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PRESENTATION BY:RAJESH JAIN MDS 1ST YEAR DEPARTMENT OF CONSERVATIVE AND ENDODONTICS ITS DENTAL COLLEGE HOSPITAL AND RESEARCH CENTER GREATER NOIDA
Contents
Introduction Review of literature History Definitions Theories of dental caries Etiology Classification Histopathology
Diagnosis
Caries protection Conclusion
References
Introduction
Dental caries is the most common chronic disease (5 billion people worldwide) It is costly in terms of time and work hours lost, money spent. In addition the expense incurred in education of health professional required to cope with this disease in terms of prevention, treatment and oral rehabilitation.
Review of literature
Psoter WJ, Reid BC, Katz RV. 2005 stated that Enamel hypoplasia, salivary glandular hypofunction and saliva compositional changes may be mechanisms through which malnutrition is associated with caries, while altered eruption timing may create a challenge in the analysis of age-specific caries rates. Hillman JD, Dzuback AL, Andrews SW(1976) concluded that streptococcus mutans was the main organism responsible for dental caries. Dreizen S, Brown LR(1987)stated that there is a strong corelation between Xerostomia and dental caries. Burke F.J.T (1998) states that presented trend in treating caries directed more towards prevention & minimal intervention rather than the traditional drill and fill dentistry.
HISTORY
Aristotle, Hippocrates and Shakespeare have all written on dental caries in their writings.
Some theories put forward are the Worm theory, Vital theory etc.
L. S. Parmly (1819)-first contributed to current understanding of caries mechanism Emil Magitot experimented using Pasteur findings. He produced artificial carious lesions in extracted teeth. W.D.Miller (1890) Chemo parasitic theory. Gottlieb (1941) Proteolysis theory. Schatz & Martin(1955) Proteolysis chelation theory.
Definitions
Dental caries is a microbial disease of the calcified tissues, characterized by demineralization of the inorganic portion and destruction of organic portion of the tooth. (Shafer) Dental caries is an infectious microbiologic disease of the teeth that results in localized dissolution and destruction of the calcified tissues. (Sturdevant) Dental caries is defined as a progressive, irreversible multifactorial in nature affecting the calcified tissues of teeth, characterized by demineralization of the inorganic portion and destruction of organic portion of the tooth. (Soben peter)
Theories of dental caries 1. Worms Theory 2. Humor Theory 3. Vital Theory 4. Chemical theory 5. Parasitic at septic theory 6. Chemical Parasitic theory 7. Proteolytic theory 8. Proteolysis Chelation theory 9. Acidogenic theory 10.Levines theory 11. Bandlish theory
a] Millers Chemo-parasitic / Acidogenic theory b] The proteolytic theory c] The sucrose-chelation theory
In a series of experiments following facts were demonstrated Acid was present in deep carious lesions Several types of bacteria could produce acid Lactic acid was an identifiable product Different kinds of food could decalcify the entire crown Different kinds of microorganisms had potential to invade carious dentin
Draw backs
Phenomenon of arrested caries,caries on unerupted teeth is not explained Smooth surface caries was not accounted Particular type of organisms causing caries was not explained
The organic component is most vulnerable and is attacked by hydrolytic enzymes of microorganisms ,this precedes the loss of inorganic phase
Critics
organic matrix (small %) sufficient ??
Draw backs
Organic matrix (small %) dissolution can produce sufficient amount of chelates .
Break down of organic matter by proteolysis in initiating caries lacks experimental support
I. PRIMARY FACTORS:
1.TOOTH a. Susceptible tooth surface b. biochemical characteristic of tooth 2.DENTAL PLAQUE 3.DIET 4.TIME
II.MODIFYING FACTORS:
1. SALIVA 2. SYSTEMIC HEALTH 3. SEX 4. RACE 5. GEOGRAPHIC ENVIRONMENT 6. OCCUPATION
CLASSIFICATION
I. STURDEVANT
Based on - Location
- Extent - Rate of progression
According to location:
a. Primary caries b. Caries of pit and fissure origin c. Caries of enamel smooth surface origin d. Backward caries e. Forward caries f. Residual caries g. Root surface caries
According to extent:
a. Incipient (reversible) caries
b. Cavitated (irreversible) caries
Class IV- Restorations on anterior teeth that involve the incisal angles. Class V- Restorations on all gingival third of facial or lingual surfaces of all teeth (except pit and fissure lesions) Class VI- restorations on incisal edge of anterior teeth or the occlusal cusp heights of posterior teeth.
proposed by Simon 1. Simple caries: one surface is involved Compound caries: two surfaces are involved Complex caries: three or more surfaces are involved
2.
3.
WHO classification
The shape and the depth of the carious lesion can be scored on a 4 point scale D1 -Clinically detectable enamel lesions with intact (non cavitated) surfaces D2 -Clinically detectable cavities limited to enamel
Limited to the occlusal surfaces of molars and premolars - buccal pits of molars - lingual surfaces of maxillary anterior teeth Poor self-cleansing features Usually occurs before smooth surface caries Clinically - black or brown in color - slightly soft consistency - catch the tip of a fine explorer Adjacent enamel appears bluish white Internal Caries
Cervical Caries
Appears as crescent shaped lesion.
Backward Caries
Lateral spread of the lesion along the DEJ exceeds the caries in the contiguous enamel, caries extends into this enamel from the junction.
Forward Caries
Caries cone in enamel is larger or at least the same size as that in dentin
Residual Caries
Caries that remains in a completed cavity preparation Not acceptable if - present at DEJ - prepared enamel wall
Initiates at the surface of a mineralized dentin and Cementum which have greater organic content
Usually have rapid clinical course
Indicates unusual susceptibility to caries attack, poor cavity preparation, defective restoration. Also indicates presence of microleakage.
Odontoclasia:
Rampant caries:
Sudden and rapid onset and almost uncontrollable destruction of teeth Involves teeth that are ordinarily caries free (mandibular incisors) Ten or more new increments of carious lesion in one year
Adolescent caries:
Acute caries attack at 11-18 years of age
Arrested caries:
Caries which becomes static or stationary and does not show any tendency for progression Almost exclusively occurs on occlusal surfaces Both dentitions are affected Lesion appears as large open cavity with lack of food retention Superficially softened and decalcified dentin gets burnished and has brown stained polished appearance Eburnation of dentin
Senile Caries
Caries activity that spurts up during the old age.
prominent enamel-rods
Appearance of transverse striations of enamel rods due to segmental demineralization Accentuation of incremental striae of
Retzius
These zones are from the dentin towards the outer enamel surface
Occurs due to formation of submicroscopic pores at enamel rod boundaries and striae of Retzius. This zone is slightly more porous than sound enamel having a pore volume of 1% compared to 0.1% of sound enamel.
DARK ZONE: Lies superficial to translucent zone. Called positive zone as it is always present. Pore volume is 2 4%. Increased porosity in this zone is due to greater degree of demineralization in this zone.
BODY OF LESION: Forms bulk of the lesion and lies between relatively unaffected surface zone and dark zone. Area of greatest demineralization, having a pore volume of 5% near the periphery to about 25% in the center of body of lesion.
SURFACE ZONE: Interestingly, this zone not only remains intact during the early stages of attack by caries, but also REMAINS MORE HEAVILY MINERALIZED. Pore volume of only 1%. Ions for remineralization come either from those within plaque or from reprecipitation of calcium and phosphate ions diffusing outwards as deeper layers are demineralized. Eventually, this zone is demineralized by the time caries penetrates dentin.
Dentinal Caries
Once lesion spreads to DEJ, there is lateral spread of caries Surface enamel gets unsupported enamel rods enamel # greater cavitation Zones of dentinal caries. Zones start from pulpal side towards dentinal side
1.
2. 3.
4. 5.
Zone of Decalcification with Bacterial Invasion Zone of Decomposed Dentin / Infected dentin
Observing from the pulpal side at the advancing edge of carious lesion following different zones can be seen ZONE 1 Zone of fatty degeneration of Tomes fibers
5 4 3 2 1
Careful examination under clean and dry condition with good illumination can reveal various signs of caries like:- brown discoloration of pits and fissures - opacity beneath pits and fissures or marginal ridges - frank cavitation of the tooth surface
TACTILE EXAMINATION:
Use
of dental explorer may help in detection of dental caries. Tactile findings suggestive of caries are: - softness at the base of a pit and fissures and discontinuity of enamel surface - catch at the explorer tip - cavitation at base of pit and fissure Cautions:excessive pressure with explorer can cause cavitation where was not present earlier infective m.org may be transferred to uninfected area
RADIOGRAPHIC EXAMINATION:
-Conventional , intraoral periapical and bitewing radiograph are employed to diagnose dental caries - bitewing is of more diagnostic value Uses of bitewing: detecting proximal caries Examining many teeth in one radiograph Checking cervical margin of restoration Monitoring the progress of arrest caries
TOOTH SEPARATION:
To detect initial proximal caries, separation of the contacting teeth can be achieved using wedges or mechanical separator Once the proximal surface is accessible, visual examination and gentle probing may help in diagnosis of the carious lesion
FIBEROPTIC TRANSILLUMINATION:
Carious lesion have lowered index of light transmission, when teeth are examined with the fiberoptic light source, caries appears as a dark shadow After drying the tooth, a fiberoptic probe can be placed in the buccal or lingual embrassures directly beneath the contact area between two adjacent teeth. If caries is present , dark shadow is seen beneath the marginal ridge Non invasive No radiation hazard No permanent record Difficulty in placing probe
XERORADIOGRAPHY:
Image is recorded on an aluminium plate coated with a layer of selenium particles These selenium particles are charged uniformly and stored in a unit called condition When x-ray is passed onto the film , it causes selective discharge of the particles which forms a latent image. This is converted into positive image by a process known as development in the process per unit Less radiation exposure No wet processing Electric charge over the film may cause discomfort
CHEMICAL MEASURES
Substances which alter tooth surface/structure
Fluorine Bis-biguanides Silver nitrate Zinc chloride & potassium ferrocyanide
NUTRITIONAL MEASURES
Diet counseling restriction of refined carbohydrates Phosphated diets Calcium phosphate rich diet. Sugar substitutes Non-caloric sweeteners-aspartame, saccharine Caloric sweeteners-sorbitol, Xylitol, Mannitol
MECHANICAL MEASURES
Dental prophylaxis
Tooth brushing
Mouth rinsing Dental floss
Oral irrigators
Chewing gum Pit & fissure sealants Preventive resin restorations
conclusion
Dental caries is an oral infection. Dental caries has a multi-factorial causation involving the interaction of host factors (tooth surface, saliva, acquired pellicle), diet, and dental plaque (biofilm). Besides these other modifying factors like socioeconomic status and behavioral patterns also greatly influence the caries process in a complex manner. A good understanding of the caries process can help in formulation of better diagnosis, prevention and treatment of dental caries .
References
1) 2) 3)
Sturdevant's Art and Science of Operative Dentistry5th edition pg74-110 Cariology Ernest Newbrun- 3rd edition Diagnosis & Risk prediction of dental caries-Per Axelsson.
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