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Introduction
Cerebrovascular accident
The sudden death of some brain cells due to lack of oxygen when the blood flow to the brain
is impaired by blockage or rupture of an artery to the brain. A CVA is also referred to as a
stroke. Symptoms of a stroke depend on the area of the brain affected. The most common
symptom is weakness or paralysis of one side of the body with partial or complete loss of
voluntary movement or sensation in a leg or arm. There can be speech problems and weak
face muscles, causing drooling. Numbness or tingling is very common. A stroke involving
the base of the brain can affect balance, vision, swallowing, breathing and even
unconsciousness.
Causes:
Risk Factor:
1. Hypertension – leading risk factor for coronary heart disease and stroke
– treatable and can be controlled.
2. Modifiable by change in lifestyle
a. smoking
b. elevated serum cholesterol
c. obesity
d. heart disease
3. Modifiable by Medical mean
a. Transient Ischemic Attack
b. Asymptomatic carotid bruit
c. Diabetes Mellitus
d. Increased blood viscosity
e. HPN
4. Non – modifiable risk factors
a. age
b. sex
c. race
d. previous stroke
Types of Stroke by Etilogy:
1.Hemorrhage stroke (intracranial hemorrhage)
• 5% of all strokes
• two division
a.Intracerebral (10%) – due to rupture of weakened vessels
within brain parenchyma as result of Hypertension,
arteriovenous malformation or tumor
b. Subarachnoid (5%) – result from aneurismal rupture of a cerebral artery with blood
loss into space surrounding the brain; evolve over 1 –2 hours.
2.Ischemic Strokes (remaining 85%)
• Large (40%) or small (20%) vessel thrombosis
-most commonly occur in presence of atherosclerotic cerebrovascular disease
-vascular changes or lipohyalinosis found in small deep penetrating arteries as associated
with chronic hypertension can lead to small vessel thrombosis.
-rapid or prolonged interval of onset and may lead last many hours
• Cerebral embolism (20%)
-usually a cardiac origin
-frequently result of chronic ischemic cardiovascular disease with secondary ventricular wall
hypokinessis or artial arrhythmia – both conditions increase risk of intracardiac thrombus
formation
-quick onset and fully develop in a matter of minutes
Temporal Classification of Stroke
1. Transient ischemic attack (TIA)
–neurologic symptoms develop and disappear over several
minutes and completely resolve in 24 hours
–most frequently associated with atherosclerotic carotid artery
disease
2. Reversible Ischemic Neurologic Deficit
–etiology unknown
–likely the result from small infarctions (Lacunes) of the deep
subcortical gray and white matter resulting in only temporary
impairment
3. Stroke in Evolution
–describe an unstable ischemic event characterized by the
progressive development of more severe neurologic impairment
–often associated with active occlusive thrombosis of a major
cerebral artery.
–Once stable called Complete Stroke
–Most important sign – Intellectual Regression
The client’s pulse rate is 98 beats per minute, his respiratory rate is 20 breaths per minute,
temperature is 36.8°c. BP- 110/ 80
General appearance
The client is in medium frame with stooped posture, the client is bedridden since he was
admitted to the hospital last January. 5, 2011. Is not well groomed with body odor.
Mental status
The client is conscious and cooperative . The client has low comprehension and difficult to
talk.
Skin
The client’s skin is of normal racial tone which is brown. It is dry and smooth. The skin
turgor is wrinkled and loss of elasticity. The body hair is evenly distributed. She doesn’t have
any edema. But she has a skin lesion on his right elbow.
Nail
The client’s nail shape is convex clubbing, the nail is rough and the nail bed is pink. The
capillary refill is within 3 seconds and these is an absence of beau’s line.
Eyes
The condition of his eyes is straight normal; the eye brows are evenly distributed. Eyelids
have effectively closure. The blink response is bilateral, eye balls are symmetrical, bulbar
conjunctiva is clear, the palpebral conjunctiva is pink and the sclera is white. The palpebral
slant is aligning with the tip of the pinna. The corneal sensitivity reflex is present cornea is
transparent, the color of her eyes are brown, the shape are equal, it is uniform in color. Pupils
are equal in size. Pupils are equally round and reactive to light and accommodation. She can
execute the occular movements. The lacrimal apparatus are moist.
Ear
The color of the ear is of normal racial tone which is brown, it is symmetrical. The
alignment of the pinna is symmetrical. The pinnas are elastic and recoil when folded. The
mastoid process is tender. The auditory canal contains some cerumen, the color is brown and
there is an absent of discharges.
Nose
The color of the client’s nose is of racial tone which is brown. His septum is in the
midline. The mucosa is pink, nostrils are both patent, nasal flaring is absent. Landmarks are
visible. Sinuses are non-tender. There is an NGT in his right nostrils.
The lips is symmetrical and pink, the consistency is smooth, buccal mucosa is pink, the
gum is pink, the tongue is in the midline, the color is pink and it is smooth. The tongue
movements are not that smooth. Its texture is rough.
Neck
The neck has involuntary movement and with resistance, the muscle strength 3/5. The trachea
is in the midline, thyroid is in the midline and it is smooth. Maxillary lymph nodes are
palpable.
Upper extremities
The client cannot resist force when asked to resist. Muscle strength is 3/5. The peripheral
pulses are equal. The IV site is in his left arm.
Lower extremities
The client cannot resist force when asked to resist. Muscle strength is 2/5. She doesn’t
have any deformity. The peripheral pulses are equal.
VI. Pathophysiology