Documente Academic
Documente Profesional
Documente Cultură
MLNGCeleste, RN, MD 3
Genetic Disorders
• Inherited or genetic disorders
-disorders that can be passed from one
generation to the next
• Genetics
-Study of why disorders occur
MLNGCeleste, RN, MD 4
Nature of Inheritance
MLNGCeleste, RN, MD 6
Normal Male Karyotype
MLNGCeleste, RN, MD 7
Nature of Inheritance
• Genes
– Basic units of heredity; structures
responsible for hereditary characteristics
– May or may not be expressed or passed to the
next generation
– According to Mendel’s Law, one gene for each
hereditary property is received from each parent;
one is dominant (expressed); one is recessive
MLNGCeleste, RN, MD 8
Karyotype
• Chromosomal pattern of a cell including genotype,
number of chromosomes and normality or
abnormality of the chromosomes
Genotype
• Actual gene composition; Sequence and
combination of genes on a chromosome
Phenotype
• Outward appearance or observable expression
of genes (hair color, eye color, body build, allergies)
MLNGCeleste, RN, MD 9
Alleles
• Pairs of genes located on the same site on
paired chromosomes
• Homozygous alleles (DD or dd)
• Heterozygous alleles are two different
alleles for the same trait (Dd)
MLNGCeleste, RN, MD 10
CONGENITAL and GENETIC are not synonymous
MLNGCeleste, RN, MD 11
Dominant and Recessive Patterns
MLNGCeleste, RN, MD 12
Dominant and Recessive Patterns
MLNGCeleste, RN, MD 13
• Homozygous dominant - an individual with 2
homozygous genes for a dominant trait
MLNGCeleste, RN, MD 14
Their children have a 100% chance of being
heterozygous for the trait.
Phenotype – brown eyed (phenotype) ; but
they will carry a recessive gene for blue eyes in
their genotype.
MLNGCeleste, RN, MD 15
The child will have an equal chance of being
brown eyed (50%) or blue eyed (50%).
MLNGCeleste, RN, MD 16
All the children will be brown- eyed. Chances
are equal that their children will be
homozygous dominant (50%) like the father or
heterozygous (50%) like the mother.
MLNGCeleste, RN, MD 17
Both parents are heterozygous. 25% chance of
their children being homozygous recessive
(blue-eyed), 50% chance of being
heterozygous (brown eyed) and a 25% chance
of being homozygous dominant (brown eyed).
MLNGCeleste, RN, MD 18
Inheritance of Disease
Mendelian or Single gene disorders
A. Autosomal disorders
1. Autosomal dominant disorders
2. Autosomal recessive disorders
B. Sex – linked disorders
1. X-linked dominant inheritance
2. X-linked recessive inheritance
Multifactorial inheritance
Chromosomal aberrations or abnormalities
MLNGCeleste, RN, MD 19
Autosomal disorders
• Occur in any chromosome pair other than the
sex chromosomes
• Result from a single altered gene or a pair of
altered genes on one of the first 22 pairs of
autosomes
• Autosomal dominant or
Autosomal recessive
MLNGCeleste, RN, MD 20
Autosomal dominant traits
• Those in which the abnormal gene dominates
the normal gene; thus, the condition is always
demonstrated when the abnormal gene is
present.
• The affected parent has a 50% CHANCE OF
PASSING ON THE ABNORMAL GENE IN EACH
PREGNANCY.
MLNGCeleste, RN, MD 21
Autosomal dominant traits
MLNGCeleste, RN, MD 22
Autosomal dominant
• Osteogenesis imperfecta (bones are exceedingly
brittle)
• Marfan syndrome (disorder of connective
tissue; child is thinner and taller than normal;
heart defects)
• Huntington’s disease
• Neurofibromatosis
• Achondroplasia (dwarfism)
MLNGCeleste, RN, MD 23
Family pedigrees findings
(Autosomal dominant )
MLNGCeleste, RN, MD 24
Autosomal recessive traits
• Require transmission of the abnormal gene
from both parents for demonstration of the
defect in the child
• Each child has a 50% CHANCE OF BEING A
CARRIER OF THE DISORDER
• Almost all carriers are free from symptoms
MLNGCeleste, RN, MD 25
MLNGCeleste, RN, MD 26
Autosomal recessive
• Albinism
• Sickle cell anemia (chronic intensely painful
episodes caused by obstruction of blood vessels
by odd-shaped RBC’s; precipitated by
dehydration, infection, exposure to cold,
trauma, fatigue, lack of oxygen, strenuous
physical activity)
- The primary nursing action in caring for an
adolescent in sickle cell crisis is directed at
maintaining adequate hydration
- the spleen usually becomes enlarged due to
congestion and engorgement with sickled cells
MLNGCeleste, RN, MD 27
Autosomal recessive
• Cystic fibrosis (multiple organ disease; the
primary pathophysiologic mechanism in cystic
fibrosis mucus buildup in the lungs and
pancreas; steatorrhea; azotorrhea)
• Inborn errors of metabolism (disorders caused
by the absence of or defect in enzymes that
metabolize proteins, fats or carbohydrates)
• Phenylketonuria or PKU (phenylalanine
hydroxylase) – brain damage and mental
retardation
• Tay Sach’s disease (hexosaminidase)- child is
attentive, passive and regresses in motor and
social development
MLNGCeleste, RN, MD 28
GROUP Disorder
MLNGCeleste, RN, MD 29
Family pedigrees findings
(Autosomal recessive)
• Both parents of a child with the disorder are
clinically free of the disorder
• The sex of the affected individual in
unimportant in terms of inheritance
• History of the disorder in the family is negative
• A known common ancestor between the
parents sometimes exists. This is how both
male and female have come to possess a like
gene for the disorder.
MLNGCeleste, RN, MD 30
X-linked disorders
• Result from an altered gene on the X
chromosome
• May be dominant or recessive; recessive is
more common
MLNGCeleste, RN, MD 31
Family pedigrees findings
(X-linked dominant)
• All individuals with the gene are affected
• Female children of affected men are all
affected; male children of affected men are
unaffected
• It appears in every generation
• All children of homozygous affected women
are affected.
• EXAMPLE: Hypophosphatemia
MLNGCeleste, RN, MD 32
MLNGCeleste, RN, MD 33
X- linked recessive
• More common
• Mother is the carrier of the disorder
• In female children, expression of the disease is
blocked
• In male children, disease will be manifested
MLNGCeleste, RN, MD 34
Family pedigrees findings
(X-linked recessive)
• Only males will have the disorder
• A history of girls dying at birth for unknown
reasons often exists
• Sons of an affected man are unaffected
• The parents of affected children do not have
the disorder
MLNGCeleste, RN, MD 35
MLNGCeleste, RN, MD 36
X-linked recessive
• Hemophilia
• Color blindness
• Duchenne-type muscular dystrophy
• Christmas disease
• Fragile X syndrome
MLNGCeleste, RN, MD 37
MLNGCeleste, RN, MD 38
Multifactorial inheritance
• Abnormalities caused by multifactorial reasons
which do not follow the mendelian laws of
inheritance because more than a single gene is
involved
• Environmental influences may be instrumental
in determining whether the disorder is
expressed
• Difficult to counsel parents regarding these
disorders because their occurrence is
unpredictable
MLNGCeleste, RN, MD 39
Multifactorial inheritance
• Cleft lip or palate
• Neural tube disorders
• Mental illness
• Pyloric stenosis
• Hypertension
• Heart disease
• diabetes
MLNGCeleste, RN, MD 40
Genetic Counseling
• Purposes
– Provide accurate information
– Provide reassurance
– Make informed choices
– Educate people about disorders
– Offer support
MLNGCeleste, RN, MD 41
Nursing Responsibilities
• Alert couple to what procedures they can
expect to undergo
• Explain how genetic screening tests are done
and when they are offered
• Assess for signs and symptoms of genetic
disorders
• Offer support
• Assist in value clarification
• Educate on procedures and tests
MLNGCeleste, RN, MD 42
Assessing for Genetic Disorders
• History
• Physical assessment
Diagnostic testing
• Karyotyping – visual presentation of
chromosomes (sample: peripheral venous
blood; scraping of cells from buccal membrane)
• Barr body determination – if a child is born with
ambiguous genitalia; scraping of cells from
buccal membrane; stained and magnified;
presence of nondominant X chromosome in
the nucleus- Barr body (chromosomally female)
MLNGCeleste, RN, MD 43
Assessing for Genetic Disorders
AFP analysis
• alpha fetoprotein (AFP) is a glycoprotein produced by
the fetal liver
• AFP level in the amniotic fluid or maternal serum will
differentiate from normal if a chromosomal or a spinal
cord disorder is present (eg, in mothers who have
gestational diabetes; infants 10x risk of having a neural
tube defect)
• Serum test is done at 15th week of pregnancy; if result
is abnormal, amniotic fluid will be assessed
• elevated 3-5x in amniotic fluid secondary to leakage
from open neural tube
• low AFP, < 5% Down syndrome
• maternal serum AFP has a false positive rate 30%; use
of triple study (AFP, estriol and hCG) reduces false
positive rate
MLNGCeleste, RN, MD 44
Assessing for Genetic Disorders
Chorionic villi sampling
• Retrieval and analysis of chorionic villi for
chromosome analysis
• Transcervical or transabdominal; may be done
as early as 5 weeks, but more commonly done
at 8-10 weeks of pregnancy
• Risks: bleeding/ loss of pregnancy; limb
reduction syndrome; infection
• Diagnosis of Sickle cell disease, thalassemia
MLNGCeleste, RN, MD 45
Chronic villi sampling
MLNGCeleste, RN, MD 46
Assessing for Genetic Disorders
Amniocentesis
• Withdrawal of amniotic fluid from the
abdominal wall for analysis at 14th to 16th
week of pregnancy
• May include karyotyping, analysis of AFP and
acetylcholinesterase
• Used to diagnose potential genetic problems
in the fetus (Down Syndrome), to estimate
fetal lung maturity or to diagnose fetal
hemolytic disease
MLNGCeleste, RN, MD 47
Amniocentesis
MLNGCeleste, RN, MD 48
Assessing for Genetic Disorders
Percutaneous umbilical blood sampling
• removal of blood from the umbilical cord using
an amniocentesis technique
• more rapid karyotyping
MLNGCeleste, RN, MD 49
Percutaneous
umbilical blood sampling
MLNGCeleste, RN, MD 50
Fetoscopy
• insertion of a fiberoptic fetoscope through a
small incision in the mother’s abdomen into the
uterus and membranes to inspect the fetus for
gross abnormalities
• can be used to confirm sonography finding,
remove skin cells for DNA analysis or perform
surgery for a congenital defect
Preimplantation diagnosis
• may be possible in the future
• to remove the fertilized ovum from the uterus
before implantation for biopsy or cell analysis
MLNGCeleste, RN, MD 51
Legal and Ethical Aspects
MLNGCeleste, RN, MD 52
Common Chromosomal Disorders
MLNGCeleste, RN, MD 53
1. Trisomy 13 syndrome
(Patau syndrome)
• Children have extra chromosome 13
• Severely cogitively challenged
• Incidence is low, .45 per 1,000 live births
• Midline body disorders present, microcephaly,
with abnormalities of the forebrain and forehead
• Eyes are smaller than normal (microphthalmos) or
absent
• Cleft lip and palate
• Low set ears
• Heart defects, VSD
• Abnormal genitalia
• Most do not survive beyond early MLNGCeleste, RN, MD
childhood 54
2. Trisomy 18 syndrome
• 3 Number 18 chromosomes
• Severely cognitively challenged
• Incidence .23 per 1,000 live births
• Small for gestational age (SGA)
• Low set ears, small jaw, congenital heart
defects, misshapen fingers and toes (Index
deviates or crosses over other fingers)
• Soles of the feet are rounded not flat (rocker-
bottom feet)
• Do not survive beyond early infancy
MLNGCeleste, RN, MD 55
3. Cri-du-chat syndrome
MLNGCeleste, RN, MD 56
4. Turner syndrome
- female with only 1 X chromosome
• Gonadal dysgenesis, 45XO
• Has only 1 functional X chromosome
• Short in stature
• Hairline at the nape is low set
• Neck may appear webbed and short
• May have edema of the hands and feet
• Congenital anomalies, eg, coarctation (stricture) of the aorta;
kidney disorders
• Streak (small and nonfunctional) gonads; may have pubic
hair in puberty, no other secondary characteristics
• Incidence is 1 per 10,000 live births
• On karyotyping, 1 X chromosome only (no Barr body
present)
• Lack of fertility; learning disabilities; socioemotional
problems
• Growth hormone may help achieve additional height;
Estrogen may induce withdrawal bleeding MLNGCeleste, RN, MD
57
5. Klinefelter syndrome
- male with an extra X chromosome
MLNGCeleste, RN, MD 58
6. Fragile X syndrome
• X linked, 1 long arm of the X chromosome is defective
• 1 in 1,000 livebirths
• Most common cause of cognitive challenge in boys
• Before puberty – maladaptive behaviors: hyperactivity
and autism
• Reduced intellectual functioning (speech and
arithmetic)
• Large head, long face with a high forehead, prominent
lower jaw, large protruding ears
• Hyperextensive joints, cardiac disorders
• After puberty – enlarged testicles; fertile
• Folic acid and phenothiazine may improve symptoms
of poor concentration and impulsivity; intellectual
function cannot be improved
MLNGCeleste, RN, MD 59
7. Down syndrome (trisomy 21)
MLNGCeleste, RN, MD 62
Reproductive
and Sexual Health
MLNGCeleste, RN, MD 63
Reproductive Anatomy and Physiology
MLNGCeleste, RN, MD 64
Reproductive Anatomy and Physiology
MLNGCeleste, RN, MD 65
MALE REPRODUCTIVE SYSTEM
MLNGCeleste, RN, MD 66
MALE REPRODUCTIVE SYSTEM: ANDROLOGY
A. External Structures
1. Penis: the male organ of copulation; a cylindrical shaft consisting
of:
a. corpora cavernosa -two lateral columns of erectile
tissue
b. corpus spongiosum - encases the urethra
3. Testes: two solid ovoid organs 4-5 cm long and 2-3 cm wide,
divided into lobes containing
Seminiferous tubules -produce spermatozoa
Leydig cells - testosterone production
67
Parts of the Penis:
2. Shaft or body
68
MALE REPRODUCTIVE SYSTEM:
A. External Structures continued
LH - release of Testosterone.
MLNGCeleste, RN, MD 72
MALE REPRODUCTIVE SYSTEM:
B. Internal Structures continued
3. Seminal vesicles: are two convoluted pouches that lie
along the lower portion of the bladder and empty into the
urethra by the way of the ejaculatory ducts
73
MALE REPRODUCTIVE SYSTEM:
B. Internal Structures continued
SEMEN:
• Is a thick whitish fluid ejaculated by the male during orgasm,
contains spermatozoa and fructose-rich nutrients.
• During ejaculation, semen receives contributions of fluid from
Prostate gland (60%)
Seminal vesicle (30%)
Epididymis ( 5%)
Bulbourethral gland (5%)
• Average pH = 7.5
• The average amount of semen released during ejaculation is
2.5 -5 ml. It can live with in the female genital tract
for about 24 to 72 hours.
• 50-200 million/ml of ejaculation
• ave. of 400 million/ejaculation
• 90 seconds- cervix
• 5 minutes- end of fallopian tube
74
Spermatogenesis
Testes
Ejaculatory duct
OUT
MLNGCeleste, RN, MD
Reproductive Anatomy and Physiology
MLNGCeleste, RN, MD 77
EXTERNAL REPRODUCTIVE SYSTEM
MLNGCeleste, RN, MD 78
FEMALE REPRODUCTIVE SYSTEM: GYNECOLOGY
A.External Structures
82
Female reproductive system
Internal structures
MLNGCeleste, RN, MD 83
FEMALE REPRODUCTIVE SYSTEM:
B. Internal Structures
84
Ovary - firm almond shaped organ covered by the
peritoneum
3 principal divisions:
a. protective layer of surface
epithelium
2. Fallopian Tubes – 4 inches (10 cm) long from each side of the
fundus
Functions:
1. receives the ova to fallopian tube; place for implantation and
nourishment during fetal growth; furnishes protection to a growing
fetus
2. aids in labor and delivery
88
3 main parts of the Uterus
1. Fundus- rounded portion superiorly
2. Corpus or Body- major portion
3. Cervix- outlet which protrudes into vagina
• Isthmus- junction between the body and the cervix
• POSITION: Anteverted and Anteflexed
MLNGCeleste, RN, MD 89
Layers of Uterine wall
1. endometrium (or mucosa) – inner layer
2. myometrium – thick, middle circular layer
(stratum vasculare)
3. epimetrium- superficial part surrounded
by the perimetrium
MLNGCeleste, RN, MD 90
Layers of the Endometrium
1. Stratum Functionale
– Stratum compactum
– Stratum spongiosum
2. Stratum basale or germinativum
MLNGCeleste, RN, MD 91
MLNGCeleste, RN, MD 92
FEMALE REPRODUCTIVE SYSTEM:
Uterus continue
Nerve Supply:
Efferent (motor) nerve- spinal ganglia (T5 to T10)
Afferent (sensory) nerve - hypogastric plexus (T-11 & T-12)
Impt: Controlling pain in labor ( Epidural anesthesia)
Uterine Ligaments:
1. Broad Ligaments – from the sides of uterus to pelvic walls
93
FEMALE REPRODUCTIVE SYSTEM:
3. Vaginal Canal – 3-4 inch long dilatable canal between the bladder
and the rectum; contains rugae that permits stretching without
tearing.
95
Vaginal canal
• Connects the cervix to the vestibule
• Fibromuscular walled tube lined with mucus and
covered with hymen
• hymen – vascular and tends to bleed when ruptured
• The remnant of hymen is called CARUNCULAE
MYRTIFORMIS
• Bulbocavernosus: a circular muscle acts as voluntary
sphincter (Kegel exercises)
MLNGCeleste, RN, MD 96
MLNGCeleste, RN, MD 97
Variations of Uterine Formation
MLNGCeleste, RN, MD 99
Anteversion Anteflexion
Retroversion Retroflexion
Female Male
Glans Clitoris Glans penis
Labia majora Scrotum
Vagina Penis
Ovaries Testes
Fallopian tubes Vas deferens
Skene’s glands Prostate glands
Bartholin’s glands Cowper’s glands
Ovum Spermatozoa
108
Reproductive Development
• Intrauterine development
-sex of an individual is determined at the moment of
conception
• Gonad- body organ that produces sex cells (ovary, testis)
PUBERTAL DEVELOPMENT:
1. growth spurt
2. increase in the transverse diameter
of the pelvis
3. breast development (thelarche)
4. growth of pubic hair (adrenarche)
5. onset of menstruation (menarche
12.5 y/o ave.)
-Ovulation occurs 1 – 2 years after
menarche
6. growth of axillary hair (adrenarche)
7. vaginal secretion
MLNGCeleste, RN, MD
MENSTRUAL CYCLE / FEMALE REPRODUCTIVE CYCLE
= EPISODIC UTERINE BLEEDING IN RESPONSE TO
HORMONAL CHANGES
= PERIODIC SERIES OF CHANGES THAT RECUR IN THE
UTERUS AND ASSOCIATED ORGANS BEGINNING AT
PUBERTY AND ENDING AT MENOPAUSE
= TAKEN FROM THE FIRST DAY OF MENSTRUATION TO
THE FIRST DAY OF THE NEXT MENSTRUATION
118
Basis for menstrual cycle is 6-12 month graphing.
Menarche – first menstrual period that occurs
typically at age 12 but may occur as early as 9 or as
late as 17
Thelarche – development of the breast buds that
occur at puberty
Adrenarche – development of pubic & axillary hair
due to androgen stimulation
119
BODY STUCTURES INVOLVED IN MENSTRUATION
1. HYPOTHALAMUS – ultimate initiator of menstrual
cycle. Secretes GnRH. Releases FSHRF during the
first half of the cycle & LHRF during the second half
of the cycle.
2. ANTERIOR PITUITARY GLAND – releases the
gonadotropin hormones (GH) FSH & LH
3. OVARIES- site of ovulation & releases estrogen &
progesterone.
4. UTERUS – the organ from which menstrual
discharge is formed. The changes in the uterine
endometrium are due to ovarian hormones
120
PITUITARY HORMONES ( GONADOTROPIC HORMONES)
WHICH REGULATE MENSTRUAL CYCLIC ACTIVITIES:
1. FOLLICLE STIMULATING HORMONE ( FSH)
2. LUTEINIZING HORMONE ( LH )
OVARIAN HORMONES WHICH REGULATE MENSTRUAL
CYCLE ACTIVITIES:
1. ESTROGEN – hormone of women; produced by the
graafian follicle
2. PROGESTERONE – hormone of mothers; produced by
the corpus luteum
121
HORMONES
OOCYTES
• in utero - 5 to 7 million
• at birth - 2 million
• 7 yrs of age only - 500,000/ovary
• Reproductive age only - 400–500 oocytes
• Menopause - none
2. Proliferative/Pre-ovulatory phase
– Increased FSH and Estrogen in small amounts
STEPS:
1. Corpus luteum of previous cycle fades,
progesterone decreases, FSH rises
(proliferative phase)
B. CESSATION OF MENSES:
- menses usually cease between Ages of 45 and 52
years,
(reduced level of estrogen from the remaining follicles
is no longer sufficient to induce endometrial
proliferation / changes capable of producing visible
menstruation)
• Uterus
- endometrial tissue become sparse, with numerous small
petecchial hemorrhages, has atrophic appearance
- myometrium atrophies, uterus decreases in size
MLNGCeleste, RN, MD 155
• Breast
- general loss of turgor, form, fullness of the breast
• Bones
- gradual loss of calcium, lading to osteoporosis,
characterized by reduction in bone density and fracture
• Hair
- with the loss of estrogen, there is relative decrease in
circulating androgens; increase quantity of hair with
male pattern distribution
Vasomotor symptoms:
- Hot flash/ flush, is the hallmark of the
menopausal woman
- last for a few seconds or several minutes
- more frequent and severe at night or during
time of stress
- coincides with a surge of luteinizing
hormones
• osteoporosis:
– Main health hazard associated with
menopause
MLNGCeleste, RN, MD 158
Menopausal syndrome:
-Such as fatigue, headache, nervousness, loss of libido,
insomnia, depression, irritability, palpitation, muscle
pain
•Atrophic changes:
- atrophy of the vaginal mucosa leads to atrophic
vaginitis, pruritus of vulvovaginal area, dyspareunia and
stenosis
- urethral changes
- increased frequency of cystitis
- vaginal, urethral and bladder symptoms
Prostaglandin myometrial
contractions muscle spasm
constricts blood vessels ischemia
and pain
Diagnosis
History and PE
MLNGCeleste, RN, MD 164
Medical Management
1. combination OCP – inhibit ovulation, decrease
prostaglandin and uterine activity
2.promote exercise
3.administer prostaglandin synthesis inhibitors –
ibuprofen, mefenamic acid
Nursing Management
1. Education and reassurance
2. adequate nutrition and rest
3. stress management
Management:
supportive
• Decidua
• Chorionic villi
• Placenta
Amniotic Membranes
• Chorionic membrane
• Amniotic membrane
II. FERTILIZATION
• union of ovum and spermatozoa
• union generally occurs in the distal third
of the fallopian tube
MLNGCeleste, RN, MD 189
• Cells of the human body develop from
chromosomes
• Normal human cell tissue contains 46 chromosomes-
22 pairs of homologous autosomes (any chromosome
other than sex chromosome) and one pair of sex
chromosomes; one chromosome of each pair of
chromosomes is received from the mother and the
other one from the father
• Sex determination occurs at the moment of conception
as a result of the sex chromosome contributed by the
male; an X-carrying sperm fertilizing the ovum
produces a female (XX), a Y-carrying sperm produces a
male (XY)
• Aberration in the number of chromosomes result in
abnormal offspring or spontaneous abortion
MLNGCeleste, RN, MD 190
Process of fertilization (conception)
• Cell division:
- occurs as the zygote travels the fallopian tube to the
uterus.it takes 3 to 4 days of cell division or mitosis for the
zygote to become morula( resemble mulberry), this
morula entering the uterus is now
called a blastocyst
• Fraternal or dizygotic
- 2 ova are being
fertilized by 2 sperm,
they are nonidentical,
there are 2 amnion, 2
chorion, 2 placenta
195
Formation of twins:
• Identical or monozygotic
twins:
- one ovum is fertilized by
one sperm and the inner
cell mass of the
blastocyst splits into 2 to
form two embryos
- maybe 2 males or 2
females, there are 2
amnion , one chorion
and one placenta
196
• Chorion - outer fetal membrane,
formed from the trophoblast
( maternal side of placenta)
• Amnion - originates in the blastocyst
during early stages of development,
expands as the fetus grows until it
slightly adheres to the chorion ( fetal
side of placenta)
• Amniotic sac - formed by 2 fetal
membranes (chorion, amnion)
MLNGCeleste,
MLNGCeleste, RN, MD RN, MD
219
week 8 - resembles human being, eyes move to face front,
heart development complete, hands and feet well
formed; bone cells begin to replace cartilage, all body
organs have begun forming
(wt-2g, L 3cm,) MLNGCeleste, RN, MD 220
Fetal Stage
MLNGCeleste,
MLNGCeleste, RN, MD RN, MD
227
System development:
-all systems in the fetus begin forming by the 8th
week
• cardiovascular system -primitive heart begins to
beat on the 21st day following conception ,the
1st to function in the embryo; congenital
malformation may develop during the 6th to 8th
weeks
228
Fetal
Circulation
umbilical vein
right atrium
right atrium
right ventricle
pulmonary arteries
( ductus arteriosus)
aorta
• Reproductive system:
- testes seen on abdomen by 7 weeks, and
begin to descend to the scrotum about 30 weeks;
ovaries develop in the abdomen and stay in the
pelvic cavity
• Respiratory system:
- lung buds form during the 6th week
- bronchi form by week 16
• Immunologic system:
- between 12-15th weeks immune capability
begins to develop
- fetus produces small amount of immunoglobulin
IgA, IgG, and IgE
-embryonic stage:
week 3 - primitive nervous system, eyes, ears,
rbc present, heart begins to beat day 21
3 PHASES:
247
COMPONENTS OF PRE NATAL VISIT
1.PRE- CONSULTATION PHASE:
PERSONAL DATA: AGE, SEX, CIVIL STATUS, WEIGHT,
HEIGHT
1 AGE : UNDER 17 OR ABOVE 35 (GREATER RISK IF
OVER 40)
** PREGNANT ADOLESCENTS HAVE A HIGHER
INCIDENCE OF PREMATURITY, PIH, CEPHALOPELVIC
DISPROPORTION, POOR NUTRITION & INADEQUATE
ANTEPARTAL CARE.
** WOMEN OVER 35 YEARS OLD ARE AT RISK FOR
CHROMOSOMAL DISORDERS IN INFANTS, PIH &
CESARIAN DELIVERY.
248
** THE DURATION OF A NORMAL PREGNANCY
IS 266 – 280 DAYS OR 38-42 WEEKS ( AVERAGE IS 40
WEEKS) ; OR 9 CALENDAR MONTHS OR 10 LUNAR
MONTHS.
** BOTH OVULATION & GESTATIONAL AGE ARE
ALSO SOMETIMES MEASURED IN LUNAR MONTHS ( 4
WEEK PERIODS) OR IN TRIMESTERS ( 3 MONTH
PERIOD) RATHER THAN IN WEEKS.
249
MLNGCeleste, RN, MD
Health Assessment
• Initial interview
– Health history
• Demographic data
• Chief concern • History of family illness
• Family profile • Gynecologic history
• History of past • Obstetric history
illnesses • Review of systems
251
Health Assessment
• Initial interview
– Support person’s role
– Physical exam
• Baseline height/weight, vital signs
• Assessment of systems
252
• Assessment of systems
– General appearance and
mental status
– Neck
– Head and scalp
– Eyes – Lymph nodes
– Nose – Heart
– Ears – Lungs
– Sinuses – Back
– Mouth, teeth and throat
– Rectum
– Extremities and skin
253
OBSTETRICAL DATA:
MENSTRUAL HISTORY: INCLUDES MENARCHE,
LENGTH & REGULARITY OF MENSES, INTERVAL
BETWEEN PERIODS, AMOUNT OF FLOW,
DYSMENORRHEA
HISTORY OF PAST PREGNANCIES:
GRAVIDA = ALL PREGNANCIES REGARDLESS OF
DURATION OR OUTCOME
PARA = PAST PREGNANCIES RESULTING IN
VIABLE FETUS ( 20 WEEKS) WHETHER BORN DEAD
OR ALIVE. ( TWINS, TRIPLETS ETC. CONSIDERED
AS ONE).
254
History
1. Initial visit
a. Obstetrical history (TPAL)
MLNGCeleste, RN, MD
• Para – number of past pregnancies that have gone
beyond the period of viability (capability of the fetus to
survive the outside of the uterus; currently considered
any time after 20-wk gestation), regardless of the
number of fetuses or whether the infant was born alive
or dead
• Nullipara – a woman who has never delivered a fetus
that reached the age of viability
• Primipara – a woman who has completed one
pregnancy to viability
• Multipara – a woman who has completed two or more
pregnancy to the age of viability
MLNGCeleste, RN, MD
• Term infant – an infant born between 38 and 42
weeks of gestation
• Preterm – an infant born before 38 weeks
• Post term – an infant born after 42 weeks
• Abortion – pregnancy that terminates before
the period of viability (20 wks)
• Live birth – a live birth is recorded when an
infant born shows sign of life
MLNGCeleste, RN, MD
• Low birth weight < 2500 grams
• Normal Birth weight 2500 – 4000 grams
• Large birth weight > 4000 grams
B. LMP
Estimated Date of Delivery/ Confinement
EDD/ EDC/EDB
Age of gestation
Measure Fundic Height
January 30 days
February 28 Total = 242 days
March 31 AOG = 242
April 30 7
May 31 34 to 35 weeks
June 30
July 31
August 31 MLNGCeleste, RN, MD 264
Obstetrical History/ Number
G__ P__ (T, P, A, L)
• Gravida – the total number of pregnancies regardless of
duration (includes present pregnancy)
• Para – number of past pregnancies that have gone
beyond the period of viability (capability of the fetus to
survive the outside of the uterus; currently considered
any time after 20-wk gestation), regardless of the
number of fetuses or whether the infant was born alive
or dead
• Term infant – an infant born between 38 and 42 weeks
of gestation
• Preterm – an infant born before 38 weeks
• Post term – an infant born after 42 weeks
• Abortion – pregnancy that terminates before the period
of viability (20 wks)
• Live birth – a live birth is recorded when an infant born
shows sign of life MLNGCeleste, RN, MD 265
OTHER COMPUTATIONS (Nice to know):
MC DONALD’S RULE = ( ESTIMATION OF AOG IN
MONTHS & WEEKS BY FUNDIC HEIGHT
MEASUREMENT)=
FORMULA :
FUNDIC HEIGHT IN CMS X 2/7
EXAMP[LE:
FUNDIC HEIGHT IS 21 CMS
21 CMS X 2 =42
42/ 7 = 6 ( AOG IN MONTHS)
6 MONTHS X 4 = 24 ( AOG IN WEEKS)
266
HAASE’S RULE = ESTIMATION OF FETAL LENGTH
RULE:
**DURING THE FIRST HALF OF PREGNANCY, SQUARE
THE NUMBER OF THE MONTH ( EX. FIRST LUNAR
MONTH: 1X1 = 1CM.
**DURING THE SECOND HALF OF PREGNANCY,
MULTIPLY THE MONTH BY 5
( EX. 6TH LUNAR MONTH: 6X5 = 30 CM.)
FORMULA: 1 TO 5 MONTHS = MONTHS SQUARED
267
EXAMPLES:
5 MONTHS X 5 = 25 CMS LENGTH
8 MONTHS X 5 = 40 CMS LENGTH
268
JOHNSON’S RULE = ESTIMATION OF WEIGHT IN GRAMS
FORMULA: FUNDIC HEIGHT IN CM – N X K
“K” IS CONSTANT, IT IS ALWAYS 155
“N” IS MINUS 11 IF PART IS NOT YET ENGAGED
MINUS 12 IF PART IS ALREADY ENGAGED
269
BARTHOLOMEW’S RULE = ESTIMATION OF AOG BY
THE RELATIVE POSITION OF THE UTERUS IN THE
ABDOMINAL CAVITY.
** BY THE 3RD LUNAR MONTH, THE FUNDUS IS
PALPABLE SLIGHTLY ABOVE THE SYMPHYSIS PUBIS
** ON THE 5TH LUNAR MONTH, THE FUNDUS IS AT THE
LEVEL OF THE UMBILICUS
** ON THE 9TH LUNAR MONTH , THE FUNDUS IS
BELOW THE LEVEL OF THE XIPHOID PROCESS
270
2. CONSULTATION PHASE = PHYSICAL ASSESSMENT
A. PHYSICAL EXAMINATION = A REVIEW OF SYSTEMS
IS INDICATED, INCLUDING INSPECTION OF THE
TEETH BECAUSE THEY ARE A COMMON CAUSE OF
INFECTION.
B. PELVIC EXAMINATION
(CARDINAL RULE: EMPTY THE BLADDER FIRST)
** INTERNAL EXAMINATION (IE) = TO DETERMINE
CHADWICK’S, GOODEL’S, HEGAR’S
271
Physical assessment
Initial visit – complete physical exam
273
E. URINE EXAMINATIONS:
** HEAT & ACETIC ACID TEST TO DETERMINE
ALBUMINURIA. ANY SIGN OF ALBUMIN ( PROTEIN) IN
THE URINE SHOULD BE REPORTED IMMEDIATELY
BECAUSE IT IS A SERIOUS SIGN OF TOXEMIA ( PIH).
** BENEDICT’S TEST FOR GLYCOSURIA, A SIGN
OF POSSIBLE GESTATIONAL DIABETES.SPECIMEN
SHOULD BE TAKEN BEFORE BREAKFAST TO AVOID
FALSE POSITIVE RESULT
274
Laboratory screening
• Initially and at routine visits, urine dipstick for glucose,
protein (pregnancy induced hypertension and UTI), CBC,
rubella IgG antibody
• Repeat GC culture late third trimester (more often if
indicated)
• Maternal serum alpha-fetoprotein (AFP) at 16-18 wk to
identify risk of neural tube defect in fetus
• Glucose screening between 24-28 wk to detect
gestational diabetes
• Repeat CBC at 24 –28 wk
• Rh antibody titers for Rh woman at 24, 28, 32, and 40 wk
• ultrasound
280
MLNGCeleste, RN, MD 281
Location of the fundus:
283
Fetal heart rate
• FHR should be 120-160
beats per minute
284
Fetal heart rate
• Assist the patient to a supine position.
• Drape her with a blanket to minimize exposure.
• Apply water soluble lubricant to her abdomen or the
monitoring device.
• To assess FHR in a fetus 20 weeks or younger, position
Doppler/Stethoscope/ fetoscope on the abdominal midline
above the symphysis pubis. After 20 weeks AOG, when you
can palpate fetal position, use Leopold’s maneuvers and
position the listening instrument over the fetal back.
• Place the earpieces in your ears and press gently on the
patient’s abdomen. If there are no earpieces, turn the
device on and adjust the volume. As needed. Start listening
at the midline, midway between the umbilicus and the
symphysis pubis.
• Move the instrument from side to side to locate the
loudest heart tones then palpate the maternal pulse.
MLNGCeleste, RN, MD 285
Fetal heart rate
• If the maternal radial pulse and FHR are the same, try to
locate the fetal thorax/ back by Leopold’s maneuver, then
reassess FHR for 60 seconds. Record FHR.
• During labor, monitor FHR during the relaxation period
between the contractions to determine baseline.
• In a low-risk labor, assess FHR every 60 minutes during the
latent phase, every 30 minutes during the active phase and
then every 15 minutes during the 2nd stage of labor. In
high risk labor, assess FHR every 30 minutes during the
latent phase, every 15 minutes during the active phase,
and every 5 minutes during the 2nd stage of labor.
• Auscultate FHR during a contraction and for 30 seconds
afterward to identify the response to the contraction.
• Auscultate FHR before administration of medications,
ambulation, and artificial rupture of membranes, changes
in the characteristics of contractions, vaginal examinations
and medications.
MLNGCeleste, RN, MD 286
LOCATING FETAL HEART SOUNDS BY FETAL POSITION
FHT – heard best at the FETAL BACK
b. True
• “INFERIOR HALF”
• FORMED BY THE PUBIS IN FRONT, THE ILIA &
THE ISCHIA ON THE SIDES & THE SACRUM &
COCCYX BEHIND
forms the passageway of the fetus during labor
MLNGCeleste, RN, MD 310
MLNGCeleste, RN, MD 311
312
** THE FALSE PELVIS IS DIVIDED FROM THE TRUE
PELVIS ONLY BY AN IMAGINARY LINE: THE LINEA
TERMINALIS DRAWN FROM THE SACRAL PROMINENCE
AT THE BACK TO THE SUPERIOR ASPECT OF THE
SYMPHYSIS PUBIS AT THE FRONT OF THE PELVIS. **
313
a.PELVIC INLET / pelvic brim= ENTRANCE TO
THE TRUE PELVIS, OR THE UPPER RING OF
BONE THROUGH WHICH THE FETUS MUST
FIRST PASS TO BE BORN VAGINALLY. ITS
TRANSVERSE DIAMETER IS WIDER THAN ITS
AP DIAMETER. THUS:
** TRANSVERSE DIAMETER = 13.5 CM
** AP DIAMETER = 11 CM
314
315
MLNGCeleste, RN, MD 316
• Consists of the following parts:
1. Inlet/ pelvic brim – entrance to true pelvis
• AP diameters:
– Diagonal Conjugate = 12.5 cm
– Obstetric Conjugate = 11 cm
(Substract 1-1.5cm from diagonal conjugate)
– True Conjugate/ Conjugate Vera = 11.5 cm
(or 10.5 – 11cm)
(Substract 1-1.5 cm (or 1.2-2cm) from
diagonal conjugate)
• Transverse diameter = 13.5 cm
• Right and left oblique diameter = 12.75 cm
MLNGCeleste, RN, MD 317
DIAGONAL CONJUGATE
• The distance between (the anterior surface of)
the sacral promontory of the sacrum and (the
anterior surface of the inferior margin of) the
symphysis pubis
• Measured clinically
• Most useful measurement for estimating the
pelvic size (AP diameter of pelvic inlet)
• AVERAGE = 12.5 TO 13 CMS
• >12.5 cm adequate for birth
324
MLNGCeleste, RN, MD
3. Pelvic Outlet –most important diameter of the
outlet is its transverse diameter or Bi-ischial
diameter =11.5 cm
AP diameter = 9.5 to 11.5 cm
Posterior sagittal diameter = 7.5 cm
FOOD SOURCES:
** PROTEIN RICH FOODS = MEAT, FISH, EGGS, MILK, POULTRY,
CHEESE, BEANS, MONGO
** VIT. A = EGGS, CARROTS, SQUASH, CHEESE, BEANS, VEGETABLES
** VIT. D = FISH, LIVER, EGGS, MILK ( EXCESS VIT.D DURING
PREGNANCY CAN LEAD TO FETAL CARDIAC PROBLEMS)
**VITAMIN E = GREEN LEAFY VEGETABLES, FISH
330
**VITAMIN C= TOMATOES, GUAVA, PAPAYA
**VITAMIN B= PROTEIN RICH FOODS
**CALCIUM/PHOSPHORUS=MILK, CHEESE
**IRON= ESPECIALLY IMPORTANT DURING THE LAST TRIMESTER
WHEN THE PREGNANT WOMAN IS GOING TO TRANSFER HER IRON
STORES FROM HERSELF TO HER FETUS SO THAT THE BABY HAS
ENOUGH IRON STORES DURING THE 1ST 3 MONTHS OF LIFE WHEN
ALL HE TAKES IS MILK(WHICH IS DEFICIENT IRON). IRON HAS A
VERY LOW ABSORPTION RATE: ONLY 10% OF THE IRON INTAKE
CAN BE ABSORBED BY THE BODY. THUS, FOR OPTIMUM
ABSORPTION, GIVE VITAMIN C.
331
IRON SHOULD BE GIVEN AFTER MEALS BECAUSE IT IS IRRITATING
TO THE GASTRIC MUCOSA.
332
** FOLIC ACID – TO PREVENT NEURAL TUBE DEFECTS ( SPINA
BIFIDA, MENINGOCOELE )
SOURCES:
** GREEN LEAFY VEGETABLES
** FRUITS
** RDA FOR SALT IN A PREGNANT WOMAN IS 3g/DAY BECAUSE
OF INC IN BLOOD VOLUME TO MAINTAIN F & E BALANCE.
333
TT IMMUNIZATION:
• TT1 GIVEN ANYTIME DURING PREGNANCY
• TT2 ONE MONTH AFTER TT1 ( 3 YEARS
PROTECTION)
• TT3 SIX MONTHS AFTER TT2 ( 5 YEARS
PROTECTION)
• TT4 ONE YEAR AFTER TT3 ( 10 YRS)
• TT5 ONE YEAR AFTER TT4 OR NEXT PREGNANCY
( LIFETIME PROTECTION)
334
** THE PROVISION OF PRENATAL CARE IS THE
PRIMARY FACTOR IN THE IMPROVEMENT OF MATERNAL
MORBIDITY & MORTALITY STATISTICS. “”
335
ANTENATAL FETAL TESTING
341
• An external ultrasound transducer and the
tocodynamometer are applied to the mother
and a tracing of at least 20 minutes’ duration
is obtained so that the FHR and the uterine
activity can be observed.
• Obtain baseline blood pressure and monitor
blood pressure frequently.
• Position mother in semi-fowler’s or side- lying
position or left lateral position to avoid vena
cava compression.
• The mother may be asked to press a button
every time she feels fetal movement; the
monitor records a mark at each point of fetal
movement, which is used as a reference point
to assess FHR response.
MLNGCeleste, RN, MD 342
RESULTS OF NST:
• REACTIVE NONSTRESS TEST:Normal/Negative
- indicates a healthy fetus
- requires 2 or more FHR accelerations of at least 15 beats
per minute, lasting at least 15 seconds from the
beginning of the acceleration to the end, in association
with fetal movement, during a 20-minute period.
Exercise:Tighten stomach
muscles and arch back
toward the ceiling. Hold.
Tighten buttocks, pelvic
floor and back muscles
and arch
back to produce hollow.
Hold. 367
G. Sit ups
1. SIT-UPS - Modified
Purpose: Strengthen abdominal
muscles. Good muscle tone is
important for maintaining good
posture, for effective pushing,
and for early return of figure
postpartum.
Exercise: Lift head and shoulders off floor, reaching hands toward
knees (lift trunk to about 45° angle). Slowly return to starting
position; do not drop back. 368
G. Sit ups
2. OBLIQUE (DIAGONAL)
SIT-UPS - Modified
Purpose: Strengthen
oblique abdominal
muscles.
5. Walking:
Most highly recommended for the pregnant
woman; ideal alternative to more strenuous
exercise. Walk uphill, downhill, and at different
speeds.
MLNGCeleste,
MLNGCeleste, RN, MD RN, MD
373
Patient Teaching:
Consult your obstetrician early in your
pregnancy. In general, you can continue your pre-
pregnant routine of exercising. Stop when
something hurts, or when you become fatigued.
Know your limits, and avoid exercising to the
point of exhaustion. It is generally advised that
you do not begin any new sport or activity during
pregnancy. You may want to taper off your sports
participation during the last few months, but you
may still continue to exercise gently. Avoid
exercising in very hot or humid weather, or at
high altitudes if you’re not used to it.
• Ovaries
• No Graafian follicles develop and no ovulation
occurs during pregnancy
• Corpus luteum of pregnancy the corpus
luteum is the chief source of hormone
progesterone during the first 12 weeks of
gestation. The corpus luteum also produces
estrogen, relaxin, inhibin and sometimes
oxytocin MLNGCeleste, RN, MD 388
• Breasts
• enlarge early in pregnancy, causing progressive
feelings of heaviness, fullness, and tenderness;
the nipple and areola become larger, darker in
color; blood vessels enlarge and become
prominent beneath the skin
414
Health Promotion During Pregnancy
• Passage
• Passenger
• Power
• Psyche
• Engagement*
• Descent
• Flexion
• Internal rotation
• Extension
• Restitution and external rotation
• Expulsion
MLNGCeleste, RN, MD 491
• Engagement - movement of the presenting part
below the plane of the pelvic inlet
• Descent – progress through the maternal pelvis;
continuous throughout labor
• Flexion – as a result of resistance from maternal
pelvis and musculature, the head flexes so that a
smaller diameter enters pelvis
• Internal rotation – head rotates from occiput
transverse or oblique position (usual position as
it enters the pelvis) to anterior/posterior at
pelvic outlet; head is under symphysis pubis and
neck is twisted
Bradycardia (<120 bpm) Fetalhypoxia or stress Place client on her left side
Maternal hypotension after Increase fluids to counteract
epidural initiation hypotension
Stop oxytocin (Pitocin) if in use
Hypoxia
MLNGCeleste, RN, MD 532
CNS anomalies
Nursing Care: First Stage
• Respect contraction time
• Change positions
• Voiding and bladder care
• Support
• Pain management
541
Providing Comfort During
Labor and Birth
Intapartal nursing management
• Stage 1
• Maternal
• Monitor vital signs, fluid and electrolyte balance,
frequency, duration, and intensity of uterine
contractions and degree of discomfort (hourly, at
minimum); urine protein and glucose with every
voiding; laboratory results; preparedness; ROM
• Provide comfort measures – e.g., positioning,
back massage/effleurage (light abdominal
stroking in rhythm with breathing during a
contraction to ease mild/moderate discomfort),
warm/cold compresses, ice chips
Complications
• Maternal – lacerations
• fetal – neonatal – soft tissue
compression or cranial injury
• Reducing anxiety
• Coping strategies
• Comfort measures
• Positioning
• Childbirth method
• Pharmacologic pain relief
Indications:
• Cephalopelvic disproportion
• Fetal malpresentation
• non reassuring EFM strip
2. Classical
- incision is made in the contractile portion of
the uterus
- risk uterine rupture
- lower segment varicosities and myomas can be
bypassed
B - breast
U - uterus
B - bowels
B - bladder
L - lochia
E - episiotomy
S - sex
H - Homan’s sign
E - emotion
621
• Elimination
• Urinary – increased output (postpartum
diuresis), urethral trauma, decreased bladder
sensation, and inability to void in the recumbent
position may cause bladder distention,
incomplete emptying and/or urinary stasis
increasing the risk of uterine relaxation and
hemorrhage and/or UTI; monitor I and O
encourage voiding every 24 h (early ambulation
and pouring warm water over perineum);
catheterization may be necessary if no voiding
after 8 h
2 3 4 5 6 7 8 9 10 11 12 13 14 15 16 17 18 19 20 21 22 23 24 25 26
1
18 DAYS
LONGEST CYCLE
2 3 4 5 6 7 8 9 10 11 12 13 14 15 16 17 18 19 20 21 22 23 24 25 26 27 28 29 30 31
1
11 DAYS
UNSAFE TIME
1 2 3 4 5 6 7 8 9 10 11 12 13 14 15 16 17 18 19 20 21 22 23 24 25 26 27 28 29 30 31
UNSAFE TIME
664
3. Cervical Changes
• Ferning or
arborization of
cervical mucus
• At the height of
estrogen stimulation
just before ovulation
• Ferning- due to
crystallization of
sodium chloride on
mucus fibers
665
Symptothermal method
• Combines BBT and cervical mucus
methods
• Estrogen suppresses
FSH and LH, thereby
suppressing ovulation
• Progesterone
decreases the
permeability of
cervical mucus
671
3. Oral Contraceptives
• Monophasic - Fixed doses of estrogen and
progesterone ; 21-28 day cycle
• Biphasic - Constant amount of estrogen
with increased progesterone
• Triphasic - Varying levels of estrogen and
progesterone
• “Morning-after pills”
• High level of estrogen
• Must be initiated within 72 hours of
unprotected intercourse
• Intramuscular injections
-administered every 12 weeks
Medroxyprogesterone (depo-provera)
-100% effective
686
5. INTRAUTERINE DEVICES
Side Effects:
• Spotting or uterine cramping
• Increased risk for PID
• Heavier menstrual flow
• Dysmenorrhea
• Ectopic pregnancy
689
6. BARRIER METHODS
• DIAPHRAGM
-mechanically blocks sperm
from entering the cervix
-soft latex dome supported by
a metal rim
-can be inserted 2 hours
before intercourse; removed
at least 6 hours after coitus
or within 24 hours
-size must fit the individual
-washable, may be used for
2-3 years
690
6. BARRIER METHODS
• CERVICAL CAP
-similar to diaphragm
but smaller
-thimble-shaped
rubber cap held onto
the cervix by suction
691
6. BARRIER METHODS
692
• MALE CONDOM
Action – prevents the ejaculate and sperm from entering
the vagina; help prevent venereal disease; effective if
properly used; OTC
695
8. Elective Termination of
Pregnancy
Procedure to deliberately end a pregnancy
before fetal viability
• Induced
(mifepristone-progesterone antagonist;
misoprostol-prostaglandin analog
• Medically induced
D&C, D&E, saline induction, hysterotomy
MLNGCeleste,
MLNGCeleste, RN, MD RN, MD
697
• To use CycleBeads a woman simply moves a
ring over the series of color-coded beads
that represent the days of her cycle.
MLNGCeleste,
MLNGCeleste, RN, MD RN, MD
698
CycleBeads are a color-coded string of
beads that represent a woman's
menstrual cycle. Each bead represents a
day of the cycle and the color helps a
woman to determine if she is likely to be
fertile that day.
MLNGCeleste,
MLNGCeleste, RN, MD RN, MD
699
The day a woman starts her period she puts
the rubber ring on the red bead.
MLNGCeleste,
MLNGCeleste, RN, MD RN, MD
700
• When the ring is on the red bead or a
dark bead, there is very low likelihood of
pregnancy, so she can have intercourse
on these days without getting pregnant.
MLNGCeleste,
MLNGCeleste, RN, MD RN, MD
701
MLNGCeleste,
MLNGCeleste, RN, MD RN, MD
702
MLNGCeleste,
MLNGCeleste, RN, MD RN, MD
703
Health Teaching
1. Remembering to move the ring
2. Checking to make sure that the ring is on the
right day.
3. Making sure that no one else moves the ring.
4. Talking to the partner about Cycle Beads and
how it works
MLNGCeleste,
MLNGCeleste, RN, MD RN, MD
704
CycleBeads
• based on a natural method of family
planning called the Standard Days Method
• developed by the Institute for Reproductive
Health at Georgetown University
• The Standard Days Method works best for
women who have regular menstrual cycles
between 26 and 32 days long.
• Days 8 through 19 of their cycles are the
days these women are likely to get pregnant
if they have unprotected intercourse. On
other days of their cycles, pregnancy is very
unlikely.
MLNGCeleste,
MLNGCeleste, RN, MD RN, MD
705
The Newborn
MLNGCeleste, RN, MD