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  • For Printing Pathophysiology DM

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    kat garcia
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    Diabetes mellitus can develop due to predisposing genetic and lifestyle factors as well as precipitating factors like obesity and age. The pathophysiology involves either impaired insulin production by the pancreas due to genetic defects or environmental influences, or the body becoming resistant to insulin's effects. This leads to hyperglycemia as glucose cannot enter cells and accumulates in the bloodstream. Over time, high blood glucose can damage various organs like the kidneys, nerves, eyes, and heart, potentially resulting in renal failure, stroke, cardiac issues, and even death.

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    For Printing Pathophysiology Dm

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    Diabetes mellitus can develop due to predisposing genetic and lifestyle factors as well as precipitating factors like obesity and age. The pathophysiology involves either impaired insulin production by the pancreas due to genetic defects or environmental influences, or the body becoming resistant to insulin's effects. This leads to hyperglycemia as glucose cannot enter cells and accumulates in the bloodstream. Over time, high blood glucose can damage various organs like the kidneys, nerves, eyes, and heart, potentially resulting in renal failure, stroke, cardiac issues, and even death.

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    © All Rights Reserved
    Descărcați ca DOCX, PDF, TXT sau citiți online pe Scribd
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    112 vizualizări1 pagină

    For Printing Pathophysiology DM

    Încărcat de

    kat garcia
    Diabetes mellitus can develop due to predisposing genetic and lifestyle factors as well as precipitating factors like obesity and age. The pathophysiology involves either impaired insulin production by the pancreas due to genetic defects or environmental influences, or the body becoming resistant to insulin's effects. This leads to hyperglycemia as glucose cannot enter cells and accumulates in the bloodstream. Over time, high blood glucose can damage various organs like the kidneys, nerves, eyes, and heart, potentially resulting in renal failure, stroke, cardiac issues, and even death.

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    din 1

    PART II.

    PATHOPHYSIOLOGY OF DIABETES MELLITUS

    Precipitating factors:
    Predisposing factors:
     family history of DM
     Age-> 40 YRS OLD or Late
     obesity
    adulthood
     Production of  Age above 40
     Sedentary Lifestyle
     Diet (Excessive sugar intake) glucose by the
     Therapeutic regimen- Non- liver
    compliance to Insulin therapy

    Altered pancreatic Impaired insulin Inability of glucose


    insulin secretions production to enter target cells

    Peripheral resistance
    UTI Vaginitis
    to insulin

    Glucose accumulation in the blood or


     Urethral flora HYPERGLYCEMIA
    Diminished (FBS  to 180 mg/dL)
    intracellular
     Acidity of urine reaction

    Low sodium levels


    Dilution of  Concentration of
    GLYCOSURIA (Hyponatremia) ECF
    SODIUM in ECF

     NAUSEA
     VOMITING
     FATIGUE
     HEADACHE
     GRADE 2 PITTING EDEMA
     DECREASED URINE
    OUTPUT

    Blood
    glucose more
     Pressure than the
    Proteinuria Stressed Filtration
    of blood
    Thickening in System Renal
    vessels in
    the glomerulus the urine threshold

    Diabetic nephropathy Renal failure End-Stage Renal Disease

    Potassium ion retention F & E imbalance POLYURIA & Diffuse glomerular sclerosis
    ALBUMINURIA

    Cardiac arrythmias Increased viscosity of


    Stroke  Blood pressure
    blood

    DEATH

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