ccAcyanotic ʹ left to right shunt!

cc
DISEASE DIFFERENT WHAT IT IS S/S WHAT RISKS SURGERIES CLINICAL
TYPES NURSES DO THERAPY
fetal ductusarteriosis 0 

 Lasix No long term Ligation Abx before dentist
O

 doesn͛t close in first c c problems if treated Closes hole
weeks of life Tachycardia Indomethacin to c c




 c c close opening in Short lifespan if VATS

 



 Makes left side of Bounding pulses premature babies untreated Closes hole w/ 3



 heart work harder c c small incisions
c Wide pulse Fluid restriction w/ c
Pulmonary pressure meds Coil
congestion/resistance c c Coil in hole
c Hypotension Prophalxis to prevents mixing of
Hypertrophy (heart) c prevent blood
c Poor development endocarditis c
Increases circulation c
to pulmonary system Thrill

Ostiumsecundum Opening between the ?asymptomatic? CHF Transcatheter Could close in first


 
 of septum atria c c device during 4 years of life
c c Over time poor Pulmonary cardiac cath c

 Ostiumprimum Increased flow of O2 growth hypertension (secundum) Dacron Patch for
Flows from left atrium to right Of lower septom blood to the heart c c med-large holes
atrium c c Over time CHF Dysthrhythmias
Sinus venosus c that cause blood
Of superior Soft systolic clots
venacava& right murmur over
atrium pulmic artery

Increase in pulmonary flow left to right shunt is oxygenated blood flow

ccAcyanotic ʹ left to right shunt! cc

 DISEASE DIFFERENT WHAT IT IS S/S WHAT RISKS SURGERIES CLINICAL
TYPES NURSES DO THERAPY

Increased blood Dyspnea Lasix Only a small number Pulmonary banding Most close on own
Ñ


 volume pumped to c c cause problems used w/ symptomatic by 6 months
the lungs Tachapnea Digoxin c infants that can͛t c

 c c c Bacterial endocarditis take Lasix or digoxin Most children
Decreased CHF O
 
 c c respond to surgery

 pulmonary vascular c 

 Pulmonary Surgical patching in and catch up on


 resistance Pulmonary infection obstructive Vascular infants with poor growth




 c c disease growth



 Right ventricular wall ï



 c
hypertrophy 
 Closure with



 transcath device

Narrowing near Dizzy, h/a Check bp in all 4 Can reoccur Dilation done in first Causes persistent
°


 insertion of the Fainting extremities c 3 months of life hypertension in
ductusarteriosis c c Hypertension c adults

 c Nosebleeds Prophylaxis for c Repairs done in 1
st
c
Increased pressure c endocarditis Aortic aneurisms year Could hear systolic


 proximal to defect Develop CHF by 3 c c murmur
head and arms months Stroke Balloon dilation occompanied by and
c c c during cath for intital ejection click
Decreased pressure Poor bp in legs, weak Ruptured aorta relief and
to obstruction, body pulse recoarctation in older
and legs c children
c High bp in arms c
Obstructs systemic c Surgical resection can
blood outflow Neck and head be performed w/ the
pulses bound subclavian artery
c used as a patch
Kids complain of
tingling in legs
c
Systolic ejection
murmur

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 Is a combination of Decreased pulmonary 


 Hypercyanotic Hypercyanotic Total repair before Monitor for acidosis
these 4 defects: blood flow c episode: episodes 6months w/ severe c
c c # Knee chest, initially c tep/blue spells Monitor for
Pulmonic stenosis Usually right to left c give O2, postpone Emobli c prolonged
c shunt unless vascular ( 
  stressful procedures, c Corrective surgery if unconsciousness
Right ventricular resistance is higher c (
 Seizures no tep spells c
hypertrophy than pulmonary
(  c c (cardiopulmonary Not all kids are cured
c resistance then it is a c Prophilaxis for LOC bypass surgery) by surgery
Ventricular Septal left to right shunt °( endocarditis c c c
Defect c Cardiac arrest Surgery improves
With a large vsd c quality of life
pressures may be = seizures
which meanes c
direction depends of death
pulmonary and
systemic resistance



 No communication Administer IV Corrective surgery Maintain pda for
(





!"  between systemic Cyanosis at birth, it prostaglandins to (arterial switch at one systemic circulation
If vsd is present risk
 and pulmonary and progresses to hypoxia keep ductusarteriosis week old) c
of CHF goes up Must rest when


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 systemic circulation and acidosis open and increase c
c



 c c blood mixing Balloon feeding
Pda will close in thhe




 Life threatening at O2 doesn͛t improve providing an sat of 75 atrilseptostomy c
neonate period


()
 birth if septal defect cyanosis or maintaining during cath as first Gross growth failure
 

( or patent c cardiac output stage starts at 2 weeks

 
 ductusarteriosis are CHF c c c


 not present c Can have pulmonary Baby will die w/o
c Tachapnic & aortic obstruction surgery
Patenetframenovale c or aortic stenosis c
is most common Murmur is tsv is
associated defect present
c

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 Failure of normal Vascular systemic Digoxin Close the vsd, excise 10% mortality
septation and division defect c pulmonary arteries c
of embryonic bulbar c diuretics from aorta and attach Long term prognosis
trunk into the Cyanosis at birth to the right ventricle unknown
pulmonary artery and c w/ homograft
aorta Increased pulse c
c pressure 

(


A single vessel c 
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overrides both Systolic murmur




ventricles and c c
provides circulation Clubbing 
(



for pulmonic, c 

systemic, and Chest contractions w/
coronary circulation breaths
c
Poor feeding
c
Poor growth
c
fatigue
Decreased pulmonary blood flow Right to left shunt Obstruction of pulmonary blood flow Right Left
A
S
V
/
V
S
C

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 Incomplete fusion of Seen in down Prophylaxis for Endocarditis Palative pulmonary Risk for arrhythmia
°  
 endocardial cushions syndrome endocarditis Pulmonary artery banding to and mitral valve
Low atrial septal Mitral regurgitation obstructive vascular decrease blood to deficiencies post
defect that is disease lung so baby can operatively
What type of shunt grow enough for
continuous with a
hight ventricular Poor growth surgery
septal defect and Tachacardia Patches/valve repairs
clefts of mital and and replacements
tricuspid valves Tachapnea

Blood flows between Resp infections

all 4 chambers Cyanosis w/ crying or
Generally from left to feeding
right 




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 Underdevelopment O
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  Prostaglandin . 
 w/o surgery baby



 of left side of the infusion (keep open)
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survives 3 days
heart, hypoplastic left + 
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    Multi stage surgery heart transplant is
ventricle/aortic  
 


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atresia O


 




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Small lft


 



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ventricle/aorta, 
 

  
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Aortic/mitral stenosis   





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1 O   Ñ ° %  %'    "ï" °% 
Cardio megaly Shows hole size Shows hole or Shows Boot shaped Egg shaped Cardiomegaly & Cardiomegaly Cardiomegaly
°1 cardiomegaly cardiomegaly heart, enlarged heart on a enlarged aorta detentation of
rt ventricle string aorta
Enlarged lft Shows hole or Atrial & bundle Rt ventricle Rt ventricle Rt&lfthytrophy Hypertrophy lft
°'
 ventricle cardiomegaly branch block hypertrophy hypertrophy ventricle
Left to rt shunt Shows shunting Dilating ventricles All 4 heart Abnormal Absence of 2 Small lft Aorta size,
°"% defect / septal defect defects postion of semilunar valves ventricle (1
st
functioning of
great arteries trimester) valves
Blood glow in heart
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Done if other If other tests are Increased O2 in rt Blood flow Enters aorta All defects and To prep for See site of
°  ° results are unclear unclear atrium though all through right structural problems additional coarction
° " regions ventricle interventions
Makes diagnosis,

dilated rt ventricle
(
 b/c of blood
overload/shunt
 size
Shows hole size




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Increased hgb, Increased
2  hct& clotting hct&hgb


 time