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Arti Kripalani
Part I P.G.
K.M.Shah Dental College
Introduction
Periodontal ligament
Physiologic tooth movement
Optimum orthodontic force
Types of forces
Histology of tooth movement
Hyalinization
Phases of tooth movement
Theories of tooth movement
Orthodontic treatment is based on the principle that if
prolonged pressure is applied to a tooth, tooth
movement will occur as the bone around the tooth
remodels.
This include:
A)Tooth Eruption.
B)Migration or drift of teeth.
C)Changes in tooth position during mastication.
Tooth eruption is the axial movement of tooth from its
developmental position in the jaw to its final position
in the oral cavity.
a)Blood pressure theory:
The tissue around the developing end of the root is highly
vascular.
This vascular pressure is believed to cause the axial
movement of teeth.
b)Root Growth:
The apical growth of roots result in an axially directed
force that brings about the eruption of teeth.
This theory was rejected.
This was pointed out for the first time by Stein and
Weinmann.
Physiologic tooth migration usually is related to mesiodistal
movements. However, the teeth also exhibit a continued
eruption, even after full emergence, accompanying the
growth in height of the alveolar processes.
PDL stretched.
Distance between alveolar process & tooth is widened.
Increased vascularity.
Mobilization of fibroblasts & osteoblasts.
Osteoid is laid down by osteoblast in PDL immediately
adjacent to lamina dura.
Lightly calcified bone mature to form woven bone.
Secondary remodelling changes:
Bony changes also takes place elsewhere to maintain the width
or thickness of alveolar bone. These changes are called
secondary remodeling changes.
Both light and heavy forces displace the tooth to same extent .
1. Collagen.
2. Hydroxyapetite.
3. Collagen hydroxyapetite interface.
4. Mucopolysaccharide.
As long as the force is maintained ,the crystal structure is
stable & no further electric effect is observed.
ORTHODONTIC FORCE
compression of PDL
formation of aneurysms
Fluid is squeezed out of vessel wall
• Prostaglandins
• Leukotriens
Others • Nitric oxide
Von Euler (1934) – prostate fluid.
Imp role in stimulating bone resorption.
One of the chief mediators of inflammation cause an
increase in intracellular cAMP and Ca++ accumulation.
Cell membrane phospholipid
phospholipase- A
ARACHIDONIC ACID
Cyclo-oxygenase lipo-oxygenase
pathway pathway
- cAMP
- cGMP
- IP3
The receptor activator of nuclear factor kappa B ligand
(RANKL), its decoy receptor (RANK) and
osteoprotegrin (OPG) were found to play important
roles in regulation of bone metabolism.
Upon activation using light forces can also produce pain for
few hours to few days.
This also greatly depends upon individual pain threshold.