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THROUGH THE
RESPIRATORY SYSTEM
.. Caused by Bacteria
Infectious agent
Mycobacterium
tuberculosis- an
organism that multiplies slowly and
are characterized as acid- fast
aerobic organism, which ca be killed
by heat, sunshine, drying and
ultraviolet light.
Sputum of the person with TB is the
most common source of the
organism.
Incubation period
From
2 to 10 weeks
ETIOLOGY:
Poverty/ overcrowded homes
Energy/ protein undernutrition
Deficiencies in Vit A, D an d C
Debilitation to intercurrent infections
prevalent among the poor decrease
resistance against infection
Children below 5 years old.
Cigarette smoking
Mode of transmission
An
PATHOGENESIS OF
TUBERCULOSIS.
initial infection
Close contact
(+) PPD test
Lowered resistance due to
alcoholism, medication therapy or
other underlying disease condition
Elderly
Clinical Manifestations
PRIMARY INFECTION
Change of behavior
Easy fatigability
Alertness to apathy
Irritability
Fleeting infection
or GIT
Crepitant rales
of the respiratory
POSTPRIMARY/PROGRESSIVE
PRIMARY
Visibly
ill
Cough that gradually becomes
distressing
Abnormal physical signs
Abnormal breath sounds
Hemoptysis (rare case)
CHRONIC PULMONARY
TB
SYSTEMIC SIGNS & SYMPTOMS
General malaise, anorexia, easy
fatigability, apathy, irritability,
indigestion, flu-like symptoms
Physical signs
Fever
Night sweat
Loss of weight
PULMONARY SIGNS
Cough
Fine crepitant rales
Hemoptysis and chest pain
Pleural pain--- pleurisy with effusion
dyspnea
Extrapulmonary TB
Mycobacterium
METHODS OF PHYSICAL
EXAMINATION
Inspection
Palpation
Auscultation
DIAGNOSIS OF THE
DISEASE
History
Physical examination
Diagnostic examination
Roentgenography (X-RAY)
Bronchoscopy & bronchography
Bacteriology Ziehl Neelsen Method
Histopathology
Hematology
TUBERCULIN TEST
1. MANTOUX TEST
A
COMPLICATIONS
Bronchial
Spontaneous
TREATMENT
Isoniazid
(INH)
Rifampicin
(RMP)
Ethambutol
(EMB)
Pyrazinamide
(PZA)
Streptomycin
( STM)
NURSING CARE
Isolation
TB
MENINGOCOCCA
L INFECTIONS
1. ACUTE
MENINCOCOCCEMIA
Usually
starts as nasopharyngitis
followed by sudden onset of high
fever, with chills, nausea, vomiting,
malaise and headache
Petechial, parpuric, ecchymotic
hemorrhages scattered all over the
entire body surface
WATERHOUSE FRIEDRICHSEN
SYNDROME- is the rapid
development of the petechia to
2. MENINGOCOCCAL
MENINGITIS
May
be ushered by a short
predromal period but often is sudden
in onset and appears to coincide with
the first symptoms of infection
Sudden
Delirium,
petechia;l
or purpuric rashes
Signs of increased ICP
Bulging fontanel in infant
Positive Macewen sign-helps to
Meningeal irritation.
Stiff
Opistothonus
Kernigs
sign
Brudzinskis
sign
MEDICATION
Rifampicin
Mynocycline
Meningococcal
polysaccharide
vaccine
Penicillin
chloramphenicol
Nursing Intervention
Patient
safety
Management of underlying
symptoms
Shock precaution- gradual decrease
in temperature is imperative
3. Bacterial Meningitides
Is
Causative agents..
Neisseria
meningitidis
Haemophilus influenza
Streptococcus pneumoniae
Incubation period
Variable
Mode of transmission
By
Period of
Communicability
24
PATHOPHYSIOLGY
Direct contact or
droplet infection
Colonization of
nasopharynx
Bacteremia
Direct
involvement of
blood vessels
Septic thrombolic
emboli
MENINGES
Clinical manifestation
Fever
Rapid
pulse;respiratory arrythmias
Hyperesthesia of the skin- increase
sensitivity to stimuli
Soreness
Sign
Cranial
nerve palsies-
a form of palsy
involving one or more of the cranial nerves.
Petechiae
Focal
cerebral signs
Encephalitis manifestations
(drowsiness-lethargy- coma
Sudden confusion
DIAGNOSTIC
PROCEDURES
1. Lumbar puncture- insertion of a
needle into the lumbar subarachnoid
space and withdrawal of CSF for
diagnostic therapeutic purpose
Complications
Subdural
effusion or empyema
Hydrocephalus
Dry tap-Not being able to collect a sample of the
fluid
Persistent
vomiting
Deafness and total blindness in one or
both eyes
Ocular conditions
Deaf- mutism
Otitis media and mastoiditis
Pyogenic
arhritis
Endocarditis, pericarditis and myocarditis
Bronchitis and pneumonia
Cystitis
Ventriculitis- inflammation of the brain
ventricles.
Motor
loss
Personality changes
Convulsions
Shock in meningococcal meningitis
TREATMENT
Ampicillin
Choramphenicol
Combination of penicillin,
Intravenous
drug therapy
Andrenocorticosteriods endotoxic
shock
Anti- convulsant drug
Nursing interventions
Prevent
Nutritional
intake
Monitoring procedures
Encourage liberal fluid intake
Be on constant alert for
complications
DIPTHERIA
An
Etiologic Agent
Corynebacterium
Loffler Bacillus)
diptheria (Klebs-
Incubation period
After
Period of
Communicability
It
Source of infection
Infection
Mode of Transmission
Transmitted
Predisposing Factors
An
Types of Diptheria
1. Nasal - with foul smelling
serosanguinous secretions from the
nose.
2. Tonsilar
The
3. Nasopharyngeal
Cervical
4. Laryngeal
Commonly
years old)
Most sever and fatal
Respiration is increased because
less air is brought to the lungs due to
the narrowing of the air passages
With moderate hoarseness, the voice
is diminished until it is finally absent
5. Wound or cutaneous
Diptheria
Affects
CLINICAL
MANIFESTATIONS
Feeling
Complications
Myocarditis
Polyneuritis
Airway obstruction
Cervical adenitis
Bronchopneumonia
Diagnostic Test
Swab
Treatment modalities
Penicillin
Antitoxin
Erthromycin
Supportive therapy
Nursing Management
Bedrest.
Diet-
BACTERIAL
PNEUMONIA
Refers
to the consolidation or
solidification of the air sacs with the
inflammatory exudate.
Etiologic agents
Streptococcus
pneumoniae
Staphylococcus aureus
Hemophilus influenzae
Klebsiela pneumoniae
Incubation period
1-3
Mode of transmission
Droplet
infection
Indirect contact
4 STAGES OF THE
DISEASE
Stage
of
Stage of
Stage of
Stage of
lung engorgement
red hepatization
gray hepatization
resolution
Clinical manifestation
Sudden
Body
malaise
Tachypnea
Rapid and bounding pulse
Flushed cheeks, bright eyes,
cyanosis
Painful cough
Labored respiration
Diaphoresis
Delirium in the acute stage of the
disease
diagnosis
physical examination
Sputum typing
Blood or serologic examinations
Neufeld- Quelling test- test for
capsular swelling
Complications
Pleurisy
Empyema or pleural effusion
Pericarditis and endocarditis with
effusion
pneumococcal meningitis
Otitis media
Hypestathic edema and hyperemia
Rare complications- arthritis, abscess
of the lungs, gangrene of the lungs,
embolus of the artery and atelactasis
Treatment
Absolute
bed rest
Adequate salt, fluid and vitamin intake
Oxygen administration
Relief of abdominal ditention, cardiac
arrhythmias and pleuritic pain
Control of pulmonary edema
Expectorants and bronchodilators
Antibiotics
Cooling enemas and potassium
permanganate irrigations
Nursing Intervention
Keep
patient warm
Do back rubbing
Do bronchial tapping
Bedrest
Elevate head and shoulders
Cushion bone prominences with pillows
Hygiene care
Increase fluid intake
High calorie diet
Monitor I and O
PERTUSSIS
(Whooping
Cough)
An
Causative agent
Bordetella
Incubation period
Seven
to
fourteen
days
Period of
communicability
Starts
Mode of transmission
Direct
contact
Droplet infection
Indirect contact
Stages of Pertussis
Catarrhal stage- most
communicable stage; lasts for 1 to
2 weeks
2. Paroxysmal stage- lasts for 4 to 6
weeks
3. Convalescent stage
1.
Clinical manifestations
Catarrhal
stage
Mucoid rhinoria
Sneezing
Lacrimation and dry bronchial cough
Irritating, hacking, nocturnal and
Paroxysmal
stage
Profuse
sweating, involuntary
urination, lethargy and exhaustion
Convulsion- intra cranial hemorrhage
Convalescent
stage
Complications
Interstitial
pneumonia
Atelactasis
Convulsions
Umbilical hernia
Otitis media
Bronchopneumonia
Severe malnutrition and starvation
Diagnostic procedures
Nasopharyngeal
Sputum
CBC
culture
swabs
TREATMENT
Supportive
therapy
Antibiotic
therapy
Hyperimmune convalescent serum
or gamma globulin are found
effective
NURSING MANAGEMENT
Isolation
Thank you