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FACILITATION
(PNF)
V. VENKATA RAMAIAH
MPT-Ortho
Associate Professor
VAPMS College of Physiotherapy
INTRODUCTION
Proprioceptive neuromuscular facilitation (PNF) is an
approach to therapeutic exercise based on the principles
of functional human anatomy and neuro physiology .
It uses proprioceptive, cutaneous, and auditory input to
produce functional improvement in motor output .
The therapeutic techniques of PNF were first used in the
treatment of patients with paralysis and various
neuromuscular disorders in the 1950s.
Originally the PNF techniques were used for
strengthening and enhancing neuromuscular control.
Since the early 1970s, the PNF techniques have also been
used extensively as a technique for increasing flexibility
and range of motion.
Proprioceptive Neuromuscular Facilitation
(PNF) uses proprioceptive (sensory receptors found in
muscles, tendons, joints, and the inner ear that detect body and
limb motion or position) input to improve (facilitate)
neuromuscular function during human
movement.
Neuromuscular function is enhanced by
providing resistance during concentric,
eccentric, and isometric muscle actions, thereby
enhancing muscle strength and endurance,
balance and posture, and stability and mobility.
Neuromuscular function is also enhanced
during stretching techniques, thus improving
joint range of motion and muscle flexibility.
History of PNF
PNF was developed by Neurophysiologist Dr. Herman
Kabat near the mid-1940s. Prior to PNF, rehabilitation
was typically done one joint and muscle at a time.
Based on neurophysiologic principles previously
established in the early 20th century by Physiologist
Charles Sherrington, in 1946 Dr. Kabat began to look
for natural movement patterns for rehabilitating the
muscles of polio patients and soon thereafter
discovered that typical human movement patterns
involved diagonal patterns employing multiple
muscles and joints.
Around 1950, physical therapists, Maggie Knott and
Dorothy Voss, began working with Dr. Kabat in
developing PNF principles and techniques.
Moreover, in the mid- to late 1950s, Mrs. Voss,
Mrs. Knott, and Dr. Kabat began publishing their
work on PNF in physical therapy, occupational
therapy, and physical medicine scientific
journals.
In the 1960s, Physical Therapy programs at
Universities began teaching PNF principles and
techniques.
In the late 1970s, PNF principles and techniques
began to be employed by athletes to increase
joint range of motion and muscle flexibility and
strength.
PNF continues to be used currently in a variety
of physical therapy settings, such as orthopaedic,
neuro, and paediatric settings
Original Concepts of Facilitation and
Inhibition
Most of the principles underlying modern therapeutic
exercise techniques can be attributed to the work of
Sherrington, who first defined the concepts of facilitation
and inhibition.
According to Sherrington, an impulse traveling down the
corticospinal tract or an afferent impulse traveling up from
peripheral receptors in the muscle causes an impulse volley
that results in the discharge of a limited number of specific
motor neurons, as well as the discharge of additional
surrounding (anatomically close) motor neurons in the
subliminal fringe area. An impulse causing the recruitment
and discharge of additional motor neurons within the
subliminal fringe is said to be facilitatory ,
Any stimulus that causes motor neurons to
drop out of the discharge zone and away from
the subliminal fringe is said to be inhibitory .
Facilitation results in increased excitability ,
and inhibition results in decreased excitability
of motor neurons.
Thus, the function of weak muscles would be
aided by facilitation, and muscle spasticity
would be decreased by inhibition.
Sherrington attributed the impulses
transmitted from the peripheral stretch
receptors via the afferent system as being the
strongest influence on the alpha motor
neurons.
Therefore, the therapist should be able to modify the
input from the peripheral receptors and thus
influence the excitability of the alpha motor neurons.
The discharge of motor neurons can be facilitated by
peripheral stimulation, which causes afferent
impulses to make contact with excitatory neurons
and results in increased muscle tone or strength of
voluntary contraction.
Motor neurons can also be inhibited by peripheral
stimulation, which causes afferent impulses to make
contact with inhibitory neurons, resulting in muscle
relaxation and allowing for stretching of the muscle.
PNF should be used to indicate any technique in
which input from peripheral receptors is used to
facilitate or inhibit.
Basic neurophysiologic principles for PNF involved
autogenic inhibition, reciprocal inhibition,
successive induction, and irradiation.
Autogenic inhibition results in inhibition of the
agonist with agonist contraction due to stimulation
of the golgi tendon organ.
Reciprocal inhibition results in the inhibition of the
agonist muscle when the antagonist muscle is
stimulated.
Successive induction results in increased activity in
the agonist immediately after activity in the
antagonist.
Irradiation results in the overflow or spread of
energy from stronger segments to weaker segments.
Basic principles to PNF:-
PNF is an integrated approach: each treatment is directed at a
total human being, not just at a specific problem or body
segment.
Based on the untapped existing potential of all patients, the
therapist will always focus on mobilizing the patient’s reserves.
The treatment approach is always positive, reinforcing and
using that, which the patient can do, on a physical and
psychological level.
The primary goal of all treatment is to help patients achieve
their highest level of function.
To reach this highest level of function, the therapist integrates
principles of motor control and motor learning. This includes
treatment on the level of body structures, on the activity level as
well as on the participation level (ICF, International
Classification of Functioning, WHO).
Basic neurophysiologic principles:
The following useful definitions were abstracted from his
work (Sherrington 1947):
After discharge: The effect of a stimulus continues after the
stimulus stops. If the strength and duration of the stimulus
increase, the after discharge increases also. The feeling of
increased power that comes after a maintained static
contraction is a result of after discharge.
Temporal summation: A succession of weak stimuli
(subliminal) occurring within a certain (short) period of time
combine (summate) to cause excitation.
Spatial summation: Weak stimuli applied simultaneously to
different areas of the body reinforce each other (summate) to
cause excitation. Temporal and spatial summation can
combine for greater activity.
Irradiation: This is a spreading and increased strength of a
response. It occurs when either the number of stimuli or
the strength of the stimuli is increased. The response may
be either excitation or inhibition.
Successive induction: An increased excitation of the
agonist muscles follows stimulation (contraction) of their
antagonists. Techniques involving reversal of antagonists
make use of this property (Induction: stimulation,
increased excitability.).
Reciprocal innervation (reciprocal inhibition): Contraction
of muscles is accompanied by simultaneous inhibition of
their antagonists. Reciprocal innervation is a necessary
part of coordinated motion. Relaxation techniques make use of
this property.
“The nervous system is continuous throughout its
extent – there are no isolated parts.”
Therapeutic Goals
The basic facilitation procedures provide tools for
the therapist to help the patient gain efficient
motor function and increased motor control.
These basic procedures are used to:
Increase the patient’s ability to move or remain
stable.
Guide the motion by proper grips and
appropriate resistance.
Help the patient achieve coordinated motion
through timing.
Increase the patient’s stamina and avoid fatigue.
Basic Principle of PNF
The basic procedures for facilitation are:
Resistance: To aid muscle contraction and
its direction.
Increase strength.
to contract
To give the patient security and confidence.
motion.
Facilitate control of the direction of the resistance.
fatiguing.
More effective control of the patient’s motion came
do it.
The therapist must always bear in mind that the command
unnecessary words.
They may be combined with passive motion to teach the
Desired movement.
The timing of the command is important to
coordinate the patient’s reactions with the therapist’s
hands and resistance.
It helps give the patient corrections for motion or
stability.
Timing of the command is also very important when
using the stretch reflex.
The initial command should come immediately before
the stretch the muscle chain to coordinate the
patient’s conscious effort with the reflex response .
The action command is repeated to urge greater effort or
redirect the motion.
In reversal techniques, proper timing between verbal
commands and muscle activity is important when we
change the direction of the resistance.
The volume of the command is given can affect the
strength of the resulting muscle contractions .
The therapist should give a louder command when a
strong muscle contraction is desired and use a softer
and calmer tone when the goal is relaxation or relief of
pain.
The command is divided into three parts:
1. Preparation: readies the patient for action
2. Action: tells the patient to start the action
3. Correction: tells the patient how to correct and modify
the action.
For example, the command for the lower extremity
pattern of flexion-adduction-external rotation with knee
flexion might be [preparation] “ready, and”; [action] “now
pull your leg up and in”; [correction] “keep pulling your
toes up” (to correct lack of dorsiflexion).
6- Vision
Therapeutic Goals:
Promote a more powerful muscle contraction.
motion.
Influence both the head and body motion.
antigravity motions.
Aid in elongation of muscle tissue when using
Therapeutic Goals:
Normal timing provides continuous, coordinated
Forearm pronated
Fingers extended
Hand Position:
Hand placed in patient's palm so that patient can grip and
above elbow
Movements to End Position:
Shoulder flexed, adducted, and externally rotated
Forearm supinated
Fingers flexed
D1 FLEXION - UL
D1 Extension :
Starting Position:
Shoulder flexed, adducted, and externally rotated
Forearm supinated
Fingers flexed
Hand Position:
Hand over dorsal-ulnar aspect of the patient's hand
above elbow
Movement to Ending Position:
Shoulder extended, abducted, and internally rotated
Forearm pronated
Fingers extened
D1 EXTENSION - UL
Upper Extremity D2 Flexion :
Starting Position:
Shoulder extended, adducted, and internally rotated
Forearm pronated
Fingers flexed
Hand Position:
Hand over dorsal-ulnar aspect of the patient's hand
Forearm supinated
Fingers extended
D2 FLEXION - UL
D2 Extension:
Starting Position:
Shoulder flexed, abducted, and externally rotated
Forearm supinated
Fingers extended
Hand Position:
Hand placed in the patient's palm so that the patient can
above elbow
Moving to Ending Position:
Shoulder extended, adducted, and internally rotated
Forearm pronated
Fingers flexed
D2 EXTENSION-UL
D1-FLEXION & EXTENSION
PNF PATTERN
Lower limb
D1 Flexion-LOWER LIMB :
Starting Position:
Hip extended, abducted, and internally rotated
Ankle plantarflexed
Foot everted
Toes flexed
Hand Position:
Hand on distal anterior-medial aspect of thigh
Ankle dorsiflexed
Foot inverted
Toes extended
D1 FLEXION - LOWER LIMB
D2 Flexion-LOWER LIMB :
Starting Position:
Hip extended, adducted, and externally rotated
Ankle plantarflexed
Foot inverted
Toes flexed
Hand Position:
Hand on distal anterior-lateral thigh
Ankle dorsiflexed
Foot everted
Toes Extended
D2 FLEXION – LOWER LIMB
D1 Extension LOWER LIMB:
Starting Position:
Hip flexed, adducted, and externally rotated
Ankle dorsiflexed
Foot inverted
Toes extended
Hand Position:
Hand on distal posterior-lateral thigh
Ankle plantarflexed
Foot everted
Toes flexed
D1 EXTENSION – LOWER LIMB
D2 Extension LOWER LIMB:
Starting Position:
Hip flexed, abducted, and internally rotated
Ankle dorsiflexed
Foot everted
Toes Entended
Hand Position:
Hand on distal posterior-medial thigh (wrapped around
foot
Movements to Ending Position:
Hip extended, adducted, and externally rotated
Ankle Plantarflexed
Foot inverted
Toes flexed
D2 EXTENSION - LOWER LIMB
D1-FLEXION,EXTENSION
&
D 2FLEXION,EXTENSION OF
UL AND LL
Trunk:
Upper trunk in sitting position:
Flexion with rotation to the left (Chopping),
Extension with rotation to the right (Lifting).
Lower Trunk
in supine position:
Flexion with rotation to the left, Extension with
rotation with the right.