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Periodontal Disease
Usefulness of Radiographs
Amount of bone present Condition of alveolar crest Bone loss in furcation areas Width of periodontal ligament Local factors: calculus, overhanging restorations Crown/root ratio
Limitations of Radiographs
No indication of morphology of bony defects No indication of successful management No indication of hard/soft tissue relationship, i.e., depth of pockets
Local Factors
Calculus Overhanging restorations Poor restoration contours
Calculus
Overhanging Restoration
Juvenile Periodontitis
Occurs in healthy individuals between puberty and age 25 Amount of bone loss is not consistent with local factors and oral Hygiene habits. Rate of bone loss is 3-4 times faster than in typical periodontitis
Juvenile Periodontitis(cont.)
Typically affects crestal bone of first molars and incisors. Eventually affects greater # of teeth. Bone loss is progressive and frequently bilaterally symmetrical. Many teeth show vertical bone loss. Host neutrophil dysfunction has been demonstrated by several investigators.
Papillon-Lefevre Syndrome
Autosomal recessive trait Hyperkeratosis of palms and soles Occasional keratosis of other skin surfaces Calcification in falx cerebri Severe destruction of alveolar bone involving all deciduous and perm. teeth Exfoliation of teeth
Hand-Schuller-Christian Disease
Most cases reported in children under 10 years. Has been reported in older individuals Skeletal and soft tissues may be involved Classic triad of symptoms:
punched out destructive bone lesions unilateral or bilateral exophthalmos diabetes insipidus
Hand-Schuller-Christian (Cont.)
Oral manifestations include:
loose teeth exfoliated teeth gingivitis loss of alveolar bone / advanced periodontitis
Sharply outlined multiple radiolucent lesions in skull, jaws and other bones
Letterer-Siwe Disease
Acute, disseminated form of disease Usually occurs before age 3. Most patients die Involves several bones and organs Skin rash Intermittent fever, enlargement of liver and spleen, lymphadenopathy common Destructive radiolucencies in jaws Loosening and premature loss of teeth
Hand-Schuller-Christian Disease
Hand-Schuller-Christian Disease
Leukemia
Leukemia
Periapical granuloma: Localized mass of chronic granulation tissue containing PMNs, lymphocytes, plasma cells.
Periapical Granuloma
Radiographically, widening of PDL or variable size of periapical radiolucency may be present
Periapical Granuloma
Periapical Granuloma
Periapical Abscess
Periapical abscess: When pus forms in the area. It may develop directly as an acute process or develop in a preexisting granuloma. Radiographically, appears identical to granuloma.
Sclerosing Osteitis
(Idiopathic) Osteosclerosis
Osteosclerosis
How do you differentiate between osteosclerosis and condensing osteitis? In osteosclerosis, the pulp is vital. There are no clinical signs or symptoms. No treatment is necessary. Sclerosing osteitis is secondary to pulp exposure. Patient is symptomatic. Endodontic treatment or extraction is indicated.
Calcific Degeneration
Calcific Degeneration
Widening of apical PDL or periapical radiolucency ( associated with indication of pulp exposure) Discontinuity of lamina dura Displacement of lamina dura Sclerosing osteitis Calcific degeneration (metamorphosis) Radiographic indication of pulp exposure
Stage II
Stage III
Multiple