appointments such as examinations and hygiene
structures (5). Traditional clinical assessments and
maintenance visits. It is beyond the scope of this
procedures remain the foundation upon which peri-
paper to review online self-screening tools in detail.
odontal diagnoses are made (35). The Parameter on
These can be evaluated online by the clinician for
Comprehensive Periodontal Examination, developed
applicability in her/his individual practice setting.
by the American Academy of Periodontology, states
Decreasing the susceptible patients’ exposure to
that all relevant clinical findings should be docu-
modifiable conditions will decrease future risk for
mented in the patient’s record (5). See Fig. 5 for rec-
gingival recession and increase the likelihood of its
ommendations regarding where to document, in the
long-term prevention. However, developing patient-
patient record, the susceptibility factors and modifi-
centered treatment plans for the management and
able conditions associated with gingival recession.
prevention of gingival recession necessitates that the
To establish a baseline and to track changes effec-
clinician first examines the patient for key susceptibil-
tively over time, all measurable and/or detectable
ity factors and modifiable conditions (Table 1). Posi-
essential clinical findings (see Fig. 5) are recorded on
tive findings need to be documented and all essential
the periodontal examination record. A comprehen-
data recorded for long-term tracking and monitoring.
sive periodontal examination should be performed
In the busy practice setting, having a practical and
for all new patients and repeated at appropriate peri-
efficient means to do so can improve workflow and
odic intervals based on the needs of the individual
decision making.

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Management & prevention of gingival recession

Fig. 5. Documenting susceptibility factors and modifiable conditions.


MH/DH, Medical History/Dental History

patient. Figure 6 is an example of a periodontal fluoride application (if needed) (5). One must also
examination record that facilitates documentation consider the time it takes to greet and exit the patient,
and recording of the comprehensive periodontal as well as to perform operatory disinfection and
examination parameters, gingival recession suscepti- chairside set up. Nevertheless, a thorough periodon-
bility factors and measurable/detectable gingival tal evaluation is a critical component of the dental
recession contributing factors and conditions. hygiene/maintenance appointment (5). A practical
Periodontal evaluation and data recording during approach is to evaluate the patient for the key peri-
dental hygiene/maintenance appointments can be odontal parameters and then document, on the peri-
very challenging, especially for adult patients with a odontal maintenance record, only those significant
relatively full complement of teeth and/or implants. negative changes relative to the most recent compre-
Within the allotted time it is not always possible to hensive data on the periodontal examination record.
perform and record a comprehensive periodontal Figure 7 is an example of a periodontal maintenance
examination, in addition to performing other essen- record that simplifies documentation and recording
tial tasks, such as: medical and dental health history and is compatible with the periodontal examination
update; review of radiographs; head and neck exami- record example in Fig. 6. Regardless of the recording
nation; appliance checks; caries and restoration systems used, examination and documentation are
check; scaling/root planing as needed; coronal pol- the important first steps for patient-centered clinical
ishing; oral hygiene review and instruction/training; decision making. After the data are gathered, use of
lifestyle assessment and recommendations; and chairside tools designed to help the clinician focus on

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Merijohn

Fig. 6. Example of a periodontal examination record.

triage, planning, communication and education can both as a reference for the clinician and a visual
facilitate the development of interventions for the communication tool for use with patients.
management and prevention of gingival recession. These chairside clinical-decision-support tools are
like other chairside tools in that they help busy clini-
cians focus on triage, evaluation, planning and
Patient education and treatment patient communication (60, 61, 63, 64, 66, 68). They
planning can be used to facilitate the development of interven-
tions for the prevention and management of gingival
After recording all clinical measures, patient history recession. Specifically, these chairside tools help sup-
and other relevant findings, patient education and port the clinician in: (i) identification of patients who
treatment planning are the next steps in the man- are susceptible to gingival recession; (ii) assessment
agement of gingival recession. Treatment algo- of key modifiable conditions associated with gingival
rithms and patient presentation tools can be recession; (iii) development of targeted and personal-
particularly beneficial in this critical stage of patient ized treatment/action plans for the prevention and
management. An example of such a tool that can management of gingival recession; and (iv) communi-
be employed is presented in Table 2 (Gingival cation with patients at the point of care, which can
Recession Checklist). This checklist is a quick refer- help increase patient awareness, develop preventive
ence chairside guide for the clinician and is to be behaviors and boost treatment acceptance.
used in the dental examination/treatment opera- Evidence supports the use of checklists in clinical
tory. The companion Gingival Recession Chairside settings to improve the quality of care and to reduce
Visual Guide (Fig. 2), on its reverse side, can serve errors and complications (40, 80). The World Health

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Management & prevention of gingival recession

Fig. 7. Example of a periodontal maintenance record.

Organization developed its validated Surgical Safety patient. For every positive finding on the Checklist, a
Checklist, a 19-item tool created by the World Health tailored and personalized treatment/action plan is
Organization in association with the Harvard School developed to address it. Interventions should be
of Public Health, modeling it after in-flight safety patient-centered and focused on clinically relevant
checklists used by the airline industry (34, 92). Clini- outcomes (6, 62, 64, 66, 68, 78). Whenever clinically
cal examination and procedural checklists have been reasonable, interventions should be instituted in a
introduced and recommended for use in dental prac- step-by-step approach, starting first with the most
tice (21, 72). Like other checklists and visual guides, effective conservative measures. As a chairside clini-
these tools are designed to be used in conjunction cal-decision-support tool, this checklist example is
with examination, decision-making or treatment- designed to be simple and easy to use (63). Like
planning appointments, including dental hygiene/ any safety checklist, it is not intended to be 100%
maintenance appointments (61, 63). This type of all-inclusive: clinicians might find that there are
checklist and visual guide can also be used for clinical exceptions.
staff training, calibration and ongoing chairside
support in facilitating patient communication.
This sample checklist presented in Table 2 is com- Using the gingival recession
prised of four sequential steps: (A) determine whether checklist and visual guide in the
the patient is susceptible to gingival recession; (B) practice setting
evaluate the patient for the presence of any gingival
recession modifiable conditions listed on the Check- It is beyond the scope of this paper to provide an in-
list; (C) develop a treatment plan to address each pos- depth review of the numerous therapeutic
itive Checklist finding; and (D) review the visual guide approaches available to clinicians for the manage-
(Fig. 2) and checklist (Table 2) findings with the ment of every modifiable condition listed on the

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Table 2. The gingival recession checklist

checklist (Table 2). Select conditions are presented highest level of inflammation occurring in subjects
below. with poor glycemic control (23, 59). Significantly
greater gingival bleeding is seen in patients with
poorly controlled diabetes than in either subjects with
Poor metabolic control of diabetes
well-controlled diabetes or nondiabetic controls (30,
Greater gingival inflammation is seen in adults with 59). Importantly, increased periodontal destruction is
type 2 diabetes than in nondiabetic controls, with the associated with poorer glycemic control (58). As both

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Management & prevention of gingival recession

plaque-induced gingivitis and/or periodontitis and same, compared with never users (8). Smoking has a
poor glycemic control are modifiable risk exposures long-term chronic negative effect on many important
for gingival recession (10, 44, 79, 81, 84, 94), for dia- aspects of the inflammatory and immune responses,
betic patients who are susceptible to gingival reces- including alterations in the vasculature and on neu-
sion, appropriate metabolic control coupled with trophil function. In addition, nicotine and other
improved plaque control will decrease the risk of gin- tobacco compounds have detrimental effects on
gival recession. fibroblast function, including proliferation, adhesion
Clinicians should routinely communicate with dia- to root surfaces and cytotoxicity (69), which may,
betic patients regarding personal glycemic control in turn, adversely affect treatments for gingival
targets and current status. Patients may be more recession.
familiar with their average glucose (eAG) than they Indirect evidence suggests that a significant rever-
are with glycated hemoglobin percentage (A1C) val- sal of periodontitis risk might be achievable within
ues. Websites, including www.diabetes.org, provide 10 years after quitting smoking (33). Dental profes-
simple conversion calculators for converting glycated sionals are in a unique position to help tobacco users
hemoglobin to average glucose. A simple reference who present for dental care by providing assistance
conversion table kept in the dental operatory will save to help them stop smoking or using other tobacco
time and improve communication efficiency. If there products (16). As such, this has become part of the
is concern regarding the patient’s glycemic control, responsibility of all oral health professionals (75), and
the dentist should fax the physician and request the tobacco users should be encouraged to quit as part of
last 2 years of glycated hemoglobin values. their overall periodontal management (20). Clinicians
For patients with good glycemic control, routine should routinely check with tobacco-using patients
dental therapy can be planned accordingly. For and actively provide ongoing encouragement, sup-
patients with poor glycemic control, anecdotal evi- port and positive reinforcement for tobacco-use ces-
dence suggests that clinicians should consider the fol- sation. Although some clinicians may feel that
lowing approach: refer to a physician for further managing in-office smoke-cessation programs may
evaluation and care; consult with a physician regard- be impractical and/or cost-prohibitive, it takes little
ing glycemic control and oral conditions; treat dental time and effort to provide tobacco-using patients
infections using conservative therapies, such as scal- with an up-to-date list of appropriate local tobacco-
ing and root planing, root canal therapy and/or cessation programs and online resources. A more
restoration of cavitated carious lesions; and delay detailed description of the benefits of structured
elective therapies, such as gingival grafting or dental tobacco-cessation programs for dental practitioners
implant placement surgery. is presented in another article in this volume of Peri-
The field of screening and assessment of blood glu- odontology 2000.
cose levels continues to evolve, and dental profes-
sionals can participate by offering in-office finger-
Other modifiable conditions associated
stick testing. Dental clinicians should give guidance,
with the major pathogenic mechanisms
in the form of consultation and reference material,
of gingival recession
supporting the key healthy lifestyle choices for
managing diabetes. These include seeing a physician, The Gingival Recession Checklist (Table 2) and the
undergoing regular physical activity, limiting con- Gingival Recession Chairside Visual Guide (Fig. 2) are
sumption of sugar-sweetened beverages, eating a examples of practical chairside support tools that
healthy diet, managing periodontal disease and con- illustrate, to patients, the modifiable conditions com-
trolling modifiable conditions associated with gingi- monly associated with gingival recession that she or
val recession (Table 2). he can personally control. Especially with patients
susceptible to gingival recession, mitigating modifi-
able conditions will probably improve the chances for
Tobacco use, including smokeless
better management and prevention of gingival reces-
tobacco
sion. Such conditions include clinically detectable
Tobacco use is an important gingival recession risk plaque, damaging oral hygiene methods (such as
factor (8, 39, 43, 79, 84). Studies demonstrate that forceful toothbrushing), damaging oral habits (such
users of smokeless tobacco tend to have more severe as picking teeth with sharp objects), damaging eating
gingival recession and clinical attachment loss, and a habits (such as patients with lower incisors that have
greater proportion of sites with higher values of the thin, delicate tissues, who habitually bite into foods

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Merijohn

such as very hard fruit and thick crusty bread) and However, with our present understanding it can be
oral jewelry in proximity to gingival tissues. considered that orthodontic treatment in general and
Using the checklist during treatment/procedure its retention phase may be considered as risk factors,
planning can also help guide clinicians toward proce- especially for the development of labial gingival
dure options that can potentially decrease the risk of recession (50, 77). Therefore, patients susceptible to
damage to tissues (e.g. avoiding mechanical tissue gingival recession will be more so during or after
abrasion/removal; preventing chemical and/or ther- orthodontic treatment, especially if they present with
mal injury). This can help improve the odds of pro- modifiable conditions (Table 2) that are not
viding better management and preventing gingival addressed before, during and after treatment. For
recession. In addition to the conditions already dis- example, patients who use forceful toothbrushing
cussed other modifiable local dental conditions methods on thin (delicate and fragile) facial gingival
should also be considered in treatment planning for tissue have a greater risk for gingival recession if
gingival recession. Three of the modifiable conditions orthodontic treatment facially proclines these teeth.
listed in Table 2 are discussed below. Prudent interdisciplinary dental teams can deter-
mine whether prospective orthodontic patients are
Subgingival restorations
susceptible to gingival recession, consider adjusting
Clinicians should determine if the physical character- tooth-movement plans and outcomes, and recom-
istics (e.g. rough surfaces or margin–tooth surface dis- mend appropriate interventional periodontal therapy
crepancies) of subgingival restorations provide – especially for teeth at increased risk for gingival
protected habitats for microorganisms that can lead recession. Even with such measures, susceptible
to biofilm-induced or plaque-induced inflammation. patients should be informed about gingival recession
If appropriate oral hygiene and periodontal treatment risks before treatment begins.
do not mitigate the inflammatory response, clinicians
can recontour and polish these restorations or
replace them with new restorations designed to Clinical decision making: just
decrease risk exposures. monitor or a candidate for surgical
Oral appliances
evaluation?
In considering the role of fixed or removable One of the more critically important and fre-
orthodontic appliances/retainers, removable prosthe- quently asked questions about gingival recession
ses, mouth guards and occlusal guards, clinicians is, ‘Can we monitor it or do we need to consider
should evaluate whether these appliances impinge on surgery?’. Therefore, are there practical criteria the
gingival tissue or if they are too close to the fragile soft clinician can use to help guide the clinical deci-
tissue. In addition, clinicians should determine sion-making process?
whether oral appliances contribute to tissue inflam- All patients susceptible to gingival recession should
mation or mechanical tissue irritation/injury and have ongoing careful monitoring as part of their over-
whether these devices should be modified or replaced all case management. Monitoring is an active ongoing
with new ones that mitigate these risk exposures. process that engages both clinician and patient. It is
particularly important at clinical decision-making
Orthodontic tooth movement in patients susceptible
points (e.g. examination/evaluation appointments,
to gingival recession
patient consultations/treatment plan presentations
Contradictory statements can be found in the liter- and dental hygiene appointments). The checklist and
ature regarding orthodontic tooth movement as a visual guide examples illustrated in this paper are
risk factor for gingival recession. Some studies con- useful chairside tools that can facilitate the process.
clude that it is not a relevant risk factor (3, 25, 85), The desired goal is that all positive checklist items
whereas others demonstrate an increased risk (9, (Table 2) are reviewed with patients during appropri-
11, 45, 50, 77, 82, 90, 93). It has been suggested ate appointments until there are no more positive
that a better understanding of the relationship checklist findings. Susceptible patients, including
between orthodontic tooth movement and gingival those who have had or will have surgical interven-
recession will be gleaned from prospective, ran- tions for gingival recession defects, should have indi-
domized controlled trials that include assessment vidualized treatment/procedure plans developed that
of periodontal parameters before, during and after address all modifiable conditions. Although individ-
treatment (45, 53, 77). ual patient circumstances may determine otherwise,

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Management & prevention of gingival recession

practical clinical criteria are proposed below as gen- tooth movement; (iii) oral appliances that contact the
eral guidance to support clinicians in making appro- tissue; and/or (iv) potentially damaging dental ther-
priate clinical decisions regarding patients presenting apy (Table 2).
with gingival/soft-tissue recession; probing depths If surgery is the treatment of choice, treatment
extending beyond the mucogingival junction; should be directed to achieve key surgical outcome
absence or narrow band (<2 mm) of keratinized tis- objectives. The minimum required outcome of a sur-
sue; and/or thin (delicate and fragile) gingival tissue gical procedure for problematic gingival recession is
(4, 60, 63). These major clinical decisions can be cessation of further recession. This necessitates the
grouped into three broad categories: correction/improvement of presenting problematic
 When to monitor a patient without surgical evalu- tissue factors/conditions (5), including: thin gingival
ation. tissue; probing depths extending beyond the
 When is the patient a candidate for surgical inter- mucogingival junction; absence or narrow band of
vention? keratinized tissue; shallow vestibular depth problem;
 When should the patient be referred to a peri- frenum position problem; and/or tissue deformities
odontal specialist? (e.g. clefts or fissures). When root coverage is a surgi-
cal treatment goal, the selected approach should also
correct for all of the above presenting problematic tis-
When to monitor gingival recession
sue factors/conditions. Otherwise, after a root-cover-
without surgical evaluation
age procedure the tooth can remain susceptible to
It is proposed that clinicians can monitor gingival progressive gingival recession.
recession without surgical evaluation in such patients
when all of the following criteria are met: (i) no docu-
When should a patient be referred to a
mented evidence of progressive gingival recession; (ii)
periodontal specialist?
clinical attachment loss (probing depth plus gingival
recession) ≤5 mm; (iii) gingival recession <2 mm; and Although there is no single, specific clinical tipping
(iv) all of the above and the teeth in question will not point that can be used as a guideline for all
have subgingival restorations, orthodontic tooth patients in deciding when to do so, referral to a
movement, oral appliances that can or will be in con- periodontist is appropriate when one or more of
tact with the tissue or dental therapy that is poten- the following criteria are met: (i) the patient prefers
tially damaging to thin gingival tissue (Table 2). referral; (ii) the dentist decides that she/he does
not possess sufficient knowledge, skills and/or
experience to provide the patient with the neces-
When is a patient a candidate for surgical
sary surgical outcome objectives; (iii) the dentist
evaluation?
decides that referral is in the best interest of the
It is proposed that a patient presenting with the con- patient; and/or (iv) the dentist decides that clinical
ditions noted above is a candidate for surgical evalua- uncertainty dictates additional input into the case.
tion when at least one of the following criteria are Situations that increase clinical uncertainty include:
met: (i) documented evidence of progressive gingival some medically complex patients; advanced-severe
recession; (ii) persistent gingival inflammation (e.g. gingival recession defects; multitooth involvement;
bleeding on probing, swelling, edema, redness and/or and/or recession risk that cannot be readily modi-
tenderness) despite appropriate therapeutic interven- fied. According to the Principles of Ethics and Code
tions, in combination with clinical attachment loss of Professional Conduct of the American Dental
>5 mm and/or gingival recession ≥2 mm; and/or (iii) Association, “All dentists. . . have the obligation of
persistent gingival inflammation despite appropriate keeping their knowledge and skill current.” With
therapeutic interventions and association of the respect to consultation and referral, “Dentists shall
inflammation with shallow vestibular depth that be obliged to seek consultation, if possible, when-
restricts access for effective oral hygiene, frenum ever the welfare of patients will be safeguarded or
position that compromises effective oral hygiene advanced by utilizing those who have special skills,
and/or tissue deformities (e.g. clefts or fissures). The knowledge, and experience.” (7). Every clinician
patient is also a candidate for surgical evaluation if must self-determine whether they have the knowl-
found positive for any of the above three criteria and edge, skills and experience to monitor or plan/per-
any of the following are planned for the teeth in ques- form surgery on patients for the management of
tion: (i) subgingival restorations; (ii) orthodontic gingival recession defects.

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Concluding remarks 6. American Dental Association. Glossary of Dental Clinical


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The author acknowledges Gary Armitage for his
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