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I. History:
Prior injuries, treatment and outcomes Medications Family history Social history:
Vocational Education Tobacco / ETOH / Illicit drugs Function: ADLs & Mobility
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Litigation
Pain Specifics:
Onset:
Gradual: DDD Acute: Disc abnormality, strain, compression fractures
Severity / Intensity Frequency: Constant vs. Intermittent Duration Exacerbating and Alleviating Factors Time of Day: If nocturnal, consider malignancy
Red Flags:
Significant trauma history, or minor in older adults Nocturnal pain in supine position with history of cancer Bladder or bowel incontinence or dysfunction Constitutional symptoms:
Fever / chills Weight loss Lymph node enlargement
II. Examination:
A. Physical:
Posture:
Splinting Body language
Gait:
Antalgia Heel / Toe pattern Trendelenberg
Musculoskeletal:
ROM Leg length Vascular Atrophy
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Abdomen:
Presence of masses
Back:
Inspection Palpation ROM Scoliosis
Neurological:
Sensation Motor DTRs
Rectal if indicated:
Evaluation of sphincter tone
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Give-away weakness: Inconsistent effort on manual motor testing with ratcheting rather than smooth resistance
C. Pathological Examination:
Spurlings maneuver: Lateral rotation and extension of spine resulting
in neuroforaminal narrowing and nerve root encroachment, clinically reproducing extremity pain, usually in dermatomal distribution
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A. Epidemiology:
Incidence of LBP: 60-90 % lifetime incidence 5 % annual incidence 90 % of cases of LBP resolve without treatment within 6-12 weeks 40-50 % LBP cases resolve without treatment in 1 week 75 % of cases with nerve root involvement can resolve in 6 months LBP and lumbar surgery are: 2nd and 3rd highest reasons for physician visits 5th leading cause for hospitalization 3rd leading cause for surgery 12
B. Disability:
Age and LBP:
Leading cause of disability of adults < 45 years old Third cause of disability in those > 45 years old
Prevalence rate:
Increased 140 % from 1970 to 1981 with only 125 % population growth Nearly 5 million people in the U.S. are on disability for LBP
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E. Differential Diagnoses:
Lumbar strain Disc bulge / protrusion / extrusion producing radiculopathy Degenerative disc disease Spinal stenosis Spondyloarthropathy Spondylosis Spondylolisthesis Sacro-iliac dysfunction
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F. Diagnostic Tools:
1. Laboratory:
No evidence to support value in first 7 weeks unless with red flags Specifics:
WBC ESR or CRP HLA-B27 Tumor markers:
Kidney Breast Lung Thyroid Prostate
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2. Radiographs:
Pre-existing DJD is most common diagnosis Usually 3 views adequate with obliques only if equivocal findings Indications:
History of trauma with continued pain Less than 20 years or greater than 55 years with severe or persistent pain Noted spinal deformity on exam Signs / symptoms suggestive of spondyloarthropathy Suspicion for infection or tumor
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4. Bone scan:
Very sensitive but nonspecific Useful for:
Malignancy screening Detection for early infection Detection for early or occult fracture
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5. Myelogram:
Procedure of injecting contrast material into the spinal canal with imaging via plain radiographs versus CT In past, considered the gold standard for evaluation of the spinal canal and neurological compression With potential complications, as well as advent of MRI and CT, is less utilized:
More common: Headache, nausea / vomiting Less common: Seizure, pain, neurological change, anaphylaxis
6. CT with myelogram:
Can demonstrate much better anatomical detail than myelogram alone Utilized for:
Demonstrating anatomical detail in multi-level disease in preoperative state Determining nerve root compression etiology of disc versus osteophyte Surgical screening tool if equivocal MRI or CT
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7. CT:
Best for bony changes of spinal or foraminal stenosis Also best for bony detail to determine:
Fracture DJD Malignancy
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Usually followed by CT to evaluate internal architecture, but also may utilize MRI As outcome predictor (Coulhoun study 1988 JBJS):
89 % of those with pain response received benefit from surgery 52 % of those with structural change received surgical benefit
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9. MRI:
Best diagnostic tool for: Soft tissue abnormalities:
Infection Bone marrow changes Spinal canal and neural foraminal contents
Emergent screening:
Cauda equina syndrome Spinal cored injury Vascular occlusion Radiculopathy
Benign vs. malignant compression fractures Osteomyelitis evaluation Evaluation with prior spinal surgery
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Has essentially replaced CT and myelograms for initial evaluations Boden study 1990 JBJS: 20 % of asymptomatic population less than 60 years with HNP 36 % of asymptomatic population of 60 years Jensen study 1995 NEJM: 52 % of asymptomatic patients with disc bulge at one or more levels 27 % of asymptomatic patients with disc protrusion 1 % of asymptomatic patients with disc extrusion
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Post-operatively can take 2-6 months for reduction of mass effect on posterior disc and anterior epidural soft tissues which can resemble pre-operative studies Only indications in immediate post-operative period:
Hemorrhage Disc infection
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Includes:
Pain Assessment Report, which combines: McGill Pain Questionnaire Mooney Pain Drawing Test MMPI Middlesex Hospital Questionnaire Cornell Medical Index Eysenck Personality Inventory
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MRI Nomenclature:
Anular fissure:
transverse distribution
(PER NASS)
Disc bulge:
Circumferential, diffuse, symmetric extension of anulus beyond the adjacent vertebral end plates by 3 or more mm, usually due to weakened or lax anular fibers Focal, asymmetric extension of disc segment beyond margin of vertebral end plates into the spinal canal with most of anular fibers intact Focal, asymmetric extension of disc segment and / or nucleus pulposis through the anular containment into the epidural space with migration into the canal
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Specificity / Sensitivity
Diagnosis
Disc Herniation
Test
CT MRI CT Myelo
Sensitivity
0.90 0.90 0.90 0.90 0.90 0.77
Specificity
0.70 0.70 0.70 0.80-0.95 0.75-0.95 0.70
Spinal Stenosis
CT MRI Myelogram
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G. Treatment
Medications
NSAIDS Membrane stabilizers
TCA / Neurontin re-establish sleep pain reduce radicular dysesthesias
Muscle relaxers:
re-establish sleep patterns more useful in myofascial/muscular pain
Narcotics: rarely indicated Steroids: more useful for radiculitis Non-narcotic analgesics: Ultram
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Physical therapy
Modalities electrical stimulation/TENS Postural education / body mechanics Massage / mobilization / myofascial release Stretching / body work Exercise / strengthening Traction Pre-conditioning / work-conditioning
Injections
Epidural blocks Facet blocks Trigger point SNRB SI joint
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Surgery:
Laminectomy Fusion Discectomy Percutaneous Lumbar Discectomy
Success rate variable 50 -85 % Low rate of complications:
Infection Peripheral nerve injury
Benefits:
Outpatient procedure Minimal to no epidural scarring No general anesthesia Spine stability preservation Decreased cost
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Acupuncture Biofeedback
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A. Sacroiliitis:
History:
Trauma is very common Repetitive LS motion--lumbar rotation or axial loading No specific correlation with exacerbating activities Commonly have leg length discrepancy or condition contributing
Biomechanics:
Movement of the SIJ is involuntary, usually from muscle imbalances Can occur at multiple levels: lower extremities, hip, LS spine Motion is complex and not single-axis based
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Differential Diagnosis:
a. Fracture
Traumatic Insufficiency stress fractures: elderly patient with osteoporosis
without history of trauma
b. Infection
Hematogenous spread with predisposing history Usually unilateral symptoms present
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f. Seronegative spondyloarthropathies
RA--usually not until late in course of disease Ankylosing spondylitis Psoriatic arthritis
g. Primary SI tumor
Rare and usually synovial villoadenomas
h. Iatrogenic instability
Via pelvic tumor resection or bone graft site
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Diagnostic Tools:
X-rays: Up to 25 % of asymptomatic adults over 50 years
can have abnormalities
MRI / CT: Only if looking for tumor Bone scan: Good for fractures but less favorable for inflammation
Treatment:
Medications: NSAIDS Physical therapy Correct limb discrepancy Injection: Fluoroscopy-guided vs. local Surgical fusion: Few figures for efficacy
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Treatment:
Urgent decompression is mandatory for prevention of irreparable / irreversible bladder damage 12 hours is the maximum time prior to irreversible changes
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Occupational forces
Progression of disc narrowing leads to degenerative changes of bony structures, especially posterior components, leading to spondylosis
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Treatment:
Medications Physical therapy Lifestyle changes:
Smoking cessation Weight loss Vocational changes
Injections:
Less helpful if pain is limited to central low back only
Surgery:
Laminectomy Fusion
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D. Spinal Stenosis:
Clinical:
Results from narrowing of spinal canal and / or neural foramina (CONGENITAL OR DEGENERATIVE) Most common complaint is leg pain limiting walking Neurogenic / Pseudoclaudication = pain in lower extremities with gait Relief can occur with:
stopping activity sitting, stooping or bending forward
Common are complaints of weakness and numbness of extremities Usually becomes symptomatic in 6th decade
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Diagnosis:
CT and MRI may yield false-positive results, therefore EMG / NCV can be helpful to confirm diagnosis Myelography also can be confirmatory and pre-surgical screening tool
Treatment:
Medications Physical therapy TENS Epidural injections Surgical decompression laminectomy
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E. HNP:
Clinical:
Low back pain wit associated leg symptoms Positions can induce radicular symptoms Posterolateral disc pathology most common:
Area where anular fibers least protected by PLL Greatest shear forces occur with forward or lateral bend
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Treatment:
Conservative treatment: Saul and Saul study 1989 Spine:
> 90 % success rate of symptom resolution with non-operative management
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Spondylolisthesis
Progression of spondylolysis with separation
Grades assigned I-IV for level of translation Most common levels are L5-S1 (70 %) and L4-L5 (25 %)
Treatment:
Medication Physical Therapy Injections Surgery
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Workers compensation: Laws have become counterproductive-financial compensation or open claim may discourage desire for return work and impede recovery
Psychosocial
Medical
Job availability
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Always consider return to work, whether modified duty with restrictions or limiting hours worked
If patients poorly tolerate standard therapy, consider pool therapy intervention which allows elimination of gravity effects
Functional Capacity Evaluations utilized if patients are not progressing through therapy or if have reached a plateau and abilities as well as restrictions need to be assessed
Job site evaluations appropriate if concerns re: ergonomics
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E. Final Thoughts:
It is the patient, not the diagnostic test, that is treated 80 % of patients will recover from acute low back pain within 3 days to 3 weeks, with or without treatment, with up to 90 % resolved in 6-12 weeks
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