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INTERVIEW PREPOUESTIONS
#1tape, staples #2
~~It:..
Nylon or polYP.tQPylene Os TngoD1JDl Os SupI~cuJare. (mL~~ for avulsionfx ta1ar head) Lumbricales, Nn 2nd Inf~on, vasccompromise, DM* (reJative; only wI phmol) B/c blood will dilute chemical Zadik total matrixectomy Tenninal Symetotal mabixectomy .062(anythingsmallerwouldbreak) Dorsal 2ad met For poiydactyly...based position of on duplicationin longit & tIBnsvplanes PIPJfusion, Girdl~~., Well No, li~rnent is alreadyweak!
Mulders, SuI1ivans,Bei'kowskys
~~
6v,d...
("4-,,5')
-Which
matrixectomy allows remova1 of SlIbungual exostosis d/t exposure? -Most radical procedure for dyStrophic nails?
-PIPJfusion...wbatsizeK-wire if aossing MPJ"! -Peg-in-hole... what PP cortexis left intact? -Axis of foot? -What is the Blauth andOlssonc1a~~~tion? -T~~!forPDS7 -Would you give a steroidinjectionfor PDS1
-What signs do you see wI nemoma?
ReprodIlclble pain wI Jateral compressionofFF(De1n'Oma) -Lachmann test = ? For PDS; vertical stressIdrawer test -complication of osteotomy lengd1enjng forbIa chymet? stretchNV/NVcompromise -What did DuVries believewascause tailors bunion? of l)hypertrophyof S.t wide ~ MI1I -2)con~~1taL1y -What is the SHIP ifn!'l1mt? -c/I to hemi jmpJants? -Radiographic separation b/w implant andbonenormal till? -What % of infectionsfrom implantsoccur w/in 1 mo to 2 yy plop? -What is the Pirogoff'amputation?
-Which preserves more calcaneus,Boyd or Pirogoff' ampuI:ation? -Should you remove cartilage when amputating?
cartilage
-Berkowskysign is?
3)latemlbowing of ~ MI1I SgarJatto HSimmertoe Imp~ Prosthesis (total imp~ no hinge) UncontroDed pronation,arthritisboth sidesof joint 3 months 60% (or greaterthan Y2) Modi:fiOO prox Symes preserves calcaneus Boyd
Yes; nidus for infection
surface delays healing
and uneven
and can -> IH'e8StJre points
-Who
-MPN supplies? -Neuroma is plantar or dorsal to DTn..? -Min distance b/w incisions on dorsum foot to avoid skin DfCI"OSis? D8I ). . ~ -Epineurolysis = ? IDterdigitaln. freedfrom surr S.tsw/o nerveexcision -Why is demerol called the bear bugger? Its for shivering 1aisesyourte mperabJre -Why are bunions more common in females? Ligllment wea1cP!ning hormones d/t -the ligament that is weakened is met-phalangeal (medial)
-what
-How do you treat brown recluse spider bite? -4 things you release in HA V lateral release andinordel'?
I) DM, 2)ETOH, 3)leprosy Dapsone Lateml capsule, DTJL, addlL fibular sesamoid
-How would you diagnoseCMT disease? -11mit of insulin will lower ~ blood glucose how much?
-What NSAID is used to prevent
heterotopic ossificationfollowing surgery? IIJdomedlacin -11mit ofblood I3isesyour 8gb and Hct by how much? Hgb by I aDdHct by 3
-Osteochondrosis of:fibular head?
-What is antidote to epi? -What is presenting symptom of MS1 -RA pt, what are 3 things you must do before surgery'!
Ritters disease Phenrolamine Optic neuritis C-spiDe xrdY. cover for steroid suppression,cervical collar
factorb/wPA andRA?
Noosteoporosis PA wI
-PI anergicto amideand ester,what would you use? Benzoylaioobo1 -Which NSAID causes dose-related headaches? Indomethacin -IR Ray- what is the cmciateanastomosis? l)medial branchof lit plantarmetatarsal artery 2)latera1 branchof"" " " 3)hallucalbI3Dchof medialplaDtarartery
4)11tPMA
-~ how to perfOIDl bioorrectlODal a Austin. Offset V? -What is tbe difference b/wKalishand
--
2mbonecut is madethat extends 8041/0 tbrougb the met to removea medialwedgeofboue Offset V canswivel to COnedPASA aIKI the ~is at metaph~eal junction Waterman/WatetmaD,.Green ~ +1;3 -
~ever
1)Vascu1aringrowth 2)OsteobJastproliferation 3)Osteoinduction 4)OsteocaDduction 5)Graft remodeling Large bony defects Demineralized (DBM) or decalcified bone graft
for
inlay bonegmft technique? Evans calcaneal osteotomy and bone graftprocedu re -What was the Papineau technique designed fot'! Rapidreva scuJarizatjon -List the DBM products. GI3fton, Dynagraft, ~ AlloMatrix -MC. NM causeof cavus foot? CArr (*NM is most common cause of cavus foot) -Posteriorcavusis ..
-Anterior
indicated
-What is the only osteotomy for (3VUSfoot that does Dot remove any bone 'Wedges? -AIUerior tarsal bJImel syndrome is 'What1aVe1 -FDDcaioDal taIsal tuond syOOrome d/t? is
Japas (disp~ V-siJaped ostecKomy) DeepPeroneal (~4IJDaJ11JDder N. iDfericxextmSOr retiDaa1lmn) Pronation...~~ rotmiooof leg tigiltalS flexor JetjDaCUlum; forced eversionof foot also stretd1es andoomprrsses n. contents TI' of
-#1 amplicatiooof ~
transfers?
jJ1terosse()us ~
andtransfenedto LC
arethe 21B:OCOhrr~ PT1U7 for -Wbid1)X'oced1 rq>1ac:e ~ ATf'L. {]'"I., alMlPI'f1.? -OJoDdromalacia ? = -Gold std for severe. JBiDfDI DJD'l AI -How do you fix dO:xn/pt1Wangu]ar deformity of pmiously fusedAI? -Oo1yFDA -awroYedAI ~m7 -Oo1yAJ impJantthat canbe used. post failed AJ fusion,aIMiwith A VN of talus?
-What
-What 8I1!,J1atjon of a Z-p1asty gives
~b, FDL transfer S~ff. RoseOOahl-Jmscn; utilize fascialata both Arthritic<:baDge aItiaJlarcartiJage in ADk1e fusion Wedgeresection tibia of Agility Ankle ~~ 60 degrees implant(~ DDIst iDIxt) be
the greatest skin l~gthfmin g? -Why are angles~ 1ban S CX' 4 greata:than
-Stages
l)PJasmatic stage
2~onQge 3)Reorgani~on stage 4)Reimlervation stage
-Pinch
test?
To determiDe RSn...~
a~
d1ance of~p0I3
non?
-Full thickness skin grafts are iDdicatM for...? -What bugs do you need to wony about
-cutaneous
Accelerated action of ~1!etic ClDKlrOO1astoma (CB) -IV drug for It wI NVl Promethazine (Pbeuergan) -Where is common peroneal n. block performed? 2 cm distal to fibular ~ -Pt bascavus~ aodiJD-~ ~.. what dL~~~1 l..f-!l-~.J1ar DysIJq)hy -Tmf toe is caused by: Hyperextension compression wI -Clinical findings wI ~ sclerosis? Periungual fibroma, ~ adenoma. tbicr.~P4 skin -Li~-iDe toxicity affects what system first? CNS; CVS is affected2m
-First Iadiographic sign of osteODecrosis? -What haA)eDs when you give an amide anesthetic to a It wI Iiver problems? -MC. bmIOrin epiplysjs?
CuIaneouspedicle~ : be wide enoughto supportflap AtteJiat depeodmton dimeosioDS artery am length of of 8rtelYterritory. not 00 pediclewidth Sclerosis
-Pt wI RayDa1xls aDd calcinosis has... -MC. location of osteoid osteoma? -What lab value is high wI bone tumor?
-MC. fungalinfection L'nD1Qnosuwressed? in -Inaease in cal~~ fat pad is ~ wI what di.~~ -PurposeofiDsertingan~~iIIto lit MPJ after performKeller? -Can roo give ~ to a p wI g12~?
-STJ easiest to displace in what position?
r~dida , Acromegaly
Acts asa spacer No
Inversion EquiDUs PT
Cavemosa hemangioma
-SY!!~~c nJptme seen Weber wi _7 -one of thediffeI~ diagooses Kaposis7 for -Sickle aisis t1'eatmeJ]t: cell
seenwi: -~ in 1xme width ~ wi: -HSllllDaIk LisFI3DCS fradm'e1 -Excise~ ~oids; what might result?
-Inaease in bone width
Met
-Six cardinal stages/signs of cbrooic osteomyelitis: 1)I3diol~ - ostcolysis, decreasedck:osity, Jossof ttabeculation
2~
3 )sequestra
repair
density
for pus)
--
u,~~..~5)cloaca
siDUs ttact
in 00De~ decreased
--
density
(*o1Jtlet
-Fmn, 1KBl~ ~ !~~ m dc:Ksum of foot; which of the following is it: ganglioniccyst. lipoma, granuloma, fibroma'! -Pt JB'eseDtS b/o AI sprainsam ankle wi occasionally locks; what is the problem? -Avulsion ~ of calc aIderiorP'Oce5S d/t1 -Incision ~ over tendonmay result in? -TxforRSD: -MC. bonetumor in ca~j: -Abx for PCN-allergicpt wi DM, aId liver dx with aIbnres positive for gram ~ nxf! -Clinical ha11Tn1ni1: of pityriasis rosca1
-Male IX wi toes that bJIDblue. thm 1M?
Granuloma
om talus
Bifurcate ligament Adhesion Pr, TENS, Bier block, Psychotherapy UnicaDa"3llxme cyst (U~) Cipo HeIak1.-tdl TAD 1~e7 2)pro1iferative, 3)~Jing 1)Stimulation,2)conwlsioos,3~on, d/t directpressureon arteries ~ by Perpeodicular axis of rotationof toe to
-3 PJaSC'S wound repIiI1 of -Toxic drug reactionto LA manif~s how? -Vnl~'s isd1emia ? =
-Skin wcdge must be placed how for skin plastics?
-What is Simons Angle?
4)death edema
Line bi_~~ ta1arheadaIxi extcnds diSIallytowards I-met (APview); ifitfal1s~to )-Mr, is negative(MA) ; falling ~ to 1- Mr = (+) (rEV) SimpIifiM MA angle;bisectionr MY'aIxi bi_~OD IC * approx 3 degrees greata'than MAA
-What is
Engles Angie(AP)?
~y
subl~
.When~ ~
co~on
-Modified Heyman,Henxion and Sttoogprocedu Preserves Ie? plantMaIKilatera1 Jiga~. anduses3 dorsalincisions(mginal HHS = ~ ttaDsviJx:ision andre.!~~ an 1ip~k at LF jt) -UIt the ~ procedures MA: for
Bankart, Peabody and Muro, McCormick aJKtBlount, FowJer, Steyt1eraM VanDer Walt, Berman aIxI GardaIx1,Lepird, Brink aod Levitsky
-Which MA In'tx:cdme is a Juvara wI cresentic osteotomies of lesser mets? -Another mme for Steytler aIM1 Van Del" Walt?
-Ditr~
Lepird Modified BermanaDdGartJand TmriDsic (lEV) \ISRDrin.cic(posbJIal); intrinsic also basTN] subluxaDon. ~ ta1ar rotatim in AJ mcxtise
-MC.
~logic.
.only teIIdoDS el~~M wI TEV'l PeroDeals -8iDK)DS of 15 for TEV7 rule ---~$Wlqxali~ if.TCA.~S.~ > 15 -~m"VS.i Idease what is JO~? Cilalmferemjal re1ease~ ~ aDtedor ~!~~ an is -Primarydeforming~ in calcaoeovaJsus7 T~ of aDtrro-]afeJal ankle
-ccv ...Jcx:aboo DaViaIIaI7 of
-AchT is SU'etdIed which oongmihtl defOImity? ccv in -:MajordeformingF in Vertical Talus (VI)? PL In. = mjr deformingF wI iDaeasedproDatOry potential -Radiographic ~~s of VT7 For=! plaDtarfiexion 1ateral view TNJ disltx:atioo DOt alleviatOO whenfoot is in ~~ pJantarflexion -How to differeDbate CCV from VI' clinicany7 VI' =hollow anteriorto lat maIl~lus -T~ foraImIiIY1axis(aIMI &R)? 0.3 ml epi 1:1000 dilution 1M
-T~ -T~~
02
Fluids
for CP wlo sbcd: or respir rogin? DSW,morphine,labs -Earliest sign of shock? Tachycardia (tachypnea also an earlysign) is -Wbea is ~ in urinary ~ ~ in shockprs? Only in severe shockstates -What classofbelD(XIbage sIMJws c1assic $b(x:ksigns? ~ m -What classof hemOlThage life-threateoing? is C~ IV -How D11d lossofblo<Kiresultsin LOC? 5()8/0 ofblood loss -How muchblcxx1 you losewI pelvic fI3CtlDe1 6 uoits (3OOOml).._im~iatp.lylife tID'eat~ing! can
-Whidl
Hallux fraCbn (usnDP) RQ~~~~.~: DMA, PMA. fiOOJar plantaramrginala lDtta(&eOUs: peri~ plexus.nutrient a, metaph plexus
MOl for avulsioD=iDvcrsion injury vs Jones-foot QInDOtinvert
Desttudion, dellsity,debris.disorgani~tiOD,disl<ation
-Keratodermic
disease?
PityriasisRubIaPi1aris (PRP)
l)risk hypoglycemia 2)BS of 40 or less 3)i!!1~edia!~ recurrence of hypoglYc. following administration of glucose
.Whipples
triad =1
-Normal CrCI
=?
>SO
PCN G 10-20 million 1JDits/day Give C1~~mycin Tramnatic anemysm, A V fistuJa lDIra-aItiaJJarbemonbage Medial (Hemy), Dorsal (Muba13k), Combo *medial is fiIstest, dOIEalis 1east tnmrnmi7in~, mxl a>mOO most often usm is 21Ki 3MdegreeplaDlar burn or
-What do yon give a pt that needs tetaD1ls shot but is anergic to tetanus toxoid? *What is the IX:is PCN anergic? -What are 2 Jatesequela of arterial injmy? -An inaease mjt space on XIayplfra ~ =1 -Surgical approachesfor fasciotomy (foot)?
-When
do you hospitalize
burns?
anyzDddegrees> lSO/o body SA >6Oyyor<2yy ~ectrica1 orjnhalRtionburn -Signs ofbum sI1(x:k? = hypovolemicshoclc; circuJatory collapseand arterial VC -A very pr()Dated will obliteratewhat foot on a Lateralxray7 Middle TC facet...fake-outfor tarsalcoalition -What artbritidesarecommonlyfound at LF joint? OA (2a1 location),gout (r !DC ]DC location) *impt to ddx thesefrom Charcoton xray -MC. osteolytic conditions in the foot? IDfection,seronegative arthropathies, chronicgout -Non-erosive arthropathies (in the foot)? DISH, OA, acutegout, 01arc0t -Key to Charcot foot on xray? Subluxation -It space widening common wi what attbritis? Seronegatives
of choice for navicular stress
- T~ent
fraCttIresfor young, active pI? -Why are navicular Sb'ess fI3CbU"es m.c. in
ORIF (+) immOOilization InaeasedP (we(jgOO bones), and poor blood supply b/w
Posterior COmpartment:Sciatic n. divisions Anterior COmparbDent: Femoral n. Medial compat1ment:ObttuMor n.
Weakvastusm~a1is lnh1"bit oste<x:lastic activity -> blocks resorptionof boneaM canilage(why they're becomingpopularfor Olarcot) -Common C/I to bisphosp~~es in Charcotpts? CanIlOt if decreased use kidney function/renalfailure -How canyou stopthe progression Charcot? of ImmobilizeandNWB (w/o WB, Charcotdoesn't progress)
-Action ofbisphosphanates?
-Runners ~
=?
-Deadly triad
-What is
=?
diagnosisfor OM!
-Max dose of ASA qd? -Max dose AP AP qd? -Max dose Ibuprofen qd?
-MC. causeSlllsticity in adults? -DGI (dL~m~~ed gonococcalinfection) triad? do you do if get dorsiflexedhallux after MBA implaDtation?
Biopsywrong part ofbone 4 g; * >4g ->as gout, <4g -> hypemricemia 1 g/dose;4gi24hacute,6gi24h chronic~ (325 mg or 500 109tablets) 2.4-3.2gIday Stroke Tenosynovitis, polyarthI3lgias,dermatitis
(*teIK)S}'novitis
= unique
-What
=d/t s.t adaptation to keeping 1- MT position wI FF supjnatus...s.ts should eventually relax mI PL m/tendon should regain advantage
-Hallux
CaDDotdo AIxJH or Em., tendon transfers b/c using DTn.. as pulley for these tendoo 1I3Dsfers Secondary to chronic VD (seen in D M b/ c of autonomic ne1Jropathy causes~c -> VD)
--What ---
of Ml)!1k~gs7
-Whicll NSAID do you give pt wI ~ rd1ux? -M"inimtlm you should keep an MBA in? -Would you take an MBA out in very active kid involved in a lot of jumping?
y es~b/c jars MBA around aIKi is painful to kid (*once hit 6 mos-lyear, Sotskeep STJ in new position)
MBA: all same length. as size inaeases so does diam Futma: as size inaeases so do length aIKi diameter
-stagesof arterial occlusionin orderof ami occlusion? l)IC, 2)restpain, 3)gangreoe -Lesion pJaDlarsIhMr wh~ doesinfection ttavel? Dorsally -Lesion sub 1,2... wheredoesinfection ttavel? Medial arch
More cortices the better More screws the better V~ Principle will attack the domjnaJ1t fracture
DVT, compartsyndrome, Charcot,Venousstasis. lymphangitis,lymphedema, infection,fracture -What soakswould you Rx forpseudomonasinfection? Acetic acid (white vinegar)4: 1 H2O: AA -Youngestageits safeto do osseous procedure MA? for 6 yy
-~
-DifIcrence
Coverage areasof poor ~~~ for Reconstruction thicknesswound full Paddingoverbony PfOm-~~
Medial arch, Sjuus T aIsi 24-48 bh after jDjmy or it won )t be aca1mte
-Wherein foot t3n you harvest small FTSGs? -What time fiame 00 yon. need to do peImeal tenogram in?
-How
long after tendon tIansfer can
pt
retmn
to~activityandwhy?
-MC. causes TrS?
-Du Vries procedure for caws foot?
4 wks; blc ~eIingpbase (3I1ipbase) 15-28 lasts days bowever. manypeoplesaycan ~ at 3 wks
Pronation, Varia)Siti~
Dorsit1exoryfusionofMrJ
l)fibula to le&1gth plate and 2)tibiato]eDgth 3}pack defect 4)buttress plate m~~1y
for Amount of comm;mttion #1 TIauma;1ongIItMr, short lotMr, bypeIDKJbiie lotMr, immOOile Mr, MPE, DJD, lot ~ iifuLupad!if.c;, iatI'Ogenic
Johnson and Strom, Conti, F1DJk For PTlD; a smgical ~1&.-o:sification l)avuJsion PIT (IUpbJre) 2)midsubstaDcetear near med mall 3)longitsplitB w/o complete ropb1re 4)teDosynovitis w/o visible roptme
~~~.Qf~~:-
-What
Cbapman
Mueller Degan For MaligoantMelanoma;OaIks=pathOlogic staging, Breslows=survival ratesbasedon depth Sbot,gonwoun& Morris Knwada Vogler (=SAD) For LCL iqjmy, I=CFL, ll=I+ATFL, m-n+PTFL, IV=m-+partia1 deltoid tear In propoJsion, flexed 45 degrees, p1aDtaIflexcd KJ AI 20 degrees D=1 to at Jeast 65 ~ -7 get dorsiflexionat lit MP] to aca>UDt this! for
-Qassification systemfor medmall fraCblreS? -CJ:I~~fication systemfor ant proce3S cal~~? -What are Carks andBresiowscl:ls.~ficatiODS1 -What is Oniogscl~cificatjon for? systemfor AchT lesions'!
-CI--3S-sification
-Classification system for AchT mpture? -C1a~afication system for STJ artbroeresis7
-Brajded absoIbable synthetic sutures? -BIaided non-ebsorbable synthetic SUbJres? - Whm does Sclerosing OM of GaDe ocaJr? -Dil~Jdid is a good a1tema1ive morphine for to paUelKswi1h
Hi 1e\Ie1s systemic steroids IDfC(2ion(pDmJeoceiqia~ wooDd edges) U1KX)DttOUai DM EtDbiSDl(DDttilional depletion) Prolonged depeodmcy (edema) Dessi~on of tissues ~ AscoIbic acid deficiency (collagen syn requires) Pl8SdetiDhibitiDg drop (ASA, NSAIDS) Hepatic di~~ (~~ ~ factors, albumin) ADemia (Hb <10 am Hct <33=iDadeq ~ 02 supply)
-LFFD: is NWB castinmcatOO 0RJF7 No; casting1msaabIc w/o fixation is rmely over jts effedive -LFFD: wbatooyookd:foronxray1 AP: m,.~~c>2Dm1b'w 1._~MI'Bs MO: broken)iDeof 4d1 MI'B mn MC (~ line up exactly) -"Toe up" signofhaDox z? Sccnw/LFFD whenTAT iiItcr-~ (~~ haDax)
.g.1or
ralUdiooaff.FFD?
l)rOOuce djastasj~
2~ 3~
-:#1 ~lication
-M C. cause
Post-tnn~t'.
~ impact
will
have some
-Cause of avulsion ~
d(K'S81 c:aICBDeUs?
amd?
Navia1lar
('~
Pn'
additioo10
Watson-JOIa? TypeIV: suess ~~ fIadme -MC. DavicuJarfractme? W-JType (dorsal n Jip/chip fx) -Whal doyou0 RIF W-J Typen? Whm >20010 artiaJJar imIOlved surf. -Aviators Astmlgus =? Another name talar~ fx for -HowmanyaaaredisnJpted Hawkins for Ill7 2 a (I=1 a, n aIM! m=2 as,IV=3aa) -WbaI: themajorartm~ supplying are talus? PTA, ATA, ~ng peroneal a -Bemdt-HaJdy howdoyoudiagn(S? Need MRI to ~ thissaage Ian
-What is tr~f B-H m~? 8-12 wks castimmOO. thm amscr)' ("'DcSI1 requiIc um man oSIeotOmy ~ wI ofOCD. then castp~cxM)
-Another
Osteochondritis D1-,~~
-What. is most common taJar dome lesion? PIMP -What. type of talar fx d/t dorsiflexion injuries? Head, Deck fx -MOl for taJarbody ~? Cc,iDpfessionof tibia against talus
-MOl for ]at ~ fractmes? -ShepmUs fIadme =? -Name for talar}X)st processIx?
-What other ta1arcm~Qjfication sys includes 131ar dome lesions besides B-H1 -What % of ta1arfx are neck fx:? -What xray view is good for 1a1ar n=7
Dorstt1exion.;jnversion
Medja1 tubercle
Latemlta1arprocessfl'acbJre
= Hmiey's notchfx
From A to P wI 23.5 cortical screws (must sink the saews when placed dmJugh the head) To prevem vams/valgus rotation of head Mani- Weber (I'ype I) 12-16 wks NWB; 0Dce A VN di~gnosed,
~is~~~e?
-Most disabling pedal ftactures? -What else must be considered wi cak: .fx?
-~
NO
Rowe IC (3Dterior~ fx)
~ ~
-Palmers theory = ?
For iDIra-al'tic ca]caneal fx l)primary fx line d/t talusdriving iDI:o calc 2)secondazy line givesjt depress TI' fx vs 3)Jatwall blowout 4)cJastic recoil ->sttp-off (JatfI3l frdgmt sinksaway)
By directionof F; vertical F = TT. aDtfrior F =jt depression - Tx for SaDdezs type IV? Primaryarthrodesis level comminl~on d/t -The closer a ~ gets to sust 131i.it is_7 Harderto fixate -High Ankle SpmiDs=1 Synnesmotic spI3ins -Mor high anklesprain?
Forced ex! rotation of fOOtwI iDtemalleg rotation ORhyperd orsifiexion wI forceful ex!:rotation
-Diagnostictestfor sy!!d~ntic
sprain?
10
Deltoid 1,pment usually I~~ also Manual compiSOn of tibia BOOfibula alxJve calf level; is paiDful = syIMiestOOtlc sprain
wn
PERanklefIadme No; they are reIOOVM at 3-4 mODtbs usn (*aews left in permaJaJfiy~ fail d/t normalmotion bIw tibia andfibular)
-1
mmlattzal~~~
w/in aokle~
(~ Up to 42% LIt man fx wI AJ ~~~ kas bd1iIKI1ibia; ~ d/t ex! rotationof foot) VollcmaDDS fx
dtcreases tibio-talar contactby? -Bwwuiibs ~7 -Posterior ma1l~1a!' anklefx -7 -MC. Lauge-Hausen7 -WoIstLauge..JiaDseIl 7 -SAD Mn~7
-Which L-H bas tI8DSVfiOO1arfx on
ss.n PER IV
Fibular awlsion wI vertical mM mall fx
lat aIKioo1iq1x: fibJ)ar fx 011. AP? -Til1auxm:Wagstaftefxs can be s=D.wI wbichL-Hs? -V ~~.c fx ~ be ~ wI which Lange-F~
-What is tbe AOIASIF L'Jassificatial for
PAB
Pm n-1V c:D1y
smtm.IV;~1V
_.~Q!!-:DoIsifiexion (.-rlofL--H) SAD (b/c fx mcd man dIt \X)I11p: ~ from talus!) -Pilon fx ~ to Ic.-lh in wbat type def'UA-iiiiLy7 Varus;d/t cxIm gabjJi1y fibula of -If pilon fx is too comminnt~ to ORlFr7Tl Traction 5-8 wks then AJ fusion -T.i~oos or ~J jDjmies mrely ~ befme what age? 8 y/o -M.C. reason pbyscalgrowth diaIii'-~? for Bony bri~~~ -Salter -Hmis I may be assocwI what ~- ~~ Scurvy,~ myeioproJif.d/~
-Which L-H mech ~ 't nJIMme ~1tnid?
viJonfx?
---
-MC. Salter-Hmris'l
(smrpys fibers abseIIt before2 y/o) -Thurstml-HollaIKisign caused by? Lateml ~~1acaDt F .oD1y~ when a Dew (an ~ ~ pbysis? TiD8JX~ (bIc of 8F) -How 00 you diagnose triplane ankle~'l cr (2O,'too only xmys) wI
-Ogden mOOificatioo to Saher-Hmris? Avulsion fx epiphysis wi 0 physca1involvemt
-Keyto 5'~~~
~f!nt of physea1 iDjuries? R~~~liDg to G-A Types n, m? B/c of Gram Dega1ive bac aDd erKiotoxins (eDdotoxiDsC2I1 ~sboc1:) -WIa. are woums fromfasci otomy (COmpaItlDCDt decompression) closed? SdaysP/OP Pain -Most im.-: sign of ~ syndrome? -Why are AGs ~
-2l1li
Pressme
4-U bh (somesay 3h) 6-12 bh
-At what ~ doesirreYCIsible Im/mm. damage occur? -Partial/totalfunction deficik first seenat ?
-UDtl'eate(i COiiij:iil1~ syndrome has what
11
-Treves disease?
"fibular
sesamoid
-~~ttim disease? "tibial ~--!!!~id -GSW - whatdo yon do once stabilize ~? CheckNY andteaaDUs status -L VM GSW=whatvelocity? <2000ft/sec -When is s.t uaumaconsideJ'ed conm~t~? >6 hh post tIamna
- T etaDUS prone wounds = ?
>6hhpostiDjury bum,frostbite, auch or missile injmy stellate wound,abIasion,or avulsionof skiD devitatiz~ ischemictissue contamin~nts (dirt, feces,saliva) -caPS - whenis remis.~on most likely?
-What causes Sudeks Atrophy?
Phase ill ("ciamc" iDi1ial phase) Hyperemia, increased meduDaty P, decreasedpH (*NOT d/t disuse)
-CRPS phases?
IA: ooJd/VC (2-6 wks) (akaacute) IB: Sweaty/VD.(2-6 mos) (akaaaJte) ll: S~ Atrophy (ab dystrophic) ill: deadphase(6-12mos) (akaatrophic)
do to
CRPS IX?
-What.
should
you never
.
NEVER.immobilizethe area
Type I - partjal tear less than 500/0
-Knwada
classification
of AchT ~?
---
Typen - completetear <3cm ~ Typem - completetear 3-6 cm defect T:Ype - completetear> 6cm defect IV -2 reasons Ach1T~ usn at Watershed area?
dea-easedva scuIarity, ~
torque (AchT twists prox to distal}
~kioM
105 degrees -Which ligament aosses bothAJ andSTJ? CFL -Valgus tilting of talus sigofies? Complete mptID'e~cial. & deep deltoid -complete mJXme usn ~ in how much
lataa1t31ardispla~
-Should
-Nail ~
-BY~ngbase
12
-~
-Bum
3.5%
SermnaIKiUriDc ~l8!i1y (kidneyfm), eledrolyres,Hct (volmDem~) -Sb(H!]d give bum pt JXDPhylaClic roo Abx? No blc may ~~~on off A or ~ -4dodcsreefrosd)jtcresu1rs in? Lossof bodypart -O1i1blain/pemio ? c ChImic~~Jlitis of dermis ~ by cold -4 ~ for 1/3nJbu1ar pJate? DeUtl31i~on (fiOOlar ~ fx), plate.Do>. talsioo baodingplate -Spnmg Mortises ? SyDliesmroc ~ -Whm do you changea dmin? q12hCX' Y2 if full -PeroDeal sOOIuxatioo...a ortXJlx:WOJk I'" Try Sot. tbeJ1 to groove(k;q)eDmgproceds, 1., go c
Deoplan, djd,
.-.
~P1~~
-Kilby
;JDDIImogeoic, con~~;w. ~-.mm. tIa1DDa, aMkgiIIe, eJc~ll'bi~) MN feels bdrer wi !,11mt1rrf1exion, PDS feels worse Seenwi excessproDatioo; 1attJ:alprocess of talus hiWDg ~ l~S (d)Stmcts siIIIs 1arSi) ~ -. . ~
-What NSAID
-NSAIDS ~
-6 ~
for AI b1odc'l
Tibial,~s.
deep ~
IDCN.IDOl, sural
eIytbo, - -
oK-wire ~
-V8DOOJaksam~7 ...A~-tbat-D=1"DO'~-adjustmatt?
- ABX for pegDaDt women? -ABX for mtRin! moms? -can JUegDaDt women get an MRrl
-Rule of lh- am 1/3 for 1xJDe?
~30-40,trou8h5-l0 . -A:mc"8iirEfVr(87itTiiO,~-dOij;~~~.
DmXi)
-PICCliDe=?
in kjds?
PECs(PCNs,coythro,1- and r gfD cefs (not I:dmuximc PA VB (PCNs,AGs, VaDCO, erythro) No is a CJl (una ~~ emergmcy) <30 yy & 1/3rule (1/3 = ~111aIy boDe), >40 yy = 1/2 rule (Jose cortiQ1boDe-> moremMn naryboDe) No; SlayS growth platesfor days in
-Direct R~tioo Vi ~
for fx fI3gmts?
-M.C. a)aJitionin~? -~ for panMIH ~1 Tr-tnt#S1ffor ~~ian cyst? -Whm ck> ordtr: cr scans fxs? yoo wI
13
Inhibits degredx toxic peroxides fungal cell andinhibi-ls of in Fe synthesis -MOA teltri-mfJne? Inhibits ergosterol syDdJesis -How do you treat onychomycosis pregnantpt? M~hanical debridemt in -# 1 reasonfor DM pts to be bospi~1ized1 Foot infections -Why are DM pts skin so dry? DIt giycosylationof tissues (this occmsevenin skin)
-Di:fIerence
-Definitive
-MO A ciclopirox?
RSD
-What is a podiatric~ for Tagamet? As treatmmt for waItS;worksbestin kids < 16y/o -How long doesit aake arthrodesisto ftJse? 10 wks or tmtil seebonytrabeaIlatiw J3di0~lly an -STJ impJant...What stIUdsdo you want to avoid? Suraln, )at cal~J!~ n, lateralaDd~ us TC Jigs, a1t=Yof tarsalcaImI, artay of simIStaIsi, deltoid artery, possiblyIDCN (~ ou JX:) -Decrease Hbalc by 1 point -> ? 43% dea'ease amputation, risk 24% decrease IezJa1 risk: failure, 14%deaease cataracts risk and:t...n, % ~ 12 risk stIoke -Which is bdter for DM pts - ABI or PPG7.PPGb/c ~ systolictoeP am medial ~~-5 doesn't usuextemtotocs -TAL ~ plantarpessuresin DM pts by? 2"/0 decrease ~ in -What is d1e mosteffectiveway to ~med oo1umn? TN] fusion -What is d1emoststablefixation for TN] fusion? Circum~ fixation (3 screws...2 crossed screws - --. -.wI 3Msaew Jatcra1 to.~) --What
--
are sequelae
of TNJ fusion
if do not address
NC fault/sagging (*othc; sequelae ~ TN} fusion = CC1subJDxation) -PTID p1ns fixed valgusheel- how to correct? Triple arthrodesis needed restorealignmt,funcboo to -How doesthe Evansfix flatfoot? By tealigning CCJandTN] sothey ~ not pamlle1, hypermooilityis ~~ and stability is iDa'eased -What arethejt destlUcti\Ie pI~? In.. Artm'OOesis, Imp1aI!!, KeUer,O1ei1ectomy -What is biggerthan 100suture? 0,0-0,0-1,0-2,0-3 -Diffelmce b/w chronicand aaIte woUDds?Chronichavepersisting~ phase--> exudate hi [ Js wI of:MMPs (mab:ixmetall oprotei~-~) *~ MMPs resuhin degredaUon CXtI3ceU matrix proteins aDdinactivati of growth factors(--> delayhealing) on
-What is promogran?
equiDDS?
A wound matrix; used for venous stasis u1ceIS *binds }\.,IMpsaDdpromotes moist wound environment Classic/MediteII3Dean FnrlP:lnit'J Afiican
Epidemic/AIDS-related Transplant-related/Immlmodeficiency
-Myositis ~~~--:!!S circumscripla ? Heterotopic05$i~~Jionthat occmsafter tramna -What bone tumor is in the ddx for myositis ossificans? Osteosarc (same age group)
-Difference
-LateI31
b/w secondarY
ixlines
1T horiz, Joint~
JoiDt~on
on vertical
intta-articuJar fx
14
-DKA cbarada~
by what 3lab~?
-potentially
-Why
lethal ofDKA? SE
~
ck:tes mt ~ DKA
as fIeq~
as non-ketotic does?
-Treatm~
Non~hYJa
osmolar ooma
SIeqJ 8Dd~
BMD test
sing~
-~graft
vs.Dynagrafl
Denua~ BUN~
artbtitis?
Debridement. Drainage,D~sion
Gmm stain aDd (+) ~ fluid aJ1nD'e
-F21}m,ntr
Closed ~
SA N. pOJlbca SA; Gram~
~on
qd to dminjoiDt
eoaaic ba~~
-MC. causesepticarthritis in kids? -MC. ~ septicarthriaisin Y.A ? -MC. causesepticSInIn;ti~indde.dy7 -Top 4 ~ of ~Jllary iDfBrct?
-What is
a '-natoma b~
For reduction of fx; inject. same amt of local you would for regional bloclL (ie aDkle bkd: for taJar fx/~is!~~) ~ ~ it iDo ~ ofhE,..1Sit.~...d ..the ~ of ~~J bJeed!ngis aIQ m worst fmctme. . ..by doing a more location ~c block, the ptexperi=1 ~ 1rss.-m both during and post reduction
u~ age 40, DOmIalboDe is 112:112 oortjcal:canceIloos; after age 40, cortical bone ID8$ ~ aIKI result is 1/3:2/3 a)I1jr21:~Jlous_- .this is a maIka' for~ osis Eating diDdeI. ~~0Ibea. oste.opeDja
-caws foot IDOIC Iikdy to ~ to what type ~ fx? -Flatfoot more likely to leadto what ~ ~ fx:?
-High
Tibial ~ MT~fx
fx
jDci~
RA ~iaDC
of 2mMT ~
caD. ~
-What.
nOOu]e fuiiDatioD?
MTX
-What other~~~
-What ~~~ canIMuce RA nodules? -What is focusof surgical ~OD of ankle fx?
-What
causesepi~1
1nl'Jnc1nn cysts?
Q)l~hicine Restore length of fibu]a. syndesmooc stability, a~~1y realignankle mortise UAJallyd/t h3,..lmlic impJaotation ofepj demJal cells intodennaltissue STJdislocationcauses !oct.to look like this the High ~ injury wI forceful inversionof plaDtarfiexed foot
1~
-Most
of taJar htad to osvjco1ar, sbooId be amgromt on all views (normal) *wI STJdislocation, will ~ intact CCJbut not TNJ US (mole 8CCIlra1e than MRI MRI ~'t showall
-Why ~'t
cl1i1dren / RicketsstalxilwaJk? w
~) WaIkmg/~g om drJayed stI'IIdmal suppcxt hlc isn't alffid=t ...chiJdIal who do tty to walk usn deve1op ~ fx aro1DMi~ epipbysjmK:tion
ELISA; det=s IgG and IgMAbs of the spirochete WhitetoBJuetoRed rnEST (w/o ~~, its just RayDaIK1s) Assymebic oligoartbritis ofLE wiD rest aftriad ~ pain is usn very pc.~.mi..?2it this form wi Heclpain
-What
-Incomplete ReitfI'Sp~
~TalaIgia =7 -Pre-ballux c?
-If
as?
A~
DaviaJ]ar
ABX before yoo ck> aJ1bJres7 wI Dai1bed ~tion... what is #1 thiDgywcbeci:? -t 1/2 ofhqJarin1 ~fx:
mfu.48haffABX
NVs8abIS,tha1cbedctdaDDS -_. O.5-2h(afta" 1~ ~ Jifc. Je\Ie1s 9Jb.;fbtraj1elWcb/c ale ~ is gone)
~"11in
amta7Ji)3a3m?
-}..ngm~tin ooJJ1ajns amt amoxi and clav acid? 650 mgamoxicillin, 125mg cIavuJaDic what acid
Tim~
-~
oo~
2 g ~11iD,
~~i~
bugsby ~~.!
~afsopcrficialposteriorOOmpaI1legmm in calf regiOD/high ankle'! ~ Tom, Harry (M->L) .Does Kites aDgIe iDaase (X" deaeasewI pmatim? ~ (21 ~~ is ~1) .Wha1is the talo-navicu1ar angle? Columntali to Ime tlaDsecting 1arsus < (JM)IDJa) =60-80 ~)
-6 ~
HaIJux Yams?
~&~.
~id, iX:Iil~
-Sequential~~~
ofHT?
.order ofDeuIOmas fromM->L? -What is the CD4 Mfinihon of AIDS? -pathon. sign of osteoid~ma? -Why do gram (-)5 ~ septicemia eJdaiy'! in
-Milk of ~rnnl81Cia ?
Ext hO<xi lCRdion, at ~ 1~!;fMrin& ckJrsal ~~~, plaorarplate~~~. ardIrodesis PIPI (all ~ at MPJ) Ioplins. T~Jans, Bowsers,Mmtons, HDdcrS,~ a:>4<2OO=AIDS n~lu-~ ~ ~ arethe #1 ~ tms
Propot'ol
-What ~~r..lliates
-Why
~~
PKa
waItS aIaI't
boDeprOOlcms
and both have ahyperbi dIOtic component B/c bas 19amts t3DDicacid (dIyiDg agaIt) Weber aDd Ced1
16